Wayne A. Cabral
About
Wayne A. Cabral is a scholar working on Genetics, Rheumatology and Molecular Biology.
According to data from OpenAlex, Wayne A. Cabral has authored 55 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Genetics, 26 papers in Rheumatology and 22 papers in Molecular Biology. Recurrent topics in Wayne A. Cabral's work include Connective tissue disorders research (41 papers), Bone and Dental Protein Studies (21 papers) and Collagen: Extraction and Characterization (7 papers). Wayne A. Cabral is often cited by papers focused on Connective tissue disorders research (41 papers), Bone and Dental Protein Studies (21 papers) and Collagen: Extraction and Characterization (7 papers). Wayne A. Cabral collaborates with scholars based in United States, Italy and Canada. Wayne A. Cabral's co-authors include Joan C. Marini, Aileen M. Barnes, Antonella Forlino, Sergey Leikin, Elena Makareeva, Weizhong Chang, David R. Eyre, MaryAnn Weis, Natalia V. Kuznetsova and Edward L. Mertz and has published in prestigious journals such as New England Journal of Medicine, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.
In The Last Decade
Wayne A. Cabral
53 papers receiving 2.9k citations
Hit Papers
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late) cites · hero ref
| Name | h | Career | Trend | Papers | Cites | |||||
|---|---|---|---|---|---|---|---|---|---|---|
| Wayne A. Cabral United States | 29 | 2.1k | 1.2k | 1.0k | 498 | 462 | 55 | 3.0k | ||
| Aileen M. Barnes United States | 24 | 1.8k 0.9× | 1.0k 0.9× | 802 0.8× | 434 0.9× | 383 0.8× | 34 | 2.6k | ||
| MaryAnn Weis United States | 24 | 1.3k 0.6× | 748 0.6× | 998 1.0× | 407 0.8× | 321 0.7× | 40 | 2.4k | ||
| Noriyuki Tsumaki Japan | 34 | 586 0.3× | 1.4k 1.1× | 2.0k 2.0× | 414 0.8× | 367 0.8× | 90 | 3.7k | ||
| Motomi Enomoto‐Iwamoto United States | 43 | 863 0.4× | 1.9k 1.6× | 2.6k 2.6× | 532 1.1× | 541 1.2× | 84 | 4.7k | ||
| Sofie Symoens Belgium | 30 | 1.6k 0.8× | 575 0.5× | 952 0.9× | 285 0.6× | 256 0.6× | 82 | 2.4k | ||
| F M Pope United Kingdom | 39 | 3.4k 1.6× | 1.1k 1.0× | 1.3k 1.3× | 400 0.8× | 497 1.1× | 138 | 4.9k | ||
| Michael D. Briggs United Kingdom | 35 | 1.3k 0.6× | 1.3k 1.1× | 1.6k 1.6× | 396 0.8× | 312 0.7× | 75 | 3.4k | ||
| Jacky Bonaventure France | 35 | 2.7k 1.3× | 1.2k 1.0× | 3.4k 3.4× | 492 1.0× | 821 1.8× | 94 | 5.8k | ||
| Richard W. Kang United States | 28 | 556 0.3× | 1.1k 0.9× | 972 1.0× | 203 0.4× | 290 0.6× | 60 | 3.9k | ||
| Rosa Serra United States | 40 | 1.6k 0.7× | 980 0.8× | 3.4k 3.4× | 415 0.8× | 758 1.6× | 88 | 5.2k |
Countries citing papers authored by Wayne A. Cabral
Since
Specialization
Citations
This map shows the geographic impact of Wayne A. Cabral's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wayne A. Cabral with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wayne A. Cabral more than expected).
Fields of papers citing papers by Wayne A. Cabral
Since
Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences
This network shows the impact of papers produced by Wayne A. Cabral. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wayne A. Cabral. The network helps show where Wayne A. Cabral may publish in the future.
Co-authorship network of co-authors of Wayne A. Cabral
This figure shows the co-authorship network connecting the top 25 collaborators of Wayne A. Cabral. A scholar is included among the top collaborators of Wayne A. Cabral based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wayne A. Cabral. Wayne A. Cabral is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
1.
Cabral, Wayne A., Urraca Tavarez, Michael R. Erdos, et al.. (2024). Angiopoietin‐2 reverses endothelial cell dysfunction in progeria vasculature. Aging Cell. 24(2). e14375–e14375.
2.
Cabral, Wayne A., Masahiko Terajima, Urraca Tavarez, et al.. (2023). Bone dysplasia in Hutchinson‐Gilford progeria syndrome is associated with dysregulated differentiation and function of bone cell populations. Aging Cell. 22(9). e13903–e13903.
3.
Erdos, Michael R., Wayne A. Cabral, Urraca Tavarez, et al.. (2021). A targeted antisense therapeutic approach for Hutchinson–Gilford progeria syndrome. Nature Medicine. 27(3). 536–545.
4.
Terajima, Masahiko, Yuki Taga, Wayne A. Cabral, et al.. (2019). Cyclophilin B control of lysine post-translational modifications of skin type I collagen. PLoS Genetics. 15(6). e1008196–e1008196.
