Yumiko Kaseda
- Cellular and Molecular Neuroscience top 10%
- Cognitive Neuroscience top 10%
- Neurology top 10%
- Molecular Biology
- Developmental Neuroscience top 10%
- Co-authors
- Walter C. LowYasuyo MimoriJay SimonHiroyuki MuranakaChunhui JiangShozo TobimatsuShigenobu NakamuraTakao Yamasaki
- Topics
- Neurological disorders and treatments (6 papers)Neural dynamics and brain function (4 papers)Transcranial Magnetic Stimulation Studies (4 papers)
- Journals
- SHILAP Revista de lepidopterologíaBrain ResearchJournal of Neurochemistry
- Partner nations
- JapanUnited StatesArmenia
In The Last Decade
Yumiko Kaseda
31 papers receiving 461 citations
Peers
Comparison fields: 5 of 78
- Cellular and Molecular Neuroscience 188
- Cognitive Neuroscience 166
- Neurology 114
- Molecular Biology 101
- Developmental Neuroscience 54
Countries citing papers authored by Yumiko Kaseda
This map shows the geographic impact of Yumiko Kaseda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yumiko Kaseda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yumiko Kaseda more than expected).
Fields of papers citing papers by Yumiko Kaseda
This network shows the impact of papers produced by Yumiko Kaseda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yumiko Kaseda. The network helps show where Yumiko Kaseda may publish in the future.
Co-authorship network of co-authors of Yumiko Kaseda
This figure shows the co-authorship network connecting the top 25 collaborators of Yumiko Kaseda. A scholar is included among the top collaborators of Yumiko Kaseda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yumiko Kaseda. Yumiko Kaseda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 4 | |
| 2 | 0 | |
| 3 | 2 | |
| 4 | 5 | |
| 5 | 36 | |
| 6 | 4 | |
| 7 | 23 | |
| 8 | 13 | |
| 9 | 25 | |
| 10 | 15 | |
| 11 | 14 | |
| 12 | 28 | |
| 13 | 8 | |
| 14 | 40 | |
| 15 | 1 | |
| 16 | 8 | |
| 17 | 33 | |
| 18 | 21 | |
| 19 | 26 | |
| 20 | 25 |
About Yumiko Kaseda
Yumiko Kaseda is a scholar working on Neurology, Neurology and Cellular and Molecular Neuroscience, having authored 32 papers that have together received 473 indexed citations. Recurring topics across this work include Neurological disorders and treatments (6 papers), Neural dynamics and brain function (4 papers) and Transcranial Magnetic Stimulation Studies (4 papers). The work is most often cited by research in Developmental Neuroscience (54 citations), Cellular and Molecular Neuroscience (188 citations) and Cognitive Neuroscience (166 citations). Yumiko Kaseda has collaborated with scholars based in Japan, United States and Armenia. Frequent co-authors include Walter C. Low, Yasuyo Mimori, Jay Simon, Hiroyuki Muranaka, Chunhui Jiang, Shozo Tobimatsu, Shigenobu Nakamura, Takao Yamasaki, Bernardino Ghetti and Tatsuo Kohriyama. Their work appears in journals such as SHILAP Revista de lepidopterología, Brain Research and Journal of Neurochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.