Wanlong Lei

953 total citations
18 papers, 657 citations indexed

About

Wanlong Lei is a scholar working on Cellular and Molecular Neuroscience, Neurology and Molecular Biology. According to data from OpenAlex, Wanlong Lei has authored 18 papers receiving a total of 657 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Cellular and Molecular Neuroscience, 9 papers in Neurology and 3 papers in Molecular Biology. Recurrent topics in Wanlong Lei's work include Neuroscience and Neuropharmacology Research (9 papers), Neurological disorders and treatments (7 papers) and Parkinson's Disease Mechanisms and Treatments (6 papers). Wanlong Lei is often cited by papers focused on Neuroscience and Neuropharmacology Research (9 papers), Neurological disorders and treatments (7 papers) and Parkinson's Disease Mechanisms and Treatments (6 papers). Wanlong Lei collaborates with scholars based in China, United States and Spain. Wanlong Lei's co-authors include Anton Reiner, Nobel Del Mar, Yun Jiao, Yunping Deng, Lisi OuYang, Yuxin Ma, Jia‐Jia Wu, Jiachang Chen, Yu Jia and Y.P. Deng and has published in prestigious journals such as Journal of Neuroscience, Experimental Neurology and Behavioural Brain Research.

In The Last Decade

Wanlong Lei

17 papers receiving 651 citations

Peers

Wanlong Lei
Matthew S. Thimgan United States
Derek R. Verley United States
Jodi Becker United States
Bruce A. Chase United States
Allan‐Hermann Pool United States
Wanlong Lei
Citations per year, relative to Wanlong Lei Wanlong Lei (= 1×) peers Caihong Ji

Countries citing papers authored by Wanlong Lei

Since Specialization
Citations

This map shows the geographic impact of Wanlong Lei's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wanlong Lei with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wanlong Lei more than expected).

Fields of papers citing papers by Wanlong Lei

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wanlong Lei. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wanlong Lei. The network helps show where Wanlong Lei may publish in the future.

Co-authorship network of co-authors of Wanlong Lei

This figure shows the co-authorship network connecting the top 25 collaborators of Wanlong Lei. A scholar is included among the top collaborators of Wanlong Lei based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wanlong Lei. Wanlong Lei is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Sun, Liping, Xuefeng Zheng, Ye Zhang, et al.. (2024). Morphological changes in perisynaptic astrocytes induced by dopamine neuronal degeneration in the striatum of rats. Heliyon. 10(6). e27637–e27637.
2.
Huang, Ziyun, Liping Sun, Xuefeng Zheng, et al.. (2023). A neural tract tracing study on synaptic connections for cortical glutamatergic terminals and cervical spinal calretinin neurons in rats. Frontiers in Neural Circuits. 17. 1086873–1086873. 1 indexed citations
3.
Zheng, Xuefeng, Liping Sun, Bingbing Liu, et al.. (2021). Morphological Study of the Cortical and Thalamic Glutamatergic Synaptic Inputs of Striatal Parvalbumin Interneurons in Rats. Neurochemical Research. 46(7). 1659–1673. 4 indexed citations
4.
Zheng, Xuefeng, Ziyun Huang, Bingbing Liu, et al.. (2019). Increase in Glutamatergic Terminals in the Striatum Following Dopamine Depletion in a Rat Model of Parkinson’s Disease. Neurochemical Research. 44(5). 1079–1089. 17 indexed citations
5.
Liu, Bingbing, Xuefeng Zheng, Jia‐Jia Wu, et al.. (2019). Partial�decortication ameliorates dopamine depletion‑induced striatal neuron lesions in rats. International Journal of Molecular Medicine. 44(4). 1414–1424. 3 indexed citations
6.
Zheng, Xuefeng, Jia‐Jia Wu, Si Chen, et al.. (2018). A Comparative study for striatal-direct and -indirect pathway neurons to DA depletion-induced lesion in a PD rat model. Neurochemistry International. 118. 14–22. 6 indexed citations
7.
Wei, Jia‐You, Cuicui Liu, Handong Ouyang, et al.. (2017). Activation of RAGE/STAT3 pathway by methylglyoxal contributes to spinal central sensitization and persistent pain induced by bortezomib. Experimental Neurology. 296. 74–82. 28 indexed citations
8.
Luo, Shiqi, Lisi OuYang, Jie Wei, et al.. (2017). Neuronal Apoptosis: Pathological Basis of Behavioral Dysfunctions Induced by Angiostrongylus cantonensis in Rodents Model. Korean Journal of Parasitology. 55(3). 267–278. 11 indexed citations
9.
Wei, Jie, Fengshi Wu, Xin Zeng, et al.. (2015). Microglia activation: one of the checkpoints in the CNS inflammation caused by Angiostrongylus cantonensis infection in rodent model. Parasitology Research. 114(9). 3247–3254. 21 indexed citations
10.
Deng, Yunping, José L. Lanciego, Lydia Kerkerian‐Le Goff, et al.. (2015). Differential organization of cortical inputs to striatal projection neurons of the matrix compartment in rats. Frontiers in Systems Neuroscience. 9. 51–51. 34 indexed citations
11.
Jia, Yu, Shi‐Jing Mo, Lisi OuYang, et al.. (2014). EPO-Dependent Activation of PI3K/Akt/FoxO3a Signalling Mediates Neuroprotection in In Vitro and In Vivo Models of Parkinson’s Disease. Journal of Molecular Neuroscience. 53(1). 117–124. 64 indexed citations
12.
Ma, Yuxin, Lisi OuYang, Si Chen, et al.. (2014). The effects of unilateral 6-OHDA lesion in medial forebrain bundle on the motor, cognitive dysfunctions and vulnerability of different striatal interneuron types in rats. Behavioural Brain Research. 266. 37–45. 61 indexed citations
13.
Long, Houqing, et al.. (2013). Does diffusion tensor data reflect pathological changes in the spinal cord with chronic injury.. PubMed. 8(36). 3382–90. 7 indexed citations
14.
OuYang, Lisi, Jie Wei, Zhongdao Wu, et al.. (2012). Differences of larval development and pathological changes in permissive and nonpermissive rodent hosts for Angiostrongylus cantonensis infection. Parasitology Research. 111(4). 1547–1557. 41 indexed citations
15.
Deng, Y.P., et al.. (2007). Differential localization of the GluR1 and GluR2 subunits of the AMPA-type glutamate receptor among striatal neuron types in rats. Journal of Chemical Neuroanatomy. 33(4). 167–192. 41 indexed citations
16.
Laverghetta, Antonio, Robert C. Foehring, Y.P. Deng, et al.. (2006). Single-cell RT-PCR, in situ hybridization histochemical, and immunohistochemical studies of substance P and enkephalin co-occurrence in striatal projection neurons in rats. Journal of Chemical Neuroanatomy. 31(3). 178–199. 31 indexed citations
17.
Deng, Yunping, Wanlong Lei, & Anton Reiner. (2006). Differential perikaryal localization in rats of D1 and D2 dopamine receptors on striatal projection neuron types identified by retrograde labeling. Journal of Chemical Neuroanatomy. 32(2-4). 101–116. 90 indexed citations
18.
Lei, Wanlong, Yun Jiao, Nobel Del Mar, & Anton Reiner. (2004). Evidence for Differential Cortical Input to Direct Pathway versus Indirect Pathway Striatal Projection Neurons in Rats. Journal of Neuroscience. 24(38). 8289–8299. 197 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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