W. David Merryman

5.7k total citations
87 papers, 3.4k citations indexed

About

W. David Merryman is a scholar working on Cardiology and Cardiovascular Medicine, Surgery and Pulmonary and Respiratory Medicine. According to data from OpenAlex, W. David Merryman has authored 87 papers receiving a total of 3.4k indexed citations (citations by other indexed papers that have themselves been cited), including 58 papers in Cardiology and Cardiovascular Medicine, 24 papers in Surgery and 22 papers in Pulmonary and Respiratory Medicine. Recurrent topics in W. David Merryman's work include Cardiac Valve Diseases and Treatments (44 papers), Cardiomyopathy and Myosin Studies (14 papers) and Aortic Disease and Treatment Approaches (12 papers). W. David Merryman is often cited by papers focused on Cardiac Valve Diseases and Treatments (44 papers), Cardiomyopathy and Myosin Studies (14 papers) and Aortic Disease and Treatment Approaches (12 papers). W. David Merryman collaborates with scholars based in United States, Canada and Australia. W. David Merryman's co-authors include Michael S. Sacks, Joshua D. Hutcheson, David E. Schmidt, Alison K. Schroer, Vincent Setola, Joseph Chen, Larisa Ryzhova, Richard A. Hopkins, Frederick J. Schoen and Mary Kathryn Sewell-Loftin and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and Journal of Clinical Investigation.

In The Last Decade

W. David Merryman

86 papers receiving 3.3k citations

Peers

W. David Merryman
Padmini Sarathchandra United Kingdom
Maria A. Rupnick United States
Jessica E. Wagenseil United States
Joshua D. Hutcheson United States
Sanjay Sinha United Kingdom
Najma Latif United Kingdom
Padmini Sarathchandra United Kingdom
W. David Merryman
Citations per year, relative to W. David Merryman W. David Merryman (= 1×) peers Padmini Sarathchandra

Countries citing papers authored by W. David Merryman

Since Specialization
Citations

This map shows the geographic impact of W. David Merryman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by W. David Merryman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites W. David Merryman more than expected).

Fields of papers citing papers by W. David Merryman

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by W. David Merryman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by W. David Merryman. The network helps show where W. David Merryman may publish in the future.

