Ute M. Moll

29.3k total citations · 5 hit papers
157 papers, 15.7k citations indexed

About

Ute M. Moll is a scholar working on Oncology, Molecular Biology and Biotechnology. According to data from OpenAlex, Ute M. Moll has authored 157 papers receiving a total of 15.7k indexed citations (citations by other indexed papers that have themselves been cited), including 106 papers in Oncology, 103 papers in Molecular Biology and 44 papers in Biotechnology. Recurrent topics in Ute M. Moll's work include Cancer-related Molecular Pathways (95 papers), Cancer Research and Treatments (44 papers) and Ubiquitin and proteasome pathways (22 papers). Ute M. Moll is often cited by papers focused on Cancer-related Molecular Pathways (95 papers), Cancer Research and Treatments (44 papers) and Ubiquitin and proteasome pathways (22 papers). Ute M. Moll collaborates with scholars based in United States, Germany and France. Ute M. Moll's co-authors include Oleksi Petrenko, Alexander Zaika, Susan Erster, N D Marchenko, Angelina V. Vaseva, Neda Slade, Motohiro Mihara, Matthias Dobbelstein, G Riou and Sonja Wolff and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Ute M. Moll

155 papers receiving 15.5k citations

Hit Papers

p53 Has a Direct Apoptogenic Role at the Mitochondria 2000 2026 2008 2017 2003 2000 2003 2012 2007 400 800 1.2k

Peers

Ute M. Moll
Comparison fields: 5 of 151
  • Molecular Biology 10.6k
  • Oncology 7.9k
  • Cancer Research 3.4k
  • Biotechnology 1.9k
  • Immunology 1.3k
Replace Ygal Haupt with:
Ygal Haupt Australia
Varda Rotter Israel
Stephen N. Jones United States
Toshiyuki Miyashita Japan
Akira Nakagawara Japan
Maureen E. Murphy United States
Giovanni Blandino Italy
Yoichi Taya Japan
Jiandong Chen United States
John M. Sedivy United States
Ygal Haupt Australia View profile →
Citations per field, relative to Ute M. Moll
Ute M. Moll · 1×
Citations per year, relative to Ute M. Moll
Ute M. Moll · 1×

Countries citing papers authored by Ute M. Moll

Since Specialization
Citations

This map shows the geographic impact of Ute M. Moll's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ute M. Moll with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ute M. Moll more than expected).

Fields of papers citing papers by Ute M. Moll

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ute M. Moll. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ute M. Moll. The network helps show where Ute M. Moll may publish in the future.

Co-authorship network of co-authors of Ute M. Moll

This figure shows the co-authorship network connecting the top 25 collaborators of Ute M. Moll. A scholar is included among the top collaborators of Ute M. Moll based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ute M. Moll. Ute M. Moll is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 1
2 4
3 15
4 175
5 32
6 54
7
p53 Opens the Mitochondrial Permeability Transition Pore to Trigger Necrosis breakdown →
610
8 240
9 68
10 388
11 496
12
Aristolochic acid and the etiology of endemic (Balkan) nephropathy breakdown →
448
13 64
14
p53 Has a Direct Apoptogenic Role at the Mitochondria breakdown →
1403
15 6
16 97
17
Death Signal-induced Localization of p53 Protein to Mitochondria breakdown →
753
18 65
19 66
20 493

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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