Yuki Kagoya

2.2k total citations · 1 hit paper
53 papers, 1.6k citations indexed

About

Yuki Kagoya is a scholar working on Oncology, Immunology and Molecular Biology. According to data from OpenAlex, Yuki Kagoya has authored 53 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Oncology, 23 papers in Immunology and 21 papers in Molecular Biology. Recurrent topics in Yuki Kagoya's work include CAR-T cell therapy research (22 papers), Immune Cell Function and Interaction (20 papers) and Acute Myeloid Leukemia Research (16 papers). Yuki Kagoya is often cited by papers focused on CAR-T cell therapy research (22 papers), Immune Cell Function and Interaction (20 papers) and Acute Myeloid Leukemia Research (16 papers). Yuki Kagoya collaborates with scholars based in Japan, Canada and United States. Yuki Kagoya's co-authors include Tingxi Guo, Kayoko Saso, Mark Anczurowski, Marcus O. Butler, Naoto Hirano, Chung-Hsi Wang, Mineo Kurokawa, Mark D. Minden, Shinya Tanaka and Akihide Yoshimi and has published in prestigious journals such as Nucleic Acids Research, Journal of Clinical Investigation and Nature Medicine.

In The Last Decade

Yuki Kagoya

50 papers receiving 1.6k citations

Hit Papers

A novel chimeric antigen receptor containing a JAK–STAT s... 2018 2026 2020 2023 2018 100 200 300 400

Peers

Yuki Kagoya
Leo D. Wang United States
Francis Mussai United Kingdom
Armen Mardiros United States
Robert Berahovich United States
Ai-Hong Zhang United States
Sijie Lu United States
Leo D. Wang United States
Yuki Kagoya
Citations per year, relative to Yuki Kagoya Yuki Kagoya (= 1×) peers Leo D. Wang

Countries citing papers authored by Yuki Kagoya

Since Specialization
Citations

This map shows the geographic impact of Yuki Kagoya's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yuki Kagoya with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yuki Kagoya more than expected).

Fields of papers citing papers by Yuki Kagoya

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yuki Kagoya. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yuki Kagoya. The network helps show where Yuki Kagoya may publish in the future.

Co-authorship network of co-authors of Yuki Kagoya

This figure shows the co-authorship network connecting the top 25 collaborators of Yuki Kagoya. A scholar is included among the top collaborators of Yuki Kagoya based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yuki Kagoya. Yuki Kagoya is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Matsukawa, Tetsuya, Toshiaki Yoshikawa, Yusuke Ito, et al.. (2025). Efficient expansion of tumor-infiltrating lymphocytes from gynecologic cancer. The Journal of Immunology. 214(12). 3618–3633.
2.
Ito, Yusuke, Noriko Nakamura, Toshiaki Yoshikawa, et al.. (2025). Plasma membrane-coated nanoparticles and membrane vesicles to orchestrate multimodal antitumor immunity. Journal for ImmunoTherapy of Cancer. 13(1). e010005–e010005. 4 indexed citations
4.
Nakashima, Takahiro & Yuki Kagoya. (2024). Current progress of CAR-T-cell therapy for patients with multiple myeloma. International Journal of Hematology. 120(1). 15–22. 1 indexed citations
5.
Nakahara, Fumio, Yusuke Ito, Yosuke Masamoto, et al.. (2023). AKT2 inhibition accelerates the acquisition of phagocytic ability in induced pluripotent stem cell–derived neutrophils. Experimental Hematology. 130. 104137–104137. 1 indexed citations
6.
Kagoya, Yuki. (2023). Introduction: Synthetic Biology for Cancer Immunology Special Issue. International Immunology. 36(2). 45–48.
7.
Kagoya, Yuki. (2023). Cytokine signaling in chimeric antigen receptor T-cell therapy. International Immunology. 36(2). 49–56. 14 indexed citations
8.
Sugata, Kenji, Yukiko Matsunaga, Yūki Yamashita, et al.. (2021). Affinity-matured HLA class II dimers for robust staining of antigen-specific CD4+ T cells. Nature Biotechnology. 39(8). 958–967. 14 indexed citations
9.
Ito, Yusuke, Fumio Nakahara, Yuki Kagoya, & Mineo Kurokawa. (2021). CD62L expression level determines the cell fate of myeloid progenitors. Stem Cell Reports. 16(12). 2871–2886. 11 indexed citations
10.
Kagoya, Yuki, Tingxi Guo, Brian Yeung, et al.. (2020). Genetic Ablation of HLA Class I, Class II, and the T-cell Receptor Enables Allogeneic T Cells to Be Used for Adoptive T-cell Therapy. Cancer Immunology Research. 8(7). 926–936. 126 indexed citations
11.
Kagoya, Yuki, Hiroshi Saijo, Yukiko Matsunaga, et al.. (2018). Arginine methylation of FOXP3 is crucial for the suppressive function of regulatory T cells. Journal of Autoimmunity. 97. 10–21. 38 indexed citations
12.
Kagoya, Yuki, Munehide Nakatsugawa, Kayoko Saso, et al.. (2018). DOT1L inhibition attenuates graft-versus-host disease by allogeneic T cells in adoptive immunotherapy models. Nature Communications. 9(1). 1915–1915. 22 indexed citations
13.
Kagoya, Yuki, Shinya Tanaka, Tingxi Guo, et al.. (2018). A novel chimeric antigen receptor containing a JAK–STAT signaling domain mediates superior antitumor effects. Nature Medicine. 24(3). 352–359. 446 indexed citations breakdown →
14.
Kagoya, Yuki, Munehide Nakatsugawa, Toshiki Ochi, et al.. (2017). Transient stimulation expands superior antitumor T cells for adoptive therapy. JCI Insight. 2(2). e89580–e89580. 44 indexed citations
15.
Yamashita, Yūki, Mark Anczurowski, Munehide Nakatsugawa, et al.. (2017). HLA-DP84Gly constitutively presents endogenous peptides generated by the class I antigen processing pathway. Nature Communications. 8(1). 15244–15244. 29 indexed citations
16.
Kagoya, Yuki, Munehide Nakatsugawa, Yūki Yamashita, et al.. (2016). BET bromodomain inhibition enhances T cell persistence and function in adoptive immunotherapy models. Journal of Clinical Investigation. 126(9). 3479–3494. 168 indexed citations
17.
Nakatsugawa, Munehide, Yūki Yamashita, Toshiki Ochi, et al.. (2016). CD4+ and CD8+ TCRβ repertoires possess different potentials to generate extraordinarily high-avidity T cells. Scientific Reports. 6(1). 23821–23821. 12 indexed citations
18.
Morita, Ken, Yosuke Masamoto, Keisuke Kataoka, et al.. (2015). BAALC potentiates oncogenic ERK pathway through interactions with MEKK1 and KLF4. Leukemia. 29(11). 2248–2256. 29 indexed citations
19.
Kagoya, Yuki, Keisuke Kataoka, Akihide Yoshimi, et al.. (2015). Targeted gene correction of RUNX1 in induced pluripotent stem cells derived from familial platelet disorder with propensity to myeloid malignancy restores normal megakaryopoiesis. Experimental Hematology. 43(10). 849–857. 35 indexed citations
20.
Kagoya, Yuki, Tsuyoshi Takahashi, Yasuhito Nannya, et al.. (2011). Hyperbilirubinemia after hematopoietic stem cell transplantation: comparison of clinical and pathologic findings in 41 autopsied cases. Clinical Transplantation. 25(5). E552–7. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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