Ari M. Melnick
Impact in
- Hematology top 0.05%
- Acute Myeloid Leukemia Research
- Genetics top 0.05%
Papers in
- Hematology 147
- Acute Myeloid Leukemia Research 112
-
- Epigenetics and DNA Methylation 145
- Cancer-related gene regulation 67
- Histone Deacetylase Inhibitors Research 44
- Co-authors
- María E. Figueroa (57 shared papers)Jonathan D. Licht (27 shared papers)Christopher E. Mason (26 shared papers)Francine E. Garrett-Bakelman (22 shared papers)Leandro C. A. Cerchietti (63 shared papers)Jose Maria Polo (26 shared papers)Sheng Li (7 shared papers)Rita Shaknovich (38 shared papers)
- Journals
- Blood (136 papers)Cancer Cell (27 papers)Cancer Discovery (23 papers)Cell Reports (15 papers)Nature Communications (15 papers)
- Partner nations
- United StatesCanadaGermany
In The Last Decade
Ari M. Melnick
449 papers receiving 43.0k citations
Ari M. Melnick's Hit Papers
Peers
Comparison fields: 5 of 170
- Hematology 10.6k
- Genetics 5.9k
- Cancer Research 6.5k
- Molecular Biology 26.2k
- Pathology and Forensic Medicine 5.4k
Countries citing papers authored by Ari M. Melnick
This map shows the geographic impact of Ari M. Melnick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ari M. Melnick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ari M. Melnick more than expected).
Fields of papers citing papers by Ari M. Melnick
This network shows the impact of papers produced by Ari M. Melnick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ari M. Melnick. The network helps show where Ari M. Melnick may publish in the future.
Co-authors
The 25 scholars most cited alongside Ari M. Melnick, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 461 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Leukemic IDH1 and IDH2 Mutations Result in a Hypermethylation Phenotype, Disrupt TET2 Function, and Impair Hematopoietic Differentiation Hit paper breakdown → | 2010 | 2292 |
| 2 | IDH mutation impairs histone demethylation and results in a block to cell differentiation Hit paper breakdown → | 2012 | 1648 |
| 3 | Prognostic Relevance of Integrated Genetic Profiling in Acute Myeloid Leukemia Hit paper breakdown → | 2012 | 1628 |
| 4 | methylKit: a comprehensive R package for the analysis of genome-wide DNA methylation profiles Hit paper breakdown → | 2012 | 1562 |
| 5 | Tet2 Loss Leads to Increased Hematopoietic Stem Cell Self-Renewal and Myeloid Transformation Hit paper breakdown → | 2011 | 1109 |
| 6 | The International Consensus Classification of Mature Lymphoid Neoplasms: a report from the Clinical Advisory Committee Hit paper breakdown → | 2022 | 1026 |
| 7 | The N6-methyladenosine (m6A)-forming enzyme METTL3 controls myeloid differentiation of normal hematopoietic and leukemia cells Hit paper breakdown → | 2017 | 1007 |
| 8 | Cell type of origin influences the molecular and functional properties of mouse induced pluripotent stem cells Hit paper breakdown → | 2010 | 977 |
| 9 | Deconstructing a Disease: RAR, Its Fusion Partners, and Their Roles in the Pathogenesis of Acute Promyelocytic Leukemia Hit paper breakdown → | 1999 | 879 |
| 10 | The NASA Twins Study: A multidimensional analysis of a year-long human spaceflight Hit paper breakdown → | 2019 | 812 |
| 11 | A Molecular Roadmap of Reprogramming Somatic Cells into iPS Cells Hit paper breakdown → | 2012 | 692 |
| 12 | DNA Methylation Signatures Identify Biologically Distinct Subtypes in Acute Myeloid Leukemia Hit paper breakdown → | 2010 | 690 |
| 13 | EZH2 Is Required for Germinal Center Formation and Somatic EZH2 Mutations Promote Lymphoid Transformation Hit paper breakdown → | 2013 | 689 |
| 14 | Recurrent somatic TET2 mutations in normal elderly individuals with clonal hematopoiesis Hit paper breakdown → | 2012 | 671 |
| 15 | 2012 | 499 | |
| 16 | 2012 | 444 | |
| 17 | 2013 | 429 | |
| 18 | 2015 | 360 | |
| 19 | 2016 | 304 | |
| 20 | 2015 | 300 |
About Ari M. Melnick
Ari M. Melnick is a scholar working on Hematology, Molecular Biology, Pathology and Forensic Medicine, Immunology and Oncology, having authored 461 papers that have together received 43.6k indexed citations. Recurring topics across this work include Epigenetics and DNA Methylation (145 papers), Acute Myeloid Leukemia Research (112 papers), Lymphoma Diagnosis and Treatment (79 papers), Cancer-related gene regulation (67 papers), Immune Cell Function and Interaction (61 papers), CAR-T cell therapy research (49 papers), Histone Deacetylase Inhibitors Research (44 papers) and Acute Lymphoblastic Leukemia research (41 papers). The work is most often cited by research in Hematology (10.6k citations), Genetics (5.9k citations), Cancer Research (6.5k citations), Molecular Biology (26.2k citations) and Pathology and Forensic Medicine (5.4k citations). Ari M. Melnick has collaborated with scholars based in United States, Canada and Germany. Frequent co-authors include María E. Figueroa, Jonathan D. Licht, Christopher E. Mason, Francine E. Garrett-Bakelman, Leandro C. A. Cerchietti, Jose Maria Polo, Sheng Li, Rita Shaknovich, Matthías Kormáksson and Katerina Hatzi. Their work appears in journals such as Blood, Cancer Cell, Cancer Discovery, Cell Reports and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.