Mazhar Adli

11.4k total citations · 4 hit papers
58 papers, 5.7k citations indexed

About

Mazhar Adli is a scholar working on Molecular Biology, Cancer Research and Reproductive Medicine. According to data from OpenAlex, Mazhar Adli has authored 58 papers receiving a total of 5.7k indexed citations (citations by other indexed papers that have themselves been cited), including 43 papers in Molecular Biology, 10 papers in Cancer Research and 8 papers in Reproductive Medicine. Recurrent topics in Mazhar Adli's work include CRISPR and Genetic Engineering (16 papers), Genomics and Chromatin Dynamics (15 papers) and Epigenetics and DNA Methylation (11 papers). Mazhar Adli is often cited by papers focused on CRISPR and Genetic Engineering (16 papers), Genomics and Chromatin Dynamics (15 papers) and Epigenetics and DNA Methylation (11 papers). Mazhar Adli collaborates with scholars based in United States, Türkiye and Japan. Mazhar Adli's co-authors include Cem Kuscu, B Bernstein, Ritambhara Singh, Jeremy Thorpe, Şevki Arslan, Albert S. Baldwin, Manching Ku, Richard P. Koche, Carolyn Brokowski and Chad A. Cowan and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Mazhar Adli

56 papers receiving 5.6k citations

Hit Papers

The CRISPR tool kit for genome editin... 2008 2026 2014 2020 2018 2008 2014 2023 250 500 750 1000

Peers

Mazhar Adli
Zuzana Tóthová United States
Jia Xu China
Raj Chari United States
Wei Yu China
Anya Tsalenko United States
Jie Xu United States
Mazhar Adli
Citations per year, relative to Mazhar Adli Mazhar Adli (= 1×) peers Junjiu Huang

Countries citing papers authored by Mazhar Adli

Since Specialization
Citations

This map shows the geographic impact of Mazhar Adli's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mazhar Adli with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mazhar Adli more than expected).

Fields of papers citing papers by Mazhar Adli

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mazhar Adli. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mazhar Adli. The network helps show where Mazhar Adli may publish in the future.

Co-authorship network of co-authors of Mazhar Adli

This figure shows the co-authorship network connecting the top 25 collaborators of Mazhar Adli. A scholar is included among the top collaborators of Mazhar Adli based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mazhar Adli. Mazhar Adli is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Iizuka, Takashi, et al.. (2025). Therapeutic targeting of the tryptophan-kynurenine-aryl hydrocarbon receptor pathway with apigenin in MED12-mutant leiomyoma cells. Cancer Gene Therapy. 32(4). 393–402. 1 indexed citations
2.
Wei, Xiaolong, Lucas Carter, Jiekun Yang, et al.. (2024). Depletion of lamins B1 and B2 promotes chromatin mobility and induces differential gene expression by a mesoscale-motion-dependent mechanism. Genome biology. 25(1). 77–77. 16 indexed citations
3.
Adli, Mazhar, et al.. (2024). Single-nuclei sequencing of uterine serous carcinoma reveals racial differences in immune signaling. Proceedings of the National Academy of Sciences. 121(34). e2402998121–e2402998121. 1 indexed citations
4.
Elkafas, Hoda, et al.. (2024). Integrating leiomyoma genetics, epigenomics, and single-cell transcriptomics reveals causal genetic variants, genes, and cell types. Nature Communications. 15(1). 1169–1169. 7 indexed citations
5.
Yıldız, Şule, Jian Wei, Magdy P. Milad, et al.. (2023). Adenomyosis: single-cell transcriptomic analysis reveals a paracrine mesenchymal–epithelial interaction involving the WNT/SFRP pathway. Fertility and Sterility. 119(5). 869–882. 23 indexed citations
6.
Bulun, Serdar E., Şule Yıldız, Mazhar Adli, et al.. (2023). Endometriosis and adenomyosis: shared pathophysiology. Fertility and Sterility. 119(5). 746–750. 88 indexed citations breakdown →
7.
Davis, Brian W., et al.. (2022). Evolutionary Biology and Gene Editing of Cat Allergen, Fel d 1. The CRISPR Journal. 5(2). 213–223. 17 indexed citations
8.
Niedzialkowska, E., Limin Liu, Cem Kuscu, et al.. (2022). Tip60 acetylation of histone H3K4 temporally controls chromosome passenger complex localization. Molecular Biology of the Cell. 33(9). br15–br15. 4 indexed citations
9.
Ali, Mourad W., Matthew A.M. Devall, Christopher H. Dampier, et al.. (2021). A Functional Variant on 9p21.3 Related to Glioma Risk Affects Enhancer Activity and Modulates Expression of CDKN2B‐AS1. Human Mutation. 42(10). 1208–1214. 7 indexed citations
10.
Wei, Xiaolong, Jiekun Yang, Sara J. Adair, et al.. (2020). Targeted CRISPR screening identifies PRMT5 as synthetic lethality combinatorial target with gemcitabine in pancreatic cancer cells. Proceedings of the National Academy of Sciences. 117(45). 28068–28079. 60 indexed citations
11.
Brokowski, Carolyn & Mazhar Adli. (2020). Ethical Considerations in Therapeutic Clinical Trials Involving Novel Human Germline-Editing Technology. The CRISPR Journal. 3(1). 18–26. 6 indexed citations
12.
Shang, Stephen, Jiekun Yang, Amir A. Jazaeri, et al.. (2019). Chemotherapy-Induced Distal Enhancers Drive Transcriptional Programs to Maintain the Chemoresistant State in Ovarian Cancer. Cancer Research. 79(18). 4599–4611. 44 indexed citations
13.
Szlachta, Karol, Cem Kuscu, Turan Tufan, et al.. (2018). CRISPR knockout screening identifies combinatorial drug targets in pancreatic cancer and models cellular drug response. Nature Communications. 9(1). 4275–4275. 57 indexed citations
14.
Brown, R., Turan Tufan, Stephen Shang, et al.. (2018). Super-enhancers maintain renin-expressing cell identity and memory to preserve multi-system homeostasis. Journal of Clinical Investigation. 128(11). 4787–4803. 34 indexed citations
15.
Adli, Mazhar. (2018). The CRISPR tool kit for genome editing and beyond. Nature Communications. 9(1). 1911–1911. 1101 indexed citations breakdown →
16.
Zhu, Jiang, Mazhar Adli, James Zou, et al.. (2013). Genome-wide Chromatin State Transitions Associated with Developmental and Environmental Cues. Cell. 152(3). 642–654. 380 indexed citations
17.
Adli, Mazhar & B Bernstein. (2011). Whole-genome chromatin profiling from limited numbers of cells using nano-ChIP-seq. Nature Protocols. 6(10). 1656–1668. 131 indexed citations
18.
Goren, Alon, Fatih Ozsolak, Noam Shoresh, et al.. (2009). Chromatin profiling by directly sequencing small quantities of immunoprecipitated DNA. Nature Methods. 7(1). 47–49. 89 indexed citations
19.
Dan, Han C., Mazhar Adli, & Albert S. Baldwin. (2007). Regulation of Mammalian Target of Rapamycin Activity in PTEN-Inactive Prostate Cancer Cells by IκB Kinase α. Cancer Research. 67(13). 6263–6269. 60 indexed citations
20.
Kütük, Özgür, Mazhar Adli, Giuseppe Poli, & Hüveyda Başağa. (2004). Resveratrol protects against 4‐HNE induced oxidative stress and apoptosis in Swiss 3T3 fibroblasts. BioFactors. 20(1). 1–10. 31 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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