Wei Xiang

4.5k total citations
84 papers, 2.7k citations indexed

About

Wei Xiang is a scholar working on Molecular Biology, Neurology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Wei Xiang has authored 84 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Molecular Biology, 31 papers in Neurology and 15 papers in Cellular and Molecular Neuroscience. Recurrent topics in Wei Xiang's work include Parkinson's Disease Mechanisms and Treatments (30 papers), Prion Diseases and Protein Misfolding (11 papers) and Neurological diseases and metabolism (9 papers). Wei Xiang is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (30 papers), Prion Diseases and Protein Misfolding (11 papers) and Neurological diseases and metabolism (9 papers). Wei Xiang collaborates with scholars based in Germany, China and United States. Wei Xiang's co-authors include Jürgen Winkler, Hans A. Kretzschmar, Jochen Klucken, Beate Winner, Iryna Prots, Otto Windl, Eliezer Masliah, Reinhold Baumgartner, Florian Krach and Johannes C. M. Schlachetzki and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Blood and PLoS ONE.

In The Last Decade

Wei Xiang

80 papers receiving 2.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Wei Xiang Germany 29 1.2k 1.0k 666 616 553 84 2.7k
Philipp Albrecht Germany 33 1.1k 0.9× 956 0.9× 381 0.6× 267 0.4× 584 1.1× 162 3.7k
Ralph Lucius Germany 31 1.3k 1.1× 584 0.6× 1.0k 1.6× 458 0.7× 693 1.3× 75 3.4k
Lajos László Hungary 27 1.8k 1.5× 386 0.4× 705 1.1× 633 1.0× 336 0.6× 74 2.7k
Daniela Rossi Italy 30 2.4k 2.0× 649 0.6× 1.3k 1.9× 795 1.3× 1.5k 2.7× 65 4.7k
Anil G. Cashikar United States 20 2.5k 2.2× 886 0.9× 377 0.6× 1.1k 1.7× 551 1.0× 32 3.8k
Stefan Aigner United States 27 1.7k 1.5× 1.0k 1.0× 320 0.5× 812 1.3× 676 1.2× 60 3.5k
Daniela Boassa United States 31 2.9k 2.5× 722 0.7× 380 0.6× 655 1.1× 513 0.9× 49 4.5k
István A. Krizbai Hungary 37 1.6k 1.4× 285 0.3× 1.7k 2.5× 514 0.8× 534 1.0× 110 4.4k
Kenneth S. Shindler United States 32 1.8k 1.5× 467 0.5× 446 0.7× 294 0.5× 552 1.0× 101 3.8k
Qinwen Mao United States 24 2.5k 2.2× 506 0.5× 376 0.6× 727 1.2× 719 1.3× 102 4.0k

Countries citing papers authored by Wei Xiang

Since Specialization
Citations

This map shows the geographic impact of Wei Xiang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wei Xiang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wei Xiang more than expected).

Fields of papers citing papers by Wei Xiang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wei Xiang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wei Xiang. The network helps show where Wei Xiang may publish in the future.