5.
Barnes, Aileen M., Aarthi Ashok, Elena Makareeva, et al.. (2019). COL1A1 C-propeptide mutations cause ER mislocalization of procollagen and impair C-terminal procollagen processing. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1865(9). 2210–2223.
6.
Ben‐Gedalya, Tziona, Lorna Moll, Michal Bejerano‐Sagie, et al.. (2015). Alzheimer's disease‐causing proline substitutions lead to presenilin 1 aggregation and malfunction. The EMBO Journal. 34(22). 2820–2839.
7.
Reich, Adi, Aileen M. Barnes, Wayne A. Cabral, et al.. (2014). Type V OI Primary Osteoblasts Display Increased Mineralization Despite DecreasedCOL1A1Expression. The Journal of Clinical Endocrinology & Metabolism. 100(2). E325–E332.
8.
Boskey, Adele L., Kostas Verdelis, Lyudmila Spevak, et al.. (2013). Mineral and Matrix Changes in Brtl/+Teeth Provide Insights into Mineralization Mechanisms. BioMed Research International. 2013. 1–9.
9.
Chang, Weizhong, Aileen M. Barnes, Wayne A. Cabral, Joann Bodurtha, & Joan C. Marini. (2009). Prolyl 3-hydroxylase 1 and CRTAP are mutually stabilizing in the endoplasmic reticulum collagen prolyl 3-hydroxylation complex. Human Molecular Genetics. 19(2). 223–234.
10.
Giudici, Camilla, Nicolas Raynal, Hanna Wiedemann, et al.. (2008). Mapping of SPARC/BM-40/Osteonectin-binding Sites on Fibrillar Collagens. Journal of Biological Chemistry. 283(28). 19551–19560.
11.
Cabral, Wayne A., Weizhong Chang, Aileen M. Barnes, et al.. (2007). Prolyl 3-hydroxylase 1 deficiency causes a recessive metabolic bone disorder resembling lethal/severe osteogenesis imperfecta. Nature Genetics. 39(3). 359–365.
12.
Makareeva, Elena, Edward L. Mertz, Natalia V. Kuznetsova, et al.. (2007). Structural Heterogeneity of Type I Collagen Triple Helix and Its Role in Osteogenesis Imperfecta. Journal of Biological Chemistry. 283(8). 4787–4798.
13.
Makareeva, Elena, Wayne A. Cabral, Joan C. Marini, & Sergey Leikin. (2006). Molecular Mechanism of α1(I)-Osteogenesis Imperfecta/Ehlers-Danlos Syndrome. Journal of Biological Chemistry. 281(10). 6463–6470.
14.
Barnes, Aileen M., Weizhong Chang, Roy Morello, et al.. (2006). Deficiency of Cartilage-Associated Protein in Recessive Lethal Osteogenesis Imperfecta. New England Journal of Medicine. 355(26). 2757–2764.
15.
Walker, Lary C., Marcia Willing, Joan C. Marini, et al.. (2004). Heterogeneous basis of the type VIB form of Ehlers–Danlos syndrome (EDS VIB) that is unrelated to decreased collagen lysyl hydroxylation. American Journal of Medical Genetics Part A. 131A(2). 155–162.
16.
Kuznetsova, Natalia V., Antonella Forlino, Wayne A. Cabral, Joan C. Marini, & Sergey Leikin. (2004). Structure, stability and interactions of type I collagen with GLY349-CYS substitution in α1(I) chain in a murine Osteogenesis Imperfecta model. Matrix Biology. 23(2). 101–112.
17.
Cabral, Wayne A. & Joan C. Marini. (2004). High Proportion of Mutant Osteoblasts Is Compatible with Normal Skeletal Function in Mosaic Carriers of Osteogenesis Imperfecta**Presented as a platform presentation at the National Meeting of the American Society of Bone and Mineral Research (Late-Breaking Research Session), San Antonio, TX, September 2002.. The American Journal of Human Genetics. 74(4). 752–760.
18.
Cabral, Wayne A., Elena Makareeva, Alain Colige, et al.. (2003). Type I Collagen Triplet Duplication Mutation in Lethal Osteogenesis Imperfecta Shifts Register of α Chains throughout the Helix and Disrupts Incorporation of Mutant Helices into Fibrils and Extracellular Matrix. Journal of Biological Chemistry. 278(12). 10006–10012.
19.
Cabral, Wayne A., et al.. (2001). G76E Substitution in Type I Collagen Is the First Nonlethal Glutamic Acid Substitution in the α1(I) Chain and Alters Folding of the N-terminal End of the Helix. Molecular Genetics and Metabolism. 72(4). 326–335.
20.
Bouma, Peter, Wayne A. Cabral, William G. Cole, & Joan C. Marini. (2001). COL5A1 Exon 14 Splice Acceptor Mutation Causes a Functional Null Allele, Haploinsufficiency of α1(V) and Abnormal Heterotypic Interstitial Fibrils in Ehlers-Danlos Syndrome II. Journal of Biological Chemistry. 276(16). 13356–13364.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
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