Yang Ou

2.6k total citations · 2 hit papers
48 papers, 2.0k citations indexed

About

Yang Ou is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Yang Ou has authored 48 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Molecular Biology, 10 papers in Oncology and 10 papers in Cancer Research. Recurrent topics in Yang Ou's work include Epigenetics and DNA Methylation (8 papers), Platelet Disorders and Treatments (7 papers) and Ubiquitin and proteasome pathways (5 papers). Yang Ou is often cited by papers focused on Epigenetics and DNA Methylation (8 papers), Platelet Disorders and Treatments (7 papers) and Ubiquitin and proteasome pathways (5 papers). Yang Ou collaborates with scholars based in China, United States and Pakistan. Yang Ou's co-authors include Wei Gu, Dawei Li, Shang-Jui Wang, Bo Chu, Le Jiang, Yingming Zhao, Yue Chen, Bin Zheng, Shuang Yang and Wei Gu and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Yang Ou

42 papers receiving 2.0k citations

Hit Papers

Activation of SAT1 engages polyamine metabolism with p53-... 2016 2026 2019 2022 2016 2016 200 400 600

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yang Ou China 20 1.3k 902 837 406 208 48 2.0k
Mei Yi China 21 1.4k 1.1× 808 0.9× 279 0.3× 442 1.1× 150 0.7× 49 2.1k
Xiaowu Xu China 23 1.0k 0.8× 629 0.7× 393 0.5× 531 1.3× 226 1.1× 82 1.7k
Shouping Xu China 27 1.7k 1.3× 1.5k 1.6× 562 0.7× 290 0.7× 170 0.8× 59 2.3k
Xia Luo China 20 936 0.7× 697 0.8× 454 0.5× 619 1.5× 135 0.6× 48 1.9k
Yi Bao China 22 1.2k 0.9× 787 0.9× 304 0.4× 385 0.9× 92 0.4× 44 1.7k
Ludovica Ciuffreda Italy 21 1.4k 1.0× 549 0.6× 373 0.4× 668 1.6× 136 0.7× 41 2.0k
Lintao Liu China 14 879 0.7× 925 1.0× 534 0.6× 584 1.4× 148 0.7× 29 2.1k
Xiao Tan China 20 975 0.7× 440 0.5× 315 0.4× 222 0.5× 105 0.5× 53 1.4k
Huamei Tang China 34 2.1k 1.6× 1.2k 1.4× 361 0.4× 656 1.6× 201 1.0× 83 2.9k
Yi Shen United States 26 1.4k 1.1× 858 1.0× 345 0.4× 238 0.6× 144 0.7× 49 2.0k

Countries citing papers authored by Yang Ou

Since Specialization
Citations

This map shows the geographic impact of Yang Ou's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yang Ou with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yang Ou more than expected).

Fields of papers citing papers by Yang Ou

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yang Ou. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yang Ou. The network helps show where Yang Ou may publish in the future.