Co-authorship network of co-authors of Wei Xiang

This figure shows the co-authorship network connecting the top 25 collaborators of Wei Xiang. A scholar is included among the top collaborators of Wei Xiang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wei Xiang. Wei Xiang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Hoffmann, Lutz, S O'Brien, Sonja Plötz, et al.. (2025). Vimentin network dysregulation mediates neurite deficits in SNCA duplication Parkinson’s patient–derived midbrain neurons. Science Advances. 11(23). eadq2742–eadq2742.
2.
Li, Bin, Wei Xiang, Xiaohui Dou, et al.. (2025). Coarse-Grained Molecular Dynamics Simulation of Nucleation and Stability of Electrochemically Generated Nanobubbles. Langmuir. 41(13). 8497–8509. 3 indexed citations
3.
Hoffmann, Lutz, Eva-Maria Eckl, Marleen Bérouti, et al.. (2025). AMPylation Regulates 5′-3′ Exonuclease PLD3 Processing. Molecular & Cellular Proteomics. 24(10). 101051–101051.
4.
Xiang, Wei, et al.. (2023). Co3V2O8 nanoparticle-assembled porous sphere as a new electrocatalyst for sensitive nonenzymatic sensing of H2O2. Colloids and Surfaces A Physicochemical and Engineering Aspects. 665. 131224–131224. 1 indexed citations
5.
Xiang, Wei, Haifeng Yao, & Yongping Dong. (2023). Ionic liquid modified SiO2 nanoparticles as coreactant of Ru(bpy)32+ ECL and its application in the detection of glutathione. Journal of Photochemistry and Photobiology A Chemistry. 444. 114975–114975. 8 indexed citations
6.
Xiang, Wei, Haifeng Yao, Shang-Bing Wang, & Yongping Dong. (2023). Enhancing effect of black phosphorus quantum dots on electrochemiluminescence of Ir(dF(CF3)ppy)2(bpy) in ionic liquid medium and its sensing application. Journal of Electroanalytical Chemistry. 941. 117522–117522. 9 indexed citations
7.
Distler, Jörg H. W., Anja Schulze-Krebs, Ivanna K. Timotius, et al.. (2023). Young human alpha synuclein transgenic (BAC-SNCA) mice display sex- and gene-dose-dependent phenotypic disturbances. Behavioural Brain Research. 460. 114781–114781. 2 indexed citations
8.
Schmitt, Verena, Patrick Süß, Stephan P. Rosshart, et al.. (2023). Gut-to-brain spreading of pathology in synucleinopathies: A focus on molecular signalling mediators. Behavioural Brain Research. 452. 114574–114574. 7 indexed citations
10.
Xiang, Wei, et al.. (2022). Alpha-Synuclein and Its Role in Melanocytes. Cells. 11(13). 2087–2087. 4 indexed citations
11.
Drobny, Alice, Sonja Plötz, Tomas Koudelka, et al.. (2022). Interaction of Alpha Synuclein and Microtubule Organization Is Linked to Impaired Neuritic Integrity in Parkinson’s Patient-Derived Neuronal Cells. International Journal of Molecular Sciences. 23(3). 1812–1812. 17 indexed citations
12.
Schmitt, Verena, Iryna Prots, Beate Winner, et al.. (2022). Translocation of Distinct Alpha Synuclein Species from the Nucleus to Neuronal Processes during Neuronal Differentiation. Biomolecules. 12(8). 1108–1108. 8 indexed citations
13.
Regensburger, Martin, Sebastian Zundler, Friederike Zunke, et al.. (2022). The Quest for Anti-α-Synuclein Antibody Specificity—Lessons Learnt From Flow Cytometry Analysis. Frontiers in Neurology. 13. 869103–869103. 2 indexed citations
14.
Hartlage‐Rübsamen, Maike, Isabel Hilbrich, Max Holzer, et al.. (2021). A glutaminyl cyclase-catalyzed α-synuclein modification identified in human synucleinopathies. Acta Neuropathologica. 142(3). 399–421. 19 indexed citations
15.
Sommer, Annika, Franz Marxreiter, Florian Krach, et al.. (2018). Th17 Lymphocytes Induce Neuronal Cell Death in a Human iPSC-Based Model of Parkinson’s Disease. Cell stem cell. 23(1). 123–131.e6. 253 indexed citations
16.
Ni, Juan, et al.. (2018). Amide-linked local anesthetics preferentially target leukemia stem cell through inhibition of Wnt/β-catenin. Biochemical and Biophysical Research Communications. 503(2). 956–962. 20 indexed citations
17.
Wang, Ying, Shenggang Sun, Suiqiang Zhu, et al.. (2016). Analysis of pramipexole dose–response relationships in Parkinson's disease. Drug Design Development and Therapy. Volume11. 83–89. 8 indexed citations
18.
Xiang, Wei, Bee Ling Ng, Ralph M. Bunte, et al.. (2015). Inhibition of isoprenylcysteine carboxylmethyltransferase augments BCR-ABL1 tyrosine kinase inhibition-induced apoptosis in chronic myeloid leukemia. Experimental Hematology. 44(3). 189–193.e2. 11 indexed citations
19.
Xiang, Wei, Philipp Spitzer, Verena May, et al.. (2014). Autophagy modulates SNCA/α-synuclein release, thereby generating a hostile microenvironment. Autophagy. 10(12). 2171–2192. 181 indexed citations
20.
Xiang, Wei, Johannes C. M. Schlachetzki, Stefan Helling, et al.. (2013). Oxidative stress-induced posttranslational modifications of alpha-synuclein: Specific modification of alpha-synuclein by 4-hydroxy-2-nonenal increases dopaminergic toxicity. Molecular and Cellular Neuroscience. 54. 71–83. 119 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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