William B. Mair

10.5k total citations · 1 hit paper
51 papers, 7.8k citations indexed

About

William B. Mair is a scholar working on Aging, Molecular Biology and Endocrine and Autonomic Systems. According to data from OpenAlex, William B. Mair has authored 51 papers receiving a total of 7.8k indexed citations (citations by other indexed papers that have themselves been cited), including 41 papers in Aging, 25 papers in Molecular Biology and 19 papers in Endocrine and Autonomic Systems. Recurrent topics in William B. Mair's work include Genetics, Aging, and Longevity in Model Organisms (41 papers), Circadian rhythm and melatonin (19 papers) and Mitochondrial Function and Pathology (8 papers). William B. Mair is often cited by papers focused on Genetics, Aging, and Longevity in Model Organisms (41 papers), Circadian rhythm and melatonin (19 papers) and Mitochondrial Function and Pathology (8 papers). William B. Mair collaborates with scholars based in United States, United Kingdom and Switzerland. William B. Mair's co-authors include Linda Partridge, Andrew Dillin, Matthew D. W. Piper, Kristopher Burkewitz, Reuben J. Shaw, Scott D. Pletcher, Caroline Heintz, James A. J. Fitzpatrick, Aashish Joshi and Rebecca A. Kohnz and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

William B. Mair

49 papers receiving 7.7k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Phosphorylation of ULK1 (hATG1) by AMP-Activated Protein Kinase Connects Energy Sensing to Mitophagy

2010 2.0k citations David B. Shackelford, Maria M. Mihaylova et al. Science profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
William B. Mair United States	35	3.9k	2.7k	2.1k	1.8k	1.1k	51	7.8k
Malene Hansen United States	39	4.7k 1.2×	3.5k 1.3×	1.9k 0.9×	3.7k 2.1×	1.0k 0.9×	61	9.8k
Kevin Flurkey United States	29	2.9k 0.8×	3.1k 1.1×	2.9k 1.4×	709 0.4×	996 0.9×	57	7.0k
Rozalyn M. Anderson United States	36	2.9k 0.7×	2.3k 0.8×	4.0k 1.9×	1.1k 0.6×	823 0.7×	77	7.8k
Nancy L. Nadon United States	23	3.1k 0.8×	2.7k 1.0×	2.4k 1.1×	722 0.4×	870 0.8×	43	7.0k
Heidi A. Tissenbaum United States	36	4.8k 1.2×	6.6k 2.4×	2.9k 1.4×	602 0.3×	2.3k 2.0×	47	10.1k
Clinton M. Astle United States	34	3.2k 0.8×	2.7k 1.0×	2.6k 1.2×	720 0.4×	879 0.8×	57	7.6k
David W. Walker United States	35	2.6k 0.7×	1.7k 0.6×	1.1k 0.5×	929 0.5×	453 0.4×	77	5.5k
Adam B. Salmon United States	36	2.5k 0.6×	1.5k 0.6×	2.0k 1.0×	512 0.3×	482 0.4×	91	5.3k
Z. Dave Sharp United States	27	4.4k 1.1×	2.2k 0.8×	1.9k 0.9×	634 0.4×	708 0.6×	68	7.3k
Warren Ladiges United States	39	3.7k 1.0×	1.1k 0.4×	1.7k 0.8×	721 0.4×	293 0.3×	159	6.5k

Countries citing papers authored by William B. Mair

Since Specialization

Citations

This map shows the geographic impact of William B. Mair's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William B. Mair with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William B. Mair more than expected).

Fields of papers citing papers by William B. Mair

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William B. Mair. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William B. Mair. The network helps show where William B. Mair may publish in the future.

Co-authorship network of co-authors of William B. Mair

This figure shows the co-authorship network connecting the top 25 collaborators of William B. Mair. A scholar is included among the top collaborators of William B. Mair based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William B. Mair. William B. Mair is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Heintz, Caroline, Ayse Sena Mutlu, Mary Piper, et al.. (2025). The efficacy of longevity interventions in Caenorhabditis elegans is determined by the early life activity of RNA splicing factors. PLoS Biology. 23(11). e3003504–e3003504.

Valera‐Alberni, Miriam, et al.. (2024). Novel imaging tools to study mitochondrial morphology inCaenorhabditis elegans. Life Science Alliance. 7(11). e202402918–e202402918. 1 indexed citations

Silva-García, Carlos G., Katharina Papsdorf, Caroline Heintz, et al.. (2023). The CRTC-1 transcriptional domain is required for COMPASS complex-mediated longevity in C. elegans. Nature Aging. 3(11). 1358–1371. 2 indexed citations

Valera‐Alberni, Miriam & William B. Mair. (2023). Fast fragmenting mitochondria by TORC2. Nature Cell Biology. 25(7). 926–927.

