Prachi Umbarkar

960 total citations
22 papers, 706 citations indexed

About

Prachi Umbarkar is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Surgery. According to data from OpenAlex, Prachi Umbarkar has authored 22 papers receiving a total of 706 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Cardiology and Cardiovascular Medicine, 13 papers in Molecular Biology and 7 papers in Surgery. Recurrent topics in Prachi Umbarkar's work include Cardiac Fibrosis and Remodeling (12 papers), Wnt/β-catenin signaling in development and cancer (5 papers) and Peptidase Inhibition and Analysis (4 papers). Prachi Umbarkar is often cited by papers focused on Cardiac Fibrosis and Remodeling (12 papers), Wnt/β-catenin signaling in development and cancer (5 papers) and Peptidase Inhibition and Analysis (4 papers). Prachi Umbarkar collaborates with scholars based in United States, India and Canada. Prachi Umbarkar's co-authors include Hind Lal, Anand Prakash Singh, Sultan Tousif, Thomas Force, Manisha Gupte, Qinkun Zhang, Yuanjun Guo, Sandhya L. Sitasawad, Cristi L. Galindo and Sathiyanarayanan Lohidasan and has published in prestigious journals such as Circulation, Circulation Research and Free Radical Biology and Medicine.

In The Last Decade

Prachi Umbarkar

22 papers receiving 699 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Prachi Umbarkar United States 15 363 302 100 86 64 22 706
Aimilia Varela Greece 16 483 1.3× 284 0.9× 131 1.3× 127 1.5× 57 0.9× 33 902
Anne Aries France 13 460 1.3× 232 0.8× 101 1.0× 119 1.4× 60 0.9× 21 718
Shufang Zhao China 12 352 1.0× 265 0.9× 76 0.8× 70 0.8× 51 0.8× 23 634
Andreas Jungmann Germany 15 565 1.6× 323 1.1× 85 0.8× 79 0.9× 93 1.5× 33 959
Jingzang Tao United States 5 545 1.5× 341 1.1× 159 1.6× 74 0.9× 114 1.8× 7 970
Shoko Sugiyama Japan 11 356 1.0× 205 0.7× 123 1.2× 89 1.0× 70 1.1× 15 671
Simona Gallo Italy 13 384 1.1× 191 0.6× 114 1.1× 60 0.7× 70 1.1× 24 716
Urszula Mackiewicz Poland 16 354 1.0× 491 1.6× 39 0.4× 70 0.8× 54 0.8× 59 890
Pascal Trouvé France 17 384 1.1× 236 0.8× 144 1.4× 46 0.5× 46 0.7× 37 795
Jianfen Guo United States 16 535 1.5× 206 0.7× 48 0.5× 67 0.8× 76 1.2× 20 831

Countries citing papers authored by Prachi Umbarkar

Since Specialization
Citations

This map shows the geographic impact of Prachi Umbarkar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Prachi Umbarkar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Prachi Umbarkar more than expected).

Fields of papers citing papers by Prachi Umbarkar

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Prachi Umbarkar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Prachi Umbarkar. The network helps show where Prachi Umbarkar may publish in the future.

