Thomas E. Uveges

908 total citations
9 papers, 670 citations indexed

About

Thomas E. Uveges is a scholar working on Genetics, Oncology and Genetics. According to data from OpenAlex, Thomas E. Uveges has authored 9 papers receiving a total of 670 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Genetics, 4 papers in Oncology and 2 papers in Genetics. Recurrent topics in Thomas E. Uveges's work include Connective tissue disorders research (4 papers), Bone health and treatments (2 papers) and Corneal Surgery and Treatments (2 papers). Thomas E. Uveges is often cited by papers focused on Connective tissue disorders research (4 papers), Bone health and treatments (2 papers) and Corneal Surgery and Treatments (2 papers). Thomas E. Uveges collaborates with scholars based in United States, Italy and Israel. Thomas E. Uveges's co-authors include Joan C. Marini, Wayne A. Cabral, Clemens Bergwitz, Antonella Forlino, Kenneth M. Kozloff, Steven A. Goldstein, Amy J. Wagoner Johnson, Alla Danilkovitch, Brendan Lee and Natalia V. Kuznetsova and has published in prestigious journals such as New England Journal of Medicine, Proceedings of the National Academy of Sciences and Journal of Neuroscience.

In The Last Decade

Thomas E. Uveges

9 papers receiving 634 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Thomas E. Uveges United States 9 413 199 190 129 106 9 670
Hans E. H. Pruijs Netherlands 12 436 1.1× 207 1.0× 240 1.3× 86 0.7× 100 0.9× 16 800
Laura Tonachini Italy 12 382 0.9× 230 1.2× 282 1.5× 39 0.3× 111 1.0× 18 746
Caressa Lietman United States 14 521 1.3× 389 2.0× 391 2.1× 81 0.6× 160 1.5× 19 910
Benjamin P. Sinder United States 16 247 0.6× 170 0.9× 330 1.7× 204 1.6× 216 2.0× 23 856
MaryAnn Weis United States 10 581 1.4× 324 1.6× 331 1.7× 98 0.8× 131 1.2× 11 841
David Cholok United States 17 245 0.6× 575 2.9× 213 1.1× 72 0.6× 41 0.4× 30 842
Kimberly A. Jacobsen United States 6 159 0.4× 93 0.5× 393 2.1× 160 1.2× 171 1.6× 7 884
Joohyun Lim United States 13 233 0.6× 143 0.7× 524 2.8× 116 0.9× 160 1.5× 14 782
Shawna M. Pyott United States 9 546 1.3× 295 1.5× 263 1.4× 52 0.4× 121 1.1× 9 789
Kengo Shimono United States 10 203 0.5× 379 1.9× 272 1.4× 47 0.4× 61 0.6× 11 733

Countries citing papers authored by Thomas E. Uveges

Since Specialization
Citations

This map shows the geographic impact of Thomas E. Uveges's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas E. Uveges with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas E. Uveges more than expected).

Fields of papers citing papers by Thomas E. Uveges

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas E. Uveges. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas E. Uveges. The network helps show where Thomas E. Uveges may publish in the future.

Co-authorship network of co-authors of Thomas E. Uveges

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas E. Uveges. A scholar is included among the top collaborators of Thomas E. Uveges based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas E. Uveges. Thomas E. Uveges is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
Johnson, Amy J. Wagoner, et al.. (2015). Retention of Endogenous Viable Cells Enhances the Anti-Inflammatory Activity of Cryopreserved Amnion. Advances in Wound Care. 4(9). 523–533. 54 indexed citations
2.
Uveges, Thomas E., et al.. (2015). Angiogenic Potential of Cryopreserved Amniotic Membrane Is Enhanced Through Retention of All Tissue Components in Their Native State. Advances in Wound Care. 4(9). 513–522. 45 indexed citations
3.
4.
Uveges, Thomas E., Patricia Collin‐Osdoby, Wayne A. Cabral, et al.. (2008). Cellular Mechanism of Decreased Bone in Brtl Mouse Model of OI: Imbalance of Decreased Osteoblast Function and Increased Osteoclasts and Their Precursors. Journal of Bone and Mineral Research. 23(12). 1983–1994. 69 indexed citations
5.
Barnes, Aileen M., Weizhong Chang, Roy Morello, et al.. (2006). Deficiency of Cartilage-Associated Protein in Recessive Lethal Osteogenesis Imperfecta. New England Journal of Medicine. 355(26). 2757–2764. 228 indexed citations
6.
Miura, Yasuo, Masako Miura, Stan Gronthos, et al.. (2005). Defective osteogenesis of the stromal stem cells predisposes CD18-null mice to osteoporosis. Proceedings of the National Academy of Sciences. 102(39). 14022–14027. 60 indexed citations
7.
Kozloff, Kenneth M., Angela Carden, Clemens Bergwitz, et al.. (2004). Brittle IV Mouse Model for Osteogenesis Imperfecta IV Demonstrates Postpubertal Adaptations to Improve Whole Bone Strength. Journal of Bone and Mineral Research. 19(4). 614–622. 98 indexed citations
8.
Uveges, Thomas E., et al.. (2002). Intron 1 Is Required for Cell Type-Specific, But Not Injury-Responsive, Peripherin Gene Expression. Journal of Neuroscience. 22(18). 7959–7967. 9 indexed citations
9.
Allen, Donald L., et al.. (1997). Cell-Specific Induction of c-fos Expression in the Pituitary Gland by Estrogen*. Endocrinology. 138(5). 2128–2135. 55 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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