Osamu Komure

3.7k total citations
32 papers, 1.3k citations indexed

About

Osamu Komure is a scholar working on Neurology, Cellular and Molecular Neuroscience and Molecular Biology. According to data from OpenAlex, Osamu Komure has authored 32 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Neurology, 18 papers in Cellular and Molecular Neuroscience and 12 papers in Molecular Biology. Recurrent topics in Osamu Komure's work include Parkinson's Disease Mechanisms and Treatments (11 papers), Genetic Neurodegenerative Diseases (10 papers) and Neurological disorders and treatments (7 papers). Osamu Komure is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (11 papers), Genetic Neurodegenerative Diseases (10 papers) and Neurological disorders and treatments (7 papers). Osamu Komure collaborates with scholars based in Japan, United States and France. Osamu Komure's co-authors include Sadako Kuno, Yoshikuni Mizuno, Makio Mogi, Toshiharu Nagatsu, Akifumi Togari, Hiroshi Ichinose, Tomoyoshi Kondo, Toshikazu Kondo, Hideshi Kawakami and Akira Sano and has published in prestigious journals such as Neurology, Annals of Neurology and Biological Psychiatry.

In The Last Decade

Osamu Komure

32 papers receiving 1.3k citations

Peers

Osamu Komure
Yves Sagot Switzerland
Jason P. Covy United States
Deborah E. Cabin United States
Emma Hockly United Kingdom
Osamu Komure
Citations per year, relative to Osamu Komure Osamu Komure (= 1×) peers Yasuhiro Kawamoto

Countries citing papers authored by Osamu Komure

Since Specialization
Citations

This map shows the geographic impact of Osamu Komure's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Osamu Komure with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Osamu Komure more than expected).

Fields of papers citing papers by Osamu Komure

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Osamu Komure. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Osamu Komure. The network helps show where Osamu Komure may publish in the future.

Co-authorship network of co-authors of Osamu Komure

This figure shows the co-authorship network connecting the top 25 collaborators of Osamu Komure. A scholar is included among the top collaborators of Osamu Komure based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Osamu Komure. Osamu Komure is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Nakamura, Masayuki, Takeshi Yasuda, Teiichi Onuma, et al.. (2020). DNA analysis of benign adult familial myoclonic epilepsy reveals associations between the pathogenic TTTCA repeat insertion in SAMD12 and the nonpathogenic TTTTA repeat expansion in TNRC6A. Journal of Human Genetics. 66(4). 419–429. 10 indexed citations
2.
Ayaki, Takashi, Hidefumi Ito, Osamu Komure, et al.. (2017). Multiple Proteinopathies in Familial ALS Cases With Optineurin Mutations. Journal of Neuropathology & Experimental Neurology. 77(2). 128–138. 21 indexed citations
3.
Ito, Hidefumi, Masataka Nakamura, Osamu Komure, et al.. (2011). Clinicopathologic study on an ALS family with a heterozygous E478G optineurin mutation. Acta Neuropathologica. 122(2). 223–229. 50 indexed citations
4.
Yanagi, Masaya, Takeshi Hashimoto, Noboru Kitamura, et al.. (2008). Expression of Kruppel-like factor 5 gene in human brain and association of the gene with the susceptibility to schizophrenia. Schizophrenia Research. 100(1-3). 291–301. 27 indexed citations
5.
Yasuda, Minoru, et al.. (2005). Phenotypic heterogeneity within a new family with the MAPT p301s mutation. Annals of Neurology. 58(6). 920–928. 17 indexed citations
6.
Oda, Masaya, Hirofumi Maruyama, Osamu Komure, et al.. (2004). Possible Reduced Penetrance of Expansion of 44 to 47 CAG/CAA Repeats in the TATA-Binding Protein Gene in Spinocerebellar Ataxia Type 17. Archives of Neurology. 61(2). 209–209. 44 indexed citations
7.
Izumi, Yuishin, Hirofumi Maruyama, Masaya Oda, et al.. (2003). SCA8 Repeat Expansion: Large CTA/CTG Repeat Alleles Are More Common in Ataxic Patients, Including Those with SCA6. The American Journal of Human Genetics. 72(3). 704–709. 49 indexed citations
8.
Nishimura, Masataka, Hideshi Kawakami, Osamu Komure, et al.. (2002). Contribution of the interleukin‐1β gene polymorphism in multiple system atrophy. Movement Disorders. 17(4). 808–811. 45 indexed citations
9.
Mogi, Makio, Akifumi Togari, Toshikazu Kondo, et al.. (2000). Caspase activities and tumor necrosis factor receptor R1 (p55) level are elevated in the substantia nigra from Parkinsonian brain. Journal of Neural Transmission. 107(3). 335–341. 223 indexed citations
10.
Yasuda, Shingo, Kiyoshi Inoue, Miho Hirabayashi, et al.. (1999). Triggering of neuronal cell death by accumulation of activated SEK1 on nuclear polyglutamine aggregations in PML bodies. Genes to Cells. 4(12). 743–756. 56 indexed citations
11.
Hirano, Makito, Osamu Komure, & Satoshi Ueno. (1999). A novel missense mutant inactivates GTP cyclohydrolase I in dopa-responsive dystonia. Neuroscience Letters. 260(3). 181–184. 3 indexed citations
12.
Mogi, Makio, Akifumi Togari, Tomoyoshi Kondo, et al.. (1999). Brain-derived growth factor and nerve growth factor concentrations are decreased in the substantia nigra in Parkinson's disease. Neuroscience Letters. 270(1). 45–48. 327 indexed citations
13.
Kitamura, Noboru, Naoki Nishino, Takeshi Hashimoto, et al.. (1998). Asymmetrical changes in the fodrin α subunit in the superior temporal cortices in schizophrenia. Biological Psychiatry. 43(4). 254–262. 9 indexed citations
14.
Toji, Hiromasa, Hideshi Kawakami, Toshitaka Kawarai, et al.. (1998). No association between apolipoprotein E alleles and olivopontocerebellar atrophy. Journal of the Neurological Sciences. 158(1). 110–112. 3 indexed citations
15.
Ueno, Shuichi, Keiji Kondoh, Osamu Komure, et al.. (1995). Somatic mosaicism of CAG repeat in dentatorubral-pallidoluysian atrophy (DRPLA). Human Molecular Genetics. 4(4). 663–666. 63 indexed citations
16.
Komure, Osamu, Akira Sano, Naoki Nishino, et al.. (1995). DNA analysis in hereditary dentatorubral-pallidoluysian atrophy. Neurology. 45(1). 143–149. 106 indexed citations
17.
Kawai, Jun, Masakiyo Sasahara, Fumitada Hazama, et al.. (1993). Pallidonigroluysian degeneration with iron deposition: a study of three autopsy cases. Acta Neuropathologica. 86(6). 609–616. 15 indexed citations
18.
Hashimoto, Takeshi, Noboru Kitamura, Naoaki Saito, et al.. (1992). The loss of βII-protein kinase C in the striatum from patients with Huntington's disease. Brain Research. 585(1-2). 303–306. 10 indexed citations
19.
Sakai, Norio, Masashi Sasa, Kumatoshi Ishihara, et al.. (1991). Effects of l-threo-DOPS, a noradrenaline precursor, on the long-term potentiation in the rat hippocampal mossy fiber-CA3 region. Brain Research. 567(2). 267–273. 11 indexed citations
20.
Komure, Osamu, et al.. (1985). Intra-axonal polysaccharide deposits in the peripheral nerve seen in adult polysaccharide storage myopathy. Acta Neuropathologica. 65(3-4). 300–304. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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