Adrian L. Oblak

4.2k total citations
83 papers, 2.3k citations indexed

About

Adrian L. Oblak is a scholar working on Physiology, Molecular Biology and Neurology. According to data from OpenAlex, Adrian L. Oblak has authored 83 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Physiology, 29 papers in Molecular Biology and 28 papers in Neurology. Recurrent topics in Adrian L. Oblak's work include Alzheimer's disease research and treatments (33 papers), Neuroinflammation and Neurodegeneration Mechanisms (22 papers) and Prion Diseases and Protein Misfolding (7 papers). Adrian L. Oblak is often cited by papers focused on Alzheimer's disease research and treatments (33 papers), Neuroinflammation and Neurodegeneration Mechanisms (22 papers) and Prion Diseases and Protein Misfolding (7 papers). Adrian L. Oblak collaborates with scholars based in United States, United Kingdom and Italy. Adrian L. Oblak's co-authors include Gene J. Blatt, Terrell T. Gibbs, Bernardino Ghetti, Michel Goedert, Keith A. Johnson, Bradford C. Dickerson, Bradley F. Boeve, Bruce T. Lamb, Peter Bor‐Chian Lin and Gary E. Landreth and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Brain and Neurology.

In The Last Decade

Adrian L. Oblak

79 papers receiving 2.3k citations

Peers

Adrian L. Oblak

PHYSI

NEURO

CMN

M. Axel Wollmer Germany

Jarek Wegiel United States

Derya R. Shimshek Switzerland

Shuji Iritani Japan

Daisuke Ibi Japan

Guomei Tang United States

Merina Varghese United States

Darius Ebrahimi‐Fakhari United States

Susan E. Maloney United States

Ellen Kanter United States

M. Axel Wollmer Germany

Adrian L. Oblak

1.3k ×1.4

PHYSI

954 ×1.2

474 ×0.8

NEURO

324 ×0.6

474 ×1.1

CMN

Citations per year, relative to Adrian L. Oblak Adrian L. Oblak (= 1×) peers M. Axel Wollmer

Countries citing papers authored by Adrian L. Oblak

Since Specialization

Citations

This map shows the geographic impact of Adrian L. Oblak's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Adrian L. Oblak with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Adrian L. Oblak more than expected).

Fields of papers citing papers by Adrian L. Oblak

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Adrian L. Oblak. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Adrian L. Oblak. The network helps show where Adrian L. Oblak may publish in the future.

Co-authorship network of co-authors of Adrian L. Oblak

This figure shows the co-authorship network connecting the top 25 collaborators of Adrian L. Oblak. A scholar is included among the top collaborators of Adrian L. Oblak based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Adrian L. Oblak. Adrian L. Oblak is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Plotkin, Lilian I., et al.. (2024). Mind Gaps and Bone Snaps: Exploring the Connection Between Alzheimer’s Disease and Osteoporosis. Current Osteoporosis Reports. 22(5). 483–494. 8 indexed citations

Plotkin, Lilian I., et al.. (2024). Use of AI Language Engine ChatGPT 4.0 to Write a Scientific Review Article Examining the Intersection of Alzheimer’s Disease and Bone. Current Osteoporosis Reports. 22(1). 177–181. 17 indexed citations

Plotkin, Lilian I., et al.. (2024). From the Mind to the Spine: The Intersecting World of Alzheimer’s and Osteoporosis. Current Osteoporosis Reports. 22(1). 152–164. 13 indexed citations

Jesudason, Cynthia D., Peter Bor‐Chian Lin, Audrey Lee‐Gosselin, et al.. (2024). Optimization of SHIP1 Inhibitors for the treatment of Alzheimer’s disease. Alzheimer s & Dementia. 20(S6). 1 indexed citations

Lin, Peter Bor‐Chian, Audrey Lee‐Gosselin, Cynthia M. Ingraham, et al.. (2024). Inpp5d haplodeficiency alleviates tau pathology in the PS19 mouse model of Tauopathy. Alzheimer s & Dementia. 20(7). 4985–4998. 8 indexed citations

