Ramona Schulz
Impact in
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- Cancer-related Molecular Pathways
Papers in
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- Cell death mechanisms and regulation 2
- Ubiquitin and proteasome pathways 2
- Protein Degradation and Inhibitors 1
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- Macrophage Migration Inhibitory Factor 4
- Co-authors
- Ute M. Moll (7 shared papers)Natalia Marchenko (3 shared papers)Flaminia Talos (3 shared papers)Talía Velasco-Hernández (1 shared paper)Victoria Fischer (1 shared paper)Dun Li (1 shared paper)Matthias Dobbelstein (5 shared papers)Günter Fingerle‐Rowson (2 shared papers)
- Journals
- Cell Cycle (2 papers)The Journal of Experimental Medicine (2 papers)Molecular Cancer Research (1 paper)Glia (1 paper)Journal of Neural Transmission (1 paper)
- Partner nations
- GermanyUnited StatesUnited Kingdom
In The Last Decade
Ramona Schulz
13 papers receiving 532 citations
Peers
Comparison fields: 5 of 75
- Oncology 213
- Cancer Research 96
- Biotechnology 51
- Molecular Biology 379
- Cell Biology 77
Countries citing papers authored by Ramona Schulz
This map shows the geographic impact of Ramona Schulz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ramona Schulz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ramona Schulz more than expected).
Fields of papers citing papers by Ramona Schulz
This network shows the impact of papers produced by Ramona Schulz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ramona Schulz. The network helps show where Ramona Schulz may publish in the future.
Co-authors
The 25 scholars most cited alongside Ramona Schulz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2011 | 240 | |
| 2 | 2012 | 92 | |
| 3 | 2015 | 63 | |
| 4 | 2011 | 40 | |
| 5 | 2011 | 22 | |
| 6 | 2008 | 18 | |
| 7 | 2013 | 17 | |
| 8 | 2009 | 13 | |
| 9 | 2012 | 10 | |
| 10 | 2006 | 10 | |
| 11 | 1994 | 9 | |
| 12 | 2010 | 3 | |
| 13 | 2012 | 1 |
About Ramona Schulz
Ramona Schulz is a scholar working on Molecular Biology, Immunology, Cellular and Molecular Neuroscience, Oncology and Epidemiology, having authored 13 papers that have together received 538 indexed citations. Recurring topics across this work include Macrophage Migration Inhibitory Factor (4 papers), Cancer-related Molecular Pathways (2 papers), Cell death mechanisms and regulation (2 papers), Nuclear Receptors and Signaling (2 papers), Ubiquitin and proteasome pathways (2 papers), Autophagy in Disease and Therapy (1 paper), Paraoxonase enzyme and polymorphisms (1 paper) and Protein Degradation and Inhibitors (1 paper). The work is most often cited by research in Oncology (213 citations), Cancer Research (96 citations), Biotechnology (51 citations), Molecular Biology (379 citations) and Cell Biology (77 citations). Ramona Schulz has collaborated with scholars based in Germany, United States and United Kingdom. Frequent co-authors include Ute M. Moll, Natalia Marchenko, Flaminia Talos, Talía Velasco-Hernández, Victoria Fischer, Dun Li, Matthias Dobbelstein, Günter Fingerle‐Rowson, Lars Zender and Marina Pešić. Their work appears in journals such as Cell Cycle, The Journal of Experimental Medicine, Molecular Cancer Research, Glia and Journal of Neural Transmission.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.