José M.P. Freije

14.3k total citations · 3 hit papers
98 papers, 9.2k citations indexed

About

José M.P. Freije is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, José M.P. Freije has authored 98 papers receiving a total of 9.2k indexed citations (citations by other indexed papers that have themselves been cited), including 71 papers in Molecular Biology, 21 papers in Oncology and 20 papers in Cancer Research. Recurrent topics in José M.P. Freije's work include Nuclear Structure and Function (26 papers), RNA Research and Splicing (19 papers) and Protease and Inhibitor Mechanisms (14 papers). José M.P. Freije is often cited by papers focused on Nuclear Structure and Function (26 papers), RNA Research and Splicing (19 papers) and Protease and Inhibitor Mechanisms (14 papers). José M.P. Freije collaborates with scholars based in Spain, United States and France. José M.P. Freije's co-authors include Carlos López-Otı́n, Alberto M. Pendás, Milagros Balbı́n, Juan Cadiñanos, Luis Sánchez‐Pulido, Fernando G. Osorio, Ignacio Varela, Guido Kroemer, Francisco Rodríguez and Vı́ctor Quesada and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

José M.P. Freije

96 papers receiving 9.0k citations

Hit Papers

Molecular cloning and expression of collagenase-3, a nove... 1994 2026 2004 2015 1994 2016 2019 200 400 600

Peers

José M.P. Freije
Diego H. Castrillón United States
Parmjit Jat United Kingdom
Anne E. Willis United Kingdom
Vittorio Sartorelli United States
Gordon Peters United Kingdom
Bing Lim United States
José M.P. Freije
Citations per year, relative to José M.P. Freije José M.P. Freije (= 1×) peers Luis Sánchez‐Pulido

Countries citing papers authored by José M.P. Freije

Since Specialization
Citations

This map shows the geographic impact of José M.P. Freije's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by José M.P. Freije with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites José M.P. Freije more than expected).

Fields of papers citing papers by José M.P. Freije

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by José M.P. Freije. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by José M.P. Freije. The network helps show where José M.P. Freije may publish in the future.

