Aleksandra Ivovic

424 total citations
8 papers, 150 citations indexed

About

Aleksandra Ivovic is a scholar working on Rheumatology, Genetics and Molecular Biology. According to data from OpenAlex, Aleksandra Ivovic has authored 8 papers receiving a total of 150 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Rheumatology, 5 papers in Genetics and 2 papers in Molecular Biology. Recurrent topics in Aleksandra Ivovic's work include Dermatological and Skeletal Disorders (5 papers), Hypertrophic osteoarthropathy and related conditions (3 papers) and Heterotopic Ossification and Related Conditions (3 papers). Aleksandra Ivovic is often cited by papers focused on Dermatological and Skeletal Disorders (5 papers), Hypertrophic osteoarthropathy and related conditions (3 papers) and Heterotopic Ossification and Related Conditions (3 papers). Aleksandra Ivovic collaborates with scholars based in United States, Austria and Germany. Aleksandra Ivovic's co-authors include Richard M. Siegel, Zuoming Deng, Heeseog Kang, Nadja Fratzl‐Zelman, Klaus Klaushofer, Joan C. Marini, Smita Jha, Paul Roschger, Timothy Bhattacharyya and James D. Katz and has published in prestigious journals such as Nature Communications, The Journal of Experimental Medicine and The Journal of Immunology.

In The Last Decade

Aleksandra Ivovic

6 papers receiving 149 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Aleksandra Ivovic United States 6 90 87 43 30 16 8 150
Maha Faden Saudi Arabia 6 100 1.1× 22 0.3× 93 2.2× 20 0.7× 31 1.9× 9 178
Shuba Krishna United States 4 22 0.2× 60 0.7× 86 2.0× 17 0.6× 19 1.2× 9 121
Ali Awaji Saudi Arabia 4 170 1.9× 18 0.2× 178 4.1× 19 0.6× 27 1.7× 6 265
Daphne Heijsman Netherlands 7 57 0.6× 18 0.2× 138 3.2× 9 0.3× 14 0.9× 11 221
Nilay Güneş Türkiye 7 68 0.8× 22 0.3× 79 1.8× 23 0.8× 12 0.8× 25 126
Kati Tarkkonen Finland 8 24 0.3× 18 0.2× 158 3.7× 13 0.4× 47 2.9× 11 199
Terhi Rantamäki Finland 10 128 1.4× 16 0.2× 37 0.9× 12 0.4× 27 1.7× 12 209
Enjun Yang United States 7 48 0.5× 107 1.2× 71 1.7× 5 0.2× 50 3.1× 12 301
Marie Ravoet Belgium 7 40 0.4× 64 0.7× 33 0.8× 5 0.2× 27 1.7× 11 172
Eveline W Blom Netherlands 5 84 0.9× 21 0.2× 94 2.2× 9 0.3× 28 1.8× 8 156

Countries citing papers authored by Aleksandra Ivovic

Since Specialization
Citations

This map shows the geographic impact of Aleksandra Ivovic's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Aleksandra Ivovic with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Aleksandra Ivovic more than expected).

Fields of papers citing papers by Aleksandra Ivovic

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Aleksandra Ivovic. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Aleksandra Ivovic. The network helps show where Aleksandra Ivovic may publish in the future.

Co-authorship network of co-authors of Aleksandra Ivovic

This figure shows the co-authorship network connecting the top 25 collaborators of Aleksandra Ivovic. A scholar is included among the top collaborators of Aleksandra Ivovic based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Aleksandra Ivovic. Aleksandra Ivovic is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Koeppen, Katja, Katrin Fundel‐Clemens, Chantelle Simone-Roach, et al.. (2025). iPSC-derived macrophages: An in vitro model to study human disease-relevant macrophage biology. The Journal of Immunology. 214(11). 3115–3130.
2.
Kang, Heeseog, Smita Jha, Aleksandra Ivovic, et al.. (2020). Somatic SMAD3-activating mutations cause melorheostosis by up-regulating the TGF-β/SMAD pathway. The Journal of Experimental Medicine. 217(5). 28 indexed citations
3.
Donlin, Laura T., Sung Ho Park, Ευγενία Γιαννοπούλου, et al.. (2019). Insights into rheumatic diseases from next-generation sequencing. Nature Reviews Rheumatology. 15(6). 327–339. 23 indexed citations
4.
Fratzl‐Zelman, Nadja, Paul Roschger, Heeseog Kang, et al.. (2019). Somatic activating mutations in MAP2K1 cause melorheostosis with highly porous lesions covered by compact periosteal bone. Osteologie/Osteology.
5.
Fratzl‐Zelman, Nadja, Paul Roschger, Heeseog Kang, et al.. (2019). Melorheostotic Bone Lesions Caused by Somatic Mutations in MAP2K1 Have Deteriorated Microarchitecture and Periosteal Reaction. Journal of Bone and Mineral Research. 34(5). 883–895. 14 indexed citations
6.
Milne, Paul, Anastasia Resteu, Aleksandra Ivovic, et al.. (2019). AB0184 FLOW CYTOMETRIC IMMUNOPHENOTYPING OF SALIVARY GLANDS IN PRIMARY SJÖGREN'S SYNDROME. Annals of the Rheumatic Diseases. 78. 1550–1550. 13 indexed citations
7.
Jha, Smita, Nadja Fratzl‐Zelman, Paul Roschger, et al.. (2018). Distinct Clinical and Pathological Features of Melorheostosis Associated With Somatic MAP2K1 Mutations. Journal of Bone and Mineral Research. 34(1). 145–156. 23 indexed citations
8.
Kang, Heeseog, Smita Jha, Zuoming Deng, et al.. (2018). Somatic activating mutations in MAP2K1 cause melorheostosis. Nature Communications. 9(1). 1390–1390. 49 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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