Oblak, Adrian L., et al.. (2024). HUNK as a key regulator of tumor-associated macrophages in triple negative breast cancer. OncoImmunology. 13(1). 2364382–2364382. 4 indexed citations

Garza-Lombó, Carla, et al.. (2024). Peripheral HMGB1 is linked to O₃ pathology of disease‐associated astrocytes and amyloid. Alzheimer s & Dementia. 20(5). 3551–3566. 8 indexed citations

Yamada, Chiaki, K. Christopher García, Adrian L. Oblak, et al.. (2024). Dementia exacerbates periodontal bone loss in females. Journal of Periodontal Research. 59(3). 512–520. 2 indexed citations

Quinney, Sara K., Adrian L. Oblak, Kristen D. Onos, et al.. (2023). STOP‐AD portal: Selecting the optimal pharmaceutical for preclinical drug testing in Alzheimer's disease. Alzheimer s & Dementia. 19(11). 5289–5295. 1 indexed citations

10.

Lee‐Gosselin, Audrey, Nur Jury, Sayan Deb Dutta, et al.. (2023). TREM2-Deficient Microglia Attenuate Tau Spreading In Vivo. Cells. 12(12). 1597–1597. 10 indexed citations

11.

Moutinho, Miguel, Shweta S. Puntambekar, Andy P. Tsai, et al.. (2022). The niacin receptor HCAR2 modulates microglial response and limits disease progression in a mouse model of Alzheimer’s disease. Science Translational Medicine. 14(637). eabl7634–eabl7634. 52 indexed citations

12.

Smith, Daniel C., Byung-Wook Kim, Md. Mamun Al-Amin, et al.. (2021). Deletion of Abi3 gene locus exacerbates neuropathological features of Alzheimer’s disease in a mouse model of Aβ amyloidosis. Science Advances. 7(45). eabe3954–eabe3954. 37 indexed citations

13.

Oblak, Adrian L., Stefânia Forner, Paul R. Territo, et al.. (2020). Model organism development and evaluation for late‐onset Alzheimer's disease: MODEL‐AD. Alzheimer s & Dementia Translational Research & Clinical Interventions. 6(1). e12110–e12110. 62 indexed citations

14.

Reilly, Austin M., Andy P. Tsai, Peter Bor‐Chian Lin, et al.. (2020). Metabolic Defects Caused by High-Fat Diet Modify Disease Risk through Inflammatory and Amyloidogenic Pathways in a Mouse Model of Alzheimer’s Disease. Nutrients. 12(10). 2977–2977. 23 indexed citations

15.

Umeh, Chizoba C., Piyush Kalakoti, Silvio Notari, et al.. (2016). Clinicopathological Correlates in a PRNP P102L Mutation Carrier with Rapidly Progressing Parkinsonism-dystonia. PMC. 1 indexed citations

16.

Deters, Kacie, Shannon L. Risacher, Karmen K. Yoder, et al.. (2016). [(11)C]PiB PET in Gerstmann-Sträussler-Scheinker disease. IUScholarWorks (Indiana University). 1 indexed citations

17.

Boyd, Clara, Michael Tierney, Eric M. Wassermann, et al.. (2015). Sensitivity and Specificity of New Criteria for the Diagnosis of Corticobasal Degeneration (P5.010). Neurology. 84(14_supplement). 2 indexed citations

18.

Oblak, Adrian L., Terrell T. Gibbs, & Gene J. Blatt. (2010). Decreased GABA_B receptors in the cingulate cortex and fusiform gyrus in Autism. Journal of Neurochemistry. 114(5). 1414–1423. 149 indexed citations

19.

Oblak, Adrian L., Terrell T. Gibbs, & Gene J. Blatt. (2010). Reduced GABAA receptors and benzodiazepine binding sites in the posterior cingulate cortex and fusiform gyrus in autism. Brain Research. 1380. 218–228. 131 indexed citations

20.

Abergel, Éric & Adrian L. Oblak. (2006). [Echocardiography in athletes].. PubMed. 99(11). 969–74. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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