Co-authorship network of co-authors of Yang Ou

This figure shows the co-authorship network connecting the top 25 collaborators of Yang Ou. A scholar is included among the top collaborators of Yang Ou based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yang Ou. Yang Ou is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
He, Yu, Jing Wang, Y. Li, et al.. (2025). Device‐Measured Physical Activity, Sedentary Behaviour and Risk of Chronic Kidney Diseases Across Levels of Grip Strength. Journal of Cachexia Sarcopenia and Muscle. 16(1). e13726–e13726.
2.
Ou, Yang, Huayu Hu, Lixia Cao, et al.. (2025). USP51/GRP78/ABCB1 axis confers chemoresistance through decreasing doxorubicin accumulation in triple-negative breast cancer cells. Acta Pharmaceutica Sinica B. 15(5). 2593–2611.
3.
Ou, Yang, Kun Zhang, Huayu Hu, et al.. (2024). Abstract 4556: USP51-GRP78-ABCB1 axis promotes chemoresistance of triple negative breast cancer by decreasing the accumulation of doxorubicin in cells. Cancer Research. 84(6_Supplement). 4556–4556. 1 indexed citations
5.
Ou, Yang. (2024). The Zeb1-Cxcl1 axis impairs the antitumor immune response by inducing M2 macrophage polarization in breast cancer. American Journal of Cancer Research. 14(9). 4378–4397. 2 indexed citations
6.
Xu, Pengcheng, Ying Li, Xibing Zhuang, et al.. (2024). Changes in immune subsets during chemotherapy as prognosis biomarkers for multiple myeloma patients by longitudinal monitoring. Immunologic Research. 72(5). 1185–1197.
7.
Wang, Min, Yang Ou, Ben Niu, et al.. (2024). Heterogeneously elevated branched-chain/aromatic amino acids among new-onset type-2 diabetes mellitus patients are potentially skewed diabetes predictors. World Journal of Diabetes. 15(1). 53–71. 8 indexed citations
8.
Li, Jianjun, Xuechun Xiao, Yang Ou, et al.. (2023). USP51/PD‐L1/ITGB1‐deployed juxtacrine interaction plays a cell‐intrinsic role in promoting chemoresistant phenotypes in non‐small cell lung cancer. Cancer Communications. 43(7). 765–787. 23 indexed citations
9.
Ou, Yang, et al.. (2023). Case report: Noonan syndrome with protein-losing enteropathy. Frontiers in Genetics. 14. 1237821–1237821.
11.
Wang, Zhaoyang, Yu Liu, Shipeng Li, et al.. (2022). Hypoxia inducible factor 1α promotes interleukin-1 receptor antagonist expression during hepatic ischemia-reperfusion injury. World Journal of Gastroenterology. 28(38). 5573–5588. 6 indexed citations
12.
Jiang, Huimin, Huimin Wei, Hang Wang, et al.. (2022). Zeb1-induced metabolic reprogramming of glycolysis is essential for macrophage polarization in breast cancer. Cell Death and Disease. 13(3). 206–206. 118 indexed citations
13.
Jiang, Huimin, Chen Zhou, Qiong Wang, et al.. (2020). Jagged1-Notch1-deployed tumor perivascular niche promotes breast cancer stem cell phenotype through Zeb1. Nature Communications. 11(1). 5129–5129. 76 indexed citations
14.
Kon, Ning, Yang Ou, Shang-Jui Wang, et al.. (2020). mTOR inhibition acts as an unexpected checkpoint in p53-mediated tumor suppression. Genes & Development. 35(1-2). 59–64. 41 indexed citations
15.
Deng, Kaiyuan, Liang Liu, Zhen Zhang, et al.. (2020). WIP1 promotes cancer stem cell properties by inhibiting p38 MAPK in NSCLC. Signal Transduction and Targeted Therapy. 5(1). 36–36. 41 indexed citations
16.
Zhao, Rongrong, Wenting Xu, Le Huu Song, et al.. (2020). Helicobacter pylori infection leads to KLF4 inactivation in gastric cancer through a TET1‐mediated DNA methylation mechanism. Cancer Medicine. 9(7). 2551–2563. 24 indexed citations
17.
Li, Dawei, Yang Ou, Le Jiang, et al.. (2016). Acetylation Is Crucial for p53-Mediated Ferroptosis and Tumor Suppression. Cell Reports. 17(2). 366–373. 364 indexed citations breakdown →
18.
Ou, Yang, Shang-Jui Wang, Le Jiang, Bin Zheng, & Wei Gu. (2014). p53 Protein-mediated Regulation of Phosphoglycerate Dehydrogenase (PHGDH) Is Crucial for the Apoptotic Response upon Serine Starvation. Journal of Biological Chemistry. 290(1). 457–466. 112 indexed citations
19.
Shen, Che-Hung, Ping Yuan, Rolando Pérez‐Lorenzo, et al.. (2013). Phosphorylation of BRAF by AMPK Impairs BRAF-KSR1 Association and Cell Proliferation. Molecular Cell. 52(2). 161–172. 108 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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