Papsdorf, Katharina, Jason W. Miklas, Matías Cabruja, et al.. (2023). Lipid droplets and peroxisomes are co-regulated to drive lifespan extension in response to mono-unsaturated fatty acids. Nature Cell Biology. 25(5). 672–684. 73 indexed citations

Nwanaji‐Enwerem, Jamaji C. & William B. Mair. (2022). Redefining age-based screening and diagnostic guidelines: an opportunity for biological aging clocks in clinical medicine?. The Lancet Healthy Longevity. 3(6). e376–e377. 2 indexed citations

Liu, Yasmine J., Rebecca L. McIntyre, Georges E. Janssens, et al.. (2020). Mitochondrial translation and dynamics synergistically extend lifespan in C. elegans through HLH-30. The Journal of Cell Biology. 219(6). 36 indexed citations

Sharma, Arpit, et al.. (2020). Metabolic Communication and Healthy Aging: Where Should We Focus Our Energy?. Developmental Cell. 54(2). 196–211. 56 indexed citations

Weir, Heather J., Pallas Yao, Frank K. Huynh, et al.. (2017). Dietary Restriction and AMPK Increase Lifespan via Mitochondrial Network and Peroxisome Remodeling. Cell Metabolism. 26(6). 884–896.e5. 275 indexed citations

10.

Weir, Heather J. & William B. Mair. (2016). SnapShot: Neuronal Regulation of Aging. Cell. 166(3). 784–784.e1. 5 indexed citations

11.

Burkewitz, Kristopher, Ianessa Morantte, Heather J. Weir, et al.. (2015). Neuronal CRTC-1 Governs Systemic Mitochondrial Metabolism and Lifespan via a Catecholamine Signal. Cell. 160(5). 842–855. 139 indexed citations

12.

Hwang, Ara B., Murat Artan, Hsin‐Wen Chang, et al.. (2014). Feedback regulation via AMPK and HIF-1 mediates ROS-dependent longevity in Caenorhabditis elegans. Proceedings of the National Academy of Sciences. 111(42). E4458–67. 155 indexed citations

13.

Hine, Christopher, Eylul Harputlugil, Yue Zhang, et al.. (2014). Endogenous Hydrogen Sulfide Production Is Essential for Dietary Restriction Benefits. Cell. 160(1-2). 132–144. 425 indexed citations

14.

Heintz, Caroline & William B. Mair. (2014). You Are What You Host: Microbiome Modulation of the Aging Process. Cell. 156(3). 408–411. 163 indexed citations

15.

Shackelford, David B., Maria M. Mihaylova, Sara Gelino, et al.. (2010). Phosphorylation of ULK1 (hATG1) by AMP-Activated Protein Kinase Connects Energy Sensing to Mitophagy. Science. 331(6016). 456–461. 2048 indexed citations breakdown →

16.

Capelson, Maya, Yun Xiang Liang, Roberta Schulte, et al.. (2010). Chromatin-Bound Nuclear Pore Components Regulate Gene Expression in Higher Eukaryotes. Cell. 140(3). 372–383. 351 indexed citations

17.

Mair, William B., Siler H. Panowski, Reuben J. Shaw, & Andrew Dillin. (2009). Optimizing Dietary Restriction for Genetic Epistasis Analysis and Gene Discovery in C. elegans. PLoS ONE. 4(2). e4535–e4535. 62 indexed citations

18.

Hulbert, A. J., David J. Clancy, William B. Mair, et al.. (2004). Metabolic rate is not reduced by dietary-restriction or by lowered insulin/IGF-1 signalling and is not correlated with individual lifespan in Drosophila melanogaster. Experimental Gerontology. 39(8). 1137–1143. 120 indexed citations

19.

Mair, William B., Carla M. Sgrò, Alice Johnson, Tracey Chapman, & Linda Partridge. (2004). Lifespan extension by dietary restriction in female Drosophila melanogaster is not caused by a reduction in vitellogenesis or ovarian activity. Experimental Gerontology. 39(7). 1011–1019. 79 indexed citations

20.

Mair, William B., Patrick Goymer, Scott D. Pletcher, & Linda Partridge. (2003). Demography of Dietary Restriction and Death in Drosophila. Science. 301(5640). 1731–1733. 403 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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