William P. Gilks
- Neurology top 2%
- Molecular Biology
- Cellular and Molecular Neuroscience top 10%
- Neurology top 5%
- Physiology
- Co-authors
- Patrick M. Abou‐SleimanNicholas WoodAndrew J. LeesDaniel G. HealySonia GandhiJanice L. HoltonTamás RévészKailash P. Bhatia
- Topics
- Parkinson's Disease Mechanisms and Treatments (5 papers)Genetic Associations and Epidemiology (3 papers)Neurological diseases and metabolism (3 papers)
- Journals
- The LancetPhilosophical Transactions of the Royal Society B Biological SciencesJournal of Neurochemistry
- Partner nations
- United KingdomIrelandSweden
In The Last Decade
William P. Gilks
9 papers receiving 864 citations
Hit Papers
Peers
Comparison fields: 5 of 81
- Neurology 616
- Molecular Biology 312
- Cellular and Molecular Neuroscience 265
- Neurology 221
- Physiology 186
Countries citing papers authored by William P. Gilks
This map shows the geographic impact of William P. Gilks's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William P. Gilks with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William P. Gilks more than expected).
Fields of papers citing papers by William P. Gilks
This network shows the impact of papers produced by William P. Gilks. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William P. Gilks. The network helps show where William P. Gilks may publish in the future.
Co-authorship network of co-authors of William P. Gilks
This figure shows the co-authorship network connecting the top 25 collaborators of William P. Gilks. A scholar is included among the top collaborators of William P. Gilks based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William P. Gilks. William P. Gilks is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 77 | |
| 2 | 11 | |
| 3 | 36 | |
| 4 | 16 | |
| 5 | 138 | |
| 6 | A common LRRK2 mutation in idiopathic Parkinson's diseasebreakdown → | 575 |
| 7 | 4 | |
| 8 | 2 | |
| 9 | DJ-1 mutations in Parkinson's disease. | 20 |
About William P. Gilks
William P. Gilks is a scholar working on Neurology, Neurology and Genetics, having authored 9 papers that have together received 879 indexed citations. Recurring topics across this work include Parkinson's Disease Mechanisms and Treatments (5 papers), Genetic Associations and Epidemiology (3 papers) and Neurological diseases and metabolism (3 papers). The work is most often cited by research in Neurology (616 citations), Neurology (221 citations) and Cellular and Molecular Neuroscience (265 citations). William P. Gilks has collaborated with scholars based in United Kingdom, Ireland and Sweden. Frequent co-authors include Patrick M. Abou‐Sleiman, Nicholas Wood, Andrew J. Lees, Daniel G. Healy, Sonia Gandhi, Janice L. Holton, Tamás Révész, Kailash P. Bhatia, Andrew Singleton and Vincenzo Bonifati. Their work appears in journals such as The Lancet, Philosophical Transactions of the Royal Society B Biological Sciences and Journal of Neurochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.