Co-authorship network of co-authors of Prachi Umbarkar

This figure shows the co-authorship network connecting the top 25 collaborators of Prachi Umbarkar. A scholar is included among the top collaborators of Prachi Umbarkar based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Prachi Umbarkar. Prachi Umbarkar is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Umbarkar, Prachi, et al.. (2025). Fibroblast-specific MyD88-dependent signaling aggravates inflammation and cardiac dysfunction in the MI heart. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1871(3). 167703–167703. 1 indexed citations
2.
Gupte, Manisha, et al.. (2024). Animal models of haploinsufficiency revealed the isoform-specific role of GSK-3 in HFD-induced obesity and glucose intolerance. American Journal of Physiology-Cell Physiology. 327(6). C1349–C1358. 1 indexed citations
3.
Patil, Mallikarjun, Praveen Kumar Dubey, Sultan Tousif, et al.. (2024). Fibroblast-Specific Depletion of Human Antigen R Alleviates Myocardial Fibrosis Induced by Cardiac Stress. JACC Basic to Translational Science. 9(6). 754–770. 3 indexed citations
4.
Tousif, Sultan, Anand Prakash Singh, Prachi Umbarkar, et al.. (2023). Ponatinib Drives Cardiotoxicity by S100A8/A9-NLRP3-IL-1β Mediated Inflammation. Circulation Research. 132(3). 267–289. 36 indexed citations
5.
Umbarkar, Prachi, et al.. (2023). GSK-3 at the heart of cardiometabolic diseases: Isoform-specific targeting is critical to therapeutic benefit. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1869(6). 166724–166724. 14 indexed citations
6.
Umbarkar, Prachi, et al.. (2023). Cardiac fibroblast GSK-3α aggravates ischemic cardiac injury by promoting fibrosis, inflammation, and impairing angiogenesis. Basic Research in Cardiology. 118(1). 35–35. 10 indexed citations
7.
Gupte, Manisha, et al.. (2022). Isoform-Specific Role of GSK-3 in High Fat Diet Induced Obesity and Glucose Intolerance. Cells. 11(3). 559–559. 11 indexed citations
8.
Umbarkar, Prachi, Sultan Tousif, Anand Prakash Singh, et al.. (2022). Fibroblast GSK-3α Promotes Fibrosis via RAF-MEK-ERK Pathway in the Injured Heart. Circulation Research. 131(7). 620–636. 48 indexed citations
9.
Riley, Lance A., Matthew R. Bersi, Prachi Umbarkar, et al.. (2021). Targeting 5-HT 2B Receptor Signaling Prevents Border Zone Expansion and Improves Microstructural Remodeling After Myocardial Infarction. Circulation. 143(13). 1317–1330. 42 indexed citations
10.
Umbarkar, Prachi, et al.. (2021). Mechanisms of Fibroblast Activation and Myocardial Fibrosis: Lessons Learned from FB-Specific Conditional Mouse Models. Cells. 10(9). 2412–2412. 30 indexed citations
11.
Umbarkar, Prachi, Anand Prakash Singh, Sultan Tousif, et al.. (2021). Repurposing Nintedanib for pathological cardiac remodeling and dysfunction. Pharmacological Research. 169. 105605–105605. 16 indexed citations
12.
Gupte, Manisha, Prachi Umbarkar, Anand Prakash Singh, et al.. (2020). Deletion of Cardiomyocyte Glycogen Synthase Kinase-3 Beta (GSK-3β) Improves Systemic Glucose Tolerance with Maintained Heart Function in Established Obesity. Cells. 9(5). 1120–1120. 11 indexed citations
13.
Raucci, Frank J., Anand Prakash Singh, Jonathan H. Soslow, et al.. (2020). The BDNF rs6265 Polymorphism is a Modifier of Cardiomyocyte Contractility and Dilated Cardiomyopathy. International Journal of Molecular Sciences. 21(20). 7466–7466. 8 indexed citations
14.
Singh, Anand Prakash, Prachi Umbarkar, Sultan Tousif, & Hind Lal. (2020). Cardiotoxicity of the BCR-ABL1 tyrosine kinase inhibitors: Emphasis on ponatinib. International Journal of Cardiology. 316. 214–221. 42 indexed citations
15.
Umbarkar, Prachi, Anand Prakash Singh, Manisha Gupte, et al.. (2019). Cardiomyocyte SMAD4-Dependent TGF-β Signaling is Essential to Maintain Adult Heart Homeostasis. JACC Basic to Translational Science. 4(1). 41–53. 37 indexed citations
16.
Singh, Anand Prakash, Prachi Umbarkar, Cristi L. Galindo, et al.. (2019). Ponatinib-induced cardiotoxicity: delineating the signalling mechanisms and potential rescue strategies. Cardiovascular Research. 115(5). 966–977. 60 indexed citations
17.
Gupte, Manisha, Anand Prakash Singh, Prachi Umbarkar, et al.. (2018). Cardiomyocyte-specific deletion of GSK-3β leads to cardiac dysfunction in a diet induced obesity model. International Journal of Cardiology. 259. 145–152. 24 indexed citations
18.
Guo, Yuanjun, Manisha Gupte, Prachi Umbarkar, et al.. (2017). Entanglement of GSK-3β, β-catenin and TGF-β1 signaling network to regulate myocardial fibrosis. Journal of Molecular and Cellular Cardiology. 110. 109–120. 132 indexed citations
19.
Umbarkar, Prachi, et al.. (2016). Mitochondrial Peroxiredoxin-3 protects against hyperglycemia induced myocardial damage in Diabetic cardiomyopathy. Free Radical Biology and Medicine. 97. 489–500. 50 indexed citations
20.
Umbarkar, Prachi, et al.. (2015). Monoamine oxidase-A is an important source of oxidative stress and promotes cardiac dysfunction, apoptosis, and fibrosis in diabetic cardiomyopathy. Free Radical Biology and Medicine. 87. 263–273. 75 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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