A.J. Alldrick

966 total citations
40 papers, 599 citations indexed

About

A.J. Alldrick is a scholar working on Cancer Research, Molecular Biology and Food Science. According to data from OpenAlex, A.J. Alldrick has authored 40 papers receiving a total of 599 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Cancer Research, 17 papers in Molecular Biology and 13 papers in Food Science. Recurrent topics in A.J. Alldrick's work include Carcinogens and Genotoxicity Assessment (18 papers), Mycotoxins in Agriculture and Food (9 papers) and Genomics, phytochemicals, and oxidative stress (7 papers). A.J. Alldrick is often cited by papers focused on Carcinogens and Genotoxicity Assessment (18 papers), Mycotoxins in Agriculture and Food (9 papers) and Genomics, phytochemicals, and oxidative stress (7 papers). A.J. Alldrick collaborates with scholars based in United Kingdom, Italy and Netherlands. A.J. Alldrick's co-authors include Ian Rowland, Liana Giorgi, Huang Wei Ling, Ruijia Yang, Xiaofang Pei, John J. Flynn, Brian G. Lake, Michele Solfrizzo, Hans P. van Egmond and S. Venitt and has published in prestigious journals such as JNCI Journal of the National Cancer Institute, Carcinogenesis and Food and Chemical Toxicology.

In The Last Decade

A.J. Alldrick

38 papers receiving 568 citations

Peers

A.J. Alldrick

HTM

Fredi Schwägele Germany

Ben Lau Canada

Paul Hepburn United Kingdom

John Wenninger United States

Ian Crawford Dewhurst United Kingdom

Ib Knudsen Denmark

William I. Jones United States

Julia H. Fentem United Kingdom

Leslie J. Copp Canada

J. Richard Bacon United Kingdom

Fredi Schwägele Germany

A.J. Alldrick

486 ×2.4

319 ×1.6

43 ×0.3

163 ×1.1

225 ×3.9

HTM

Citations per year, relative to A.J. Alldrick A.J. Alldrick (= 1×) peers Fredi Schwägele

Countries citing papers authored by A.J. Alldrick

Since Specialization

Citations

This map shows the geographic impact of A.J. Alldrick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A.J. Alldrick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A.J. Alldrick more than expected).

Fields of papers citing papers by A.J. Alldrick

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A.J. Alldrick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A.J. Alldrick. The network helps show where A.J. Alldrick may publish in the future.

Co-authorship network of co-authors of A.J. Alldrick

This figure shows the co-authorship network connecting the top 25 collaborators of A.J. Alldrick. A scholar is included among the top collaborators of A.J. Alldrick based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with A.J. Alldrick. A.J. Alldrick is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Pei, Xiaofang, et al.. (2011). The China Melamine Milk Scandal and its Implications for Food Safety Regulation. SSRN Electronic Journal. 1 indexed citations

Alldrick, A.J. & Christine Knight. (2000). Mycotoxins in cereals - prevention is better than cure.. 701–706. 1 indexed citations

Alldrick, A.J., et al.. (1995). Dietary caffeine reduces the genotoxicity of MeIQx in the host‐mediated assay in mice. Nutrition and Cancer. 24(2). 143–150. 9 indexed citations

Alldrick, A.J., et al.. (1993). Influence of dietary fat on DNA binding by 2-amino-3,8-dimethylimidazo[4,5-f]quinoxaline (MeIQx) in the mouse liver. Food and Chemical Toxicology. 31(7). 483–489. 6 indexed citations

Alldrick, A.J., et al.. (1992). Effect of hepatic cytochrome P-450 inducing agents on mutagen activity in the host-mediated assay. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 268(2). 307–314. 2 indexed citations

Alldrick, A.J., et al.. (1992). Effect in vitro of arachidonic acid, eicosapentaenoic acid and docosahexaenoic acid on the hepatic activation of dietary genotoxins by rat post-mitochondrial fractions. Food and Chemical Toxicology. 30(10). 853–858. 4 indexed citations

Rowland, Ian, et al.. (1992). Inhibition of the metabolism of mutagens occurring in food by arachidonic acid. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 269(2). 279–284. 7 indexed citations

Howes, Angela, et al.. (1990). Roles of dietary fat and fibre in the disposition and metabolism of carcinogens associated with foods. Food Additives & Contaminants. 7(sup1). S53–S54. 3 indexed citations

Alldrick, A.J. & Werner K. Lutz. (1989). Covalent binding of [2-¹⁴C]2-amino-3,8-dimethylimidazo[4,5-f]-quinoxaline (MeIQx) to mouse DNA in vivo. Carcinogenesis. 10(8). 1419–1423. 6 indexed citations

10.

Alldrick, A.J., Brian G. Lake, & Ian Rowland. (1989). Modification of in vivo heterocyclic amine genotoxicity by dietary flavonoids. Mutagenesis. 4(5). 365–370. 20 indexed citations

11.

Howes, Angela, Ian Rowland, Brian G. Lake, & A.J. Alldrick. (1989). Effect of dietary fibre on the mutagenicity and distribution of 2-amino-3,4-dimethylimidazo[4,5-f]quinoline (MeIQ). Mutation research. Fundamental and molecular mechanisms of mutagenesis. 210(2). 227–235. 16 indexed citations

12.

Alldrick, A.J. & Ian Rowland. (1988). Caffeine inhibits hepatic-microsomal activation of some dietary genotoxins. Mutagenesis. 3(5). 423–427. 14 indexed citations

13.

Alldrick, A.J. & Ian Rowland. (1988). Distribution of radiolabelled [2-14C]IQ and MeIQx in the mouse. Toxicology Letters. 44(1-2). 183–190. 14 indexed citations

14.

Alldrick, A.J., et al.. (1987). Use of continuous culture to study the gastric microflora of a hypochlorhydric patient. Toxicology in Vitro. 1(1). 17–21. 6 indexed citations

15.

Alldrick, A.J., Ian Rowland, & Alan Wise. (1987). The hepatic conversion of some heterocyclic amines to bacterial mutagens is modified by dietary fat and cholesterol. Mutagenesis. 2(3). 221–224. 9 indexed citations

16.

Alldrick, A.J. & Ian Rowland. (1987). Counteraction of the genotoxicity of some cooked-food mutagens by biogenic amines. Food and Chemical Toxicology. 25(8). 575–580. 4 indexed citations

17.

Alldrick, A.J., Brian G. Lake, John J. Flynn, & Ian Rowland. (1986). Metabolic conversion of IQ and MeIQ to bacterial mutagens. Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis. 163(2). 109–114. 15 indexed citations

18.

Alldrick, A.J., John J. Flynn, & Ian Rowland. (1986). Effects of plant-derived flavonoids and polyphenolic acids on the activity of mutagens from cooked food. Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis. 163(3). 225–232. 75 indexed citations

19.

Alldrick, A.J., R.C. Cottrell, I.R. Rowland, & S.D. Gangolli. (1985). The role of DNA-repair processes in N-nitrosopyrrolidine-induced mutagenesis. Carcinogenesis. 6(1). 105–108. 3 indexed citations

20.

Alldrick, A.J. & Ian Rowland. (1984). Exposure to low concentrations of mutagens alter the mutagenic and lethal effects of 4-nitroquinoline-N-oxide and mitomycin C in Escherichia coli. Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis. 126(2). 121–126.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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