Shang-Jui Wang
Impact in
- Cancer Research top 0.5%
- Cancer, Lipids, and Metabolism
- Cancer-related molecular mechanisms research
- Cancer, Hypoxia, and Metabolism
- Pulmonary and Respiratory Medicine top 0.5%
- Ferroptosis and cancer prognosis
Papers in
- Oncology 16
- Cancer-related Molecular Pathways 6
- Cancer Immunotherapy and Biomarkers 4
-
- Cancer, Hypoxia, and Metabolism 3
Shang-Jui Wang
27 papers receiving 3.9k citations
Hit Papers
Peers
Comparison fields: 5 of 105
- Cancer Research 2.1k
- Pulmonary and Respiratory Medicine 2.5k
- Molecular Biology 2.7k
- Oncology 721
- Biochemistry 132
Countries citing papers authored by Shang-Jui Wang
This map shows the geographic impact of Shang-Jui Wang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shang-Jui Wang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shang-Jui Wang more than expected).
Fields of papers citing papers by Shang-Jui Wang
This network shows the impact of papers produced by Shang-Jui Wang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shang-Jui Wang. The network helps show where Shang-Jui Wang may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Shang-Jui Wang, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2025 | 2 | |
| 3 | 2024 | 1 | |
| 4 | 2024 | 5 | |
| 5 | 2024 | 2 | |
| 6 | 2023 | 3 | |
| 7 | 2022 | 1 | |
| 8 | 2022 | 7 | |
| 9 | 2021 | 2 | |
| 10 | 2020 | 41 | |
| 11 | 2020 | 9 | |
| 12 | 2018 | 21 | |
| 13 | Ferroptosis as a p53-mediated activity during tumour suppression Hit paper breakdown → | 2015 | 2549 |
| 14 | Ferroptosis: a missing piece of puzzle in the p53 blueprint? | 2015 | 1 |
| 15 | 2015 | 187 | |
| 16 | 2014 | 112 | |
| 17 | 2013 | 44 | |
| 18 | 2013 | 45 | |
| 19 | 2011 | 104 | |
| 20 | 2008 | 101 |
About Shang-Jui Wang
Shang-Jui Wang is a scholar working on Oncology, Cancer Research, Pulmonary and Respiratory Medicine, Radiation and Geriatrics and Gerontology, having authored 29 papers that have together received 4.0k indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (6 papers), Epigenetics and DNA Methylation (5 papers), Renal cell carcinoma treatment (4 papers), Cancer Immunotherapy and Biomarkers (4 papers), Ferroptosis and cancer prognosis (4 papers), RNA modifications and cancer (4 papers), Prostate Cancer Treatment and Research (4 papers) and Cancer, Hypoxia, and Metabolism (3 papers). The work is most often cited by research in Cancer Research (2.1k citations), Pulmonary and Respiratory Medicine (2.5k citations), Molecular Biology (2.7k citations), Oncology (721 citations) and Biochemistry (132 citations). Shang-Jui Wang has collaborated with scholars based in United States, United Kingdom and Slovakia. Frequent co-authors include Wei Gu, Le Jiang, Ning Kon, Tongyuan Li, Tao Su, Richard Baer, Hanina Hibshoosh, Yang Ou, Dawei Li and Bo Chu. Their work appears in journals such as Cell Cycle, Journal of Biological Chemistry, International Journal of Radiation Oncology*Biology*Physics, Annals of Palliative Medicine and Molecular Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.