Co-authorship network of co-authors of José M.P. Freije

This figure shows the co-authorship network connecting the top 25 collaborators of José M.P. Freije. A scholar is included among the top collaborators of José M.P. Freije based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with José M.P. Freije. José M.P. Freije is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ugalde, Alejandro P., et al.. (2024). Noncoding RNA Contribution to Aging and Lifespan. The Journals of Gerontology Series A. 79(4). 4 indexed citations
2.
López-Otı́n, Carlos, et al.. (2024). Loss of ADAM29 does not affect viability and fertility in mice but improves wound healing. iScience. 27(6). 110135–110135.
3.
Fraile, Julia M., Gabriel Bretones, Elena Bonzón‐Kulichenko, et al.. (2023). USP49 deubiquitinase regulates the mitotic spindle checkpoint and prevents aneuploidy. Cell Death and Disease. 14(1). 60–60. 1 indexed citations
4.
Santiago‐Fernández, Olaya, Fernando G. Osorio, Vı́ctor Quesada, et al.. (2019). Development of a CRISPR/Cas9-based therapy for Hutchinson–Gilford progeria syndrome. Nature Medicine. 25(3). 423–426. 115 indexed citations
5.
Rosa, Jorge de la, José M.P. Freije, Rubén Cabanillas, et al.. (2013). Prelamin A causes progeria through cell-extrinsic mechanisms and prevents cancer invasion. Nature Communications. 4(1). 2268–2268. 57 indexed citations
6.
Rodrı́guez, David, Andrew Ramsay, Vı́ctor Quesada, et al.. (2013). Functional analysis of sucrase–isomaltase mutations from chronic lymphocytic leukemia patients. Human Molecular Genetics. 22(11). 2273–2282. 17 indexed citations
7.
Freije, José M.P. & Carlos López-Otı́n. (2012). Reprogramming aging and progeria. Current Opinion in Cell Biology. 24(6). 757–764. 35 indexed citations
8.
Peinado, Juan R., Pedro M. Quirós, Marina R. Pulido, et al.. (2011). Proteomic Profiling of Adipose Tissue from Zmpste24−/− Mice, a Model of Lipodystrophy and Premature Aging, Reveals Major Changes in Mitochondrial Function and Vimentin Processing. Molecular & Cellular Proteomics. 10(11). M111.008094–M111.008094. 59 indexed citations
9.
Puente, Xosé S., Vı́ctor Quesada, Fernando G. Osorio, et al.. (2011). Exome Sequencing and Functional Analysis Identifies BANF1 Mutation as the Cause of a Hereditary Progeroid Syndrome. The American Journal of Human Genetics. 88(5). 650–656. 154 indexed citations
10.
Mariño, Guillermo, Alejandro P. Ugalde, Álvaro F. Fernández, et al.. (2010). Insulin-like growth factor 1 treatment extends longevity in a mouse model of human premature aging by restoring somatotroph axis function. Proceedings of the National Academy of Sciences. 107(37). 16268–16273. 115 indexed citations
11.
Espada, Jesús, Ignacio Varela, Ignacio Flores, et al.. (2008). Nuclear envelope defects cause stem cell dysfunction in premature-aging mice. The Journal of Cell Biology. 181(1). 27–35. 133 indexed citations
12.
Varela, Ignacio, Juan Cadiñanos, Alberto M. Pendás, et al.. (2005). Accelerated ageing in mice deficient in Zmpste24 protease is linked to p53 signalling activation. Nature. 437(7058). 564–568. 367 indexed citations
13.
Cadiñanos, Juan, Ignacio Varela, Carlos López-Otı́n, & José M.P. Freije. (2005). From Immature Lamin to Premature Aging: Molecular Pathways and Therapeutic Opportunities. Cell Cycle. 4(12). 1732–1735. 25 indexed citations
14.
Cal, Santiago, José M.P. Freije, José M. López, Yoshikazu Takada, & Carlos López-Otı́n. (2000). ADAM 23/MDC3, a Human Disintegrin That Promotes Cell Adhesion via Interaction with the αvβ3 Integrin through an RGD-independent Mechanism. Molecular Biology of the Cell. 11(4). 1457–1469. 97 indexed citations
15.
Balbı́n, Milagros, Alberto M. Pendás, José A. Urı́a, et al.. (1999). Expression and regulation of collagenase‐3 (MMP‐13) in human malignant tumors. Apmis. 107(1-6). 45–53. 72 indexed citations
16.
Urı́a, José A., María Jiménez, Milagros Balbı́n, José M.P. Freije, & Carlos López-Otı́n. (1998). Differential Effects of Transforming Growth Factor-β on the Expression of Collagenase-1 and Collagenase-3 in Human Fibroblasts. Journal of Biological Chemistry. 273(16). 9769–9777. 177 indexed citations
17.
Freije, José M.P., Pilar Blay, Nicholas J. MacDonald, Richard E. Manrow, & Patricia S. Steeg. (1997). Site-directed Mutation of Nm23-H1. Journal of Biological Chemistry. 272(9). 5525–5532. 122 indexed citations
18.
Freije, José M.P., Alberto M. Pendás, Gloria Velasco, et al.. (1993). Localization of the human cystatin D gene (CST5) to chromosome 20p11.21 by in situ hybridization. Cytogenetic and Genome Research. 62(1). 29–31. 14 indexed citations
19.
Freije, José M.P., et al.. (1992). A gene homologous to topoisomerase II in African swine fever virus. Virology. 188(2). 938–947. 42 indexed citations
20.
Freije, José M.P., Antonio Fueyo, José A. Urı́a, & Carlos López-Otı́n. (1991). Human Zn‐α2‐glycoprotein cDNA cloning and expression analysis in benign and malignant breast tissues. FEBS Letters. 290(1-2). 247–249. 37 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026