Co-authorship network of co-authors of W. David Merryman

This figure shows the co-authorship network connecting the top 25 collaborators of W. David Merryman. A scholar is included among the top collaborators of W. David Merryman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with W. David Merryman. W. David Merryman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Choudhary, Dharmendra, Abigail C. Neininger, Alaina H. Willet, et al.. (2025). Nonmuscle α-Actinin-4 Couples Sarcomere Function to Cardiac Remodeling. Circulation Research. 138(1). e326412–e326412.
2.
Bender, Aaron M., et al.. (2023). Identification of Potent, Selective, and Peripherally Restricted Serotonin Receptor 2B Antagonists from a High-Throughput Screen. Assay and Drug Development Technologies. 21(3). 89–96. 5 indexed citations
3.
Usselman, Charlotte W., Merry L. Lindsey, Austin T. Robinson, et al.. (2023). Guidelines on the use of sex and gender in cardiovascular research. American Journal of Physiology-Heart and Circulatory Physiology. 326(1). H238–H255. 34 indexed citations
4.
Riley, Lance A., et al.. (2022). Sclerostin ablation prevents aortic valve stenosis in mice. American Journal of Physiology-Heart and Circulatory Physiology. 323(5). H1037–H1047. 6 indexed citations
5.
Riley, Lance A., et al.. (2022). Loss of talin in cardiac fibroblasts results in augmented ventricular cardiomyocyte hypertrophy in response to pressure overload. American Journal of Physiology-Heart and Circulatory Physiology. 322(5). H857–H866. 8 indexed citations
6.
Riley, Lance A., Matthew R. Bersi, Prachi Umbarkar, et al.. (2021). Targeting 5-HT 2B Receptor Signaling Prevents Border Zone Expansion and Improves Microstructural Remodeling After Myocardial Infarction. Circulation. 143(13). 1317–1330. 42 indexed citations
7.
Riley, Lance A., et al.. (2021). Impaired macrophage trafficking and increased helper T-cell recruitment with loss of cadherin-11 in atherosclerotic immune response. American Journal of Physiology-Heart and Circulatory Physiology. 321(4). H756–H769. 10 indexed citations
8.
Lindman, Brian R., Devraj Sukul, Marc R. Dweck, et al.. (2021). Evaluating Medical Therapy for Calcific Aortic Stenosis. Journal of the American College of Cardiology. 78(23). 2354–2376. 52 indexed citations
9.
Huffstater, Tessa, Matthew R. Bersi, Bradley I. Reinfeld, et al.. (2020). Macrophages Promote Aortic Valve Cell Calcification and Alter STAT3 Splicing. Arteriosclerosis Thrombosis and Vascular Biology. 40(6). e153–e165. 37 indexed citations
10.
Bersi, Matthew R., et al.. (2020). Genetic ablation of serotonin receptor 2B improves aortic valve hemodynamics of Notch1 heterozygous mice in a high-cholesterol diet model. PLoS ONE. 15(11). e0238407–e0238407. 13 indexed citations
11.
Schroer, Alison K., Matthew R. Bersi, Qinkun Zhang, et al.. (2019). Cadherin-11 blockade reduces inflammation-driven fibrotic remodeling and improves outcomes after myocardial infarction. JCI Insight. 4(18). 31 indexed citations
12.
Lindman, Brian R., et al.. (2019). Celecoxib Is Associated With Dystrophic Calcification and Aortic Valve Stenosis. JACC Basic to Translational Science. 4(2). 135–143. 18 indexed citations
13.
Chen, Joseph, Larisa Ryzhova, Mary Kathryn Sewell-Loftin, et al.. (2015). Notch1 Mutation Leads to Valvular Calcification Through Enhanced Myofibroblast Mechanotransduction. Arteriosclerosis Thrombosis and Vascular Biology. 35(7). 1597–1605. 43 indexed citations
14.
Schroer, Alison K. & W. David Merryman. (2015). Mechanobiology of myofibroblast adhesion in fibrotic cardiac disease. Journal of Cell Science. 128(10). 1865–1875. 112 indexed citations
15.
Fredi, Joseph L., Michael N. Young, Douglas M. Dumont, et al.. (2015). Quantitative Imaging Assessment of an Alternative Approach to Surgical Mitral Valve Leaflet Resection: An Acute Porcine Study. Annals of Biomedical Engineering. 44(7). 2240–2250. 1 indexed citations
16.
Chen, Joseph, Beth A. Drzewiecki, W. David Merryman, & John C. Pope. (2013). Murine bladder wall biomechanics following partial bladder obstruction. Journal of Biomechanics. 46(15). 2752–2755. 18 indexed citations
17.
Hutcheson, Joshua D., Larisa Ryzhova, Vincent Setola, & W. David Merryman. (2012). 5-HT2B antagonism arrests non-canonical TGF-β1-induced valvular myofibroblast differentiation. Journal of Molecular and Cellular Cardiology. 53(5). 707–714. 82 indexed citations
18.
Fisher, Charles I., Joseph Chen, & W. David Merryman. (2012). Calcific nodule morphogenesis by heart valve interstitial cells is strain dependent. Biomechanics and Modeling in Mechanobiology. 12(1). 5–17. 88 indexed citations
19.
Parekh, Aron, Nazanin S. Ruppender, Kevin M. Branch, et al.. (2011). Sensing and Modulation of Invadopodia across a Wide Range of Rigidities. Biophysical Journal. 100(3). 573–582. 95 indexed citations
20.
Stella, John A., Jun Liao, Yi Hong, et al.. (2008). Tissue-to-cellular level deformation coupling in cell micro-integrated elastomeric scaffolds. Biomaterials. 29(22). 3228–3236. 65 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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