Andrew Ramsay

3.7k total citations
27 papers, 1.5k citations indexed

About

Andrew Ramsay is a scholar working on Genetics, Molecular Biology and Hematology. According to data from OpenAlex, Andrew Ramsay has authored 27 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Genetics, 9 papers in Molecular Biology and 9 papers in Hematology. Recurrent topics in Andrew Ramsay's work include Hemoglobinopathies and Related Disorders (9 papers), Iron Metabolism and Disorders (8 papers) and Trace Elements in Health (5 papers). Andrew Ramsay is often cited by papers focused on Hemoglobinopathies and Related Disorders (9 papers), Iron Metabolism and Disorders (8 papers) and Trace Elements in Health (5 papers). Andrew Ramsay collaborates with scholars based in Spain, Australia and Switzerland. Andrew Ramsay's co-authors include Carlos López-Otı́n, José M.P. Freije, Vı́ctor Quesada, Gloria Velasco, Juan Cadiñanos, David Rodrı́guez, Pedro M. Quirós, Fernando G. Osorio, Antonio Fueyo and Francisco Rodríguez and has published in prestigious journals such as Nature Communications, Nature Genetics and Genes & Development.

In The Last Decade

Andrew Ramsay

27 papers receiving 1.5k citations

Peers

Andrew Ramsay
N. Saha Singapore
Aristeidis Chaidos United Kingdom
Matthew J. Oberley United States
A Tobler Switzerland
Andrew Ramsay
Citations per year, relative to Andrew Ramsay Andrew Ramsay (= 1×) peers Caroline Kannengiesser

Countries citing papers authored by Andrew Ramsay

Since Specialization
Citations

This map shows the geographic impact of Andrew Ramsay's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Andrew Ramsay with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Andrew Ramsay more than expected).

Fields of papers citing papers by Andrew Ramsay

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Andrew Ramsay. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Andrew Ramsay. The network helps show where Andrew Ramsay may publish in the future.

Co-authorship network of co-authors of Andrew Ramsay

This figure shows the co-authorship network connecting the top 25 collaborators of Andrew Ramsay. A scholar is included among the top collaborators of Andrew Ramsay based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Andrew Ramsay. Andrew Ramsay is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Muttamba, Winters, Bernadette O’Hare, Parul Tyagi, et al.. (2022). A systematic review of strategies adopted to scale up COVID-19 testing in low-, middle- and high-income countries. BMJ Open. 12(11). e060838–e060838. 9 indexed citations
2.
Folgueras, Alicia R., Sandra Freitas‐Rodríguez, Andrew Ramsay, et al.. (2018). Matriptase-2 deficiency protects from obesity by modulating iron homeostasis. Nature Communications. 9(1). 1350–1350. 39 indexed citations
3.
Lawson, Lovett, Matthew Blakiston, Joshua Obasanya, et al.. (2016). Are patients with pulmonary tuberculosis who are identified through active case finding in the community different than those identified in healthcare facilities?. New Microbes and New Infections. 15. 35–39. 6 indexed citations
4.
Senga, Mikiko, Kimberly Pringle, Andrew Ramsay, et al.. (2016). Factors Underlying Ebola Virus Infection Among Health Workers, Kenema, Sierra Leone, 2014–2015. Clinical Infectious Diseases. 63(4). 454–459. 20 indexed citations
5.
Phelan, Jody, Francesc Coll, Ruth McNerney, et al.. (2016). Mycobacterium tuberculosis whole genome sequencing and protein structure modelling provides insights into anti-tuberculosis drug resistance. BMC Medicine. 14(1). 31–31. 88 indexed citations
6.
Cuevas, Rachel Anderson de, Lovett Lawson, Najla Al‐Sonboli, et al.. (2016). Patients direct costs to undergo TB diagnosis. Infectious Diseases of Poverty. 5(1). 24–24. 12 indexed citations
7.
Willemetz, Alexandra, Jean‐Christophe Deschemin, Carlos López-Otı́n, et al.. (2014). Matriptase-2 is essential for hepcidin repression during fetal life and postnatal development in mice to maintain iron homeostasis. Blood. 124(3). 441–444. 27 indexed citations
8.
Rodrı́guez, David, Andrew Ramsay, Vı́ctor Quesada, et al.. (2013). Functional analysis of sucrase–isomaltase mutations from chronic lymphocytic leukemia patients. Human Molecular Genetics. 22(11). 2273–2282. 17 indexed citations
9.
Fanjul‐Fernández, Miriam, Vı́ctor Quesada, Rubén Cabanillas, et al.. (2013). Cell–cell adhesion genes CTNNA2 and CTNNA3 are tumour suppressors frequently mutated in laryngeal carcinomas. Nature Communications. 4(1). 2531–2531. 69 indexed citations
10.
Quesada, Vı́ctor, Andrew Ramsay, David Rodrı́guez, et al.. (2013). The genomic landscape of chronic lymphocytic leukemia: clinical implications. BMC Medicine. 11(1). 124–124. 24 indexed citations
11.
Ramsay, Andrew, Vı́ctor Quesada, Miguel Foronda, et al.. (2013). POT1 mutations cause telomere dysfunction in chronic lymphocytic leukemia. Nature Genetics. 45(5). 526–530. 189 indexed citations
12.
Ramsay, Andrew, et al.. (2012). Next-generation sequencing reveals the secrets of the chronic lymphocytic leukemia genome. Clinical & Translational Oncology. 15(1). 3–8. 26 indexed citations
13.
Osorio, Fernando G., Clea Bárcena, Clara Soria‐Valles, et al.. (2012). Nuclear lamina defects cause ATM-dependent NF-κB activation and link accelerated aging to a systemic inflammatory response. Genes & Development. 26(20). 2311–2324. 225 indexed citations
14.
Quirós, Pedro M., Andrew Ramsay, David Sala, et al.. (2012). Loss of mitochondrial protease OMA1 alters processing of the GTPase OPA1 and causes obesity and defective thermogenesis in mice. The EMBO Journal. 31(9). 2117–2133. 209 indexed citations
15.
Quirós, Pedro M., Andrew Ramsay, & Carlos López-Otı́n. (2012). New roles for OMA1 metalloprotease. Adipocyte. 2(1). 7–11. 8 indexed citations
16.
Ugalde, Alejandro P., Andrew Ramsay, Jorge de la Rosa, et al.. (2011). Aging and chronic DNA damage response activate a regulatory pathway involving miR‐29 and p53. The EMBO Journal. 30(11). 2219–2232. 198 indexed citations
17.
Krijt, Jan, et al.. (2011). Liver hemojuvelin protein levels in mice deficient in matriptase-2 (Tmprss6). Blood Cells Molecules and Diseases. 47(2). 133–137. 24 indexed citations
18.
Deschemin, Jean‐Christophe, Léon Kautz, Andrew Ramsay, et al.. (2010). Iron-deficiency anemia from matriptase-2 inactivation is dependent on the presence of functional Bmp6. Blood. 117(2). 647–650. 27 indexed citations
19.
Ramsay, Andrew, Vı́ctor Quesada, Mayka Sánchez, et al.. (2009). Matriptase-2 mutations in iron-refractory iron deficiency anemia patients provide new insights into protease activation mechanisms. Human Molecular Genetics. 18(19). 3673–3683. 45 indexed citations
20.
Ramsay, Andrew, John D. Hooper, Alicia R. Folgueras, Gloria Velasco, & Carlos López-Otı́n. (2009). Matriptase-2 (TMPRSS6): a proteolytic regulator of iron homeostasis. Haematologica. 94(6). 840–849. 91 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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