Ygal Haupt

16.2k total citations · 4 hit papers
111 papers, 12.4k citations indexed

About

Ygal Haupt is a scholar working on Oncology, Molecular Biology and Biotechnology. According to data from OpenAlex, Ygal Haupt has authored 111 papers receiving a total of 12.4k indexed citations (citations by other indexed papers that have themselves been cited), including 72 papers in Oncology, 71 papers in Molecular Biology and 20 papers in Biotechnology. Recurrent topics in Ygal Haupt's work include Cancer-related Molecular Pathways (60 papers), Ubiquitin and proteasome pathways (24 papers) and Cancer Research and Treatments (20 papers). Ygal Haupt is often cited by papers focused on Cancer-related Molecular Pathways (60 papers), Ubiquitin and proteasome pathways (24 papers) and Cancer Research and Treatments (20 papers). Ygal Haupt collaborates with scholars based in Australia, Israel and United States. Ygal Haupt's co-authors include Moshe Oren, Ruth Maya, Sue Haupt, Ronit Vogt Sionov, Michael Berger, Zehavit Goldberg, Susan Haupt, Sheldon Rowan, Jerry M. Adams and Karen H. Vousden and has published in prestigious journals such as Nature, Cell and Nucleic Acids Research.

In The Last Decade

Ygal Haupt

111 papers receiving 12.1k citations

Hit Papers

Mdm2 promotes the rapid d... 1997 2026 2006 2016 1997 2003 2017 2021 1000 2.0k 3.0k

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ygal Haupt Australia 48 9.0k 7.1k 2.2k 1.5k 973 111 12.4k
Jiandong Chen United States 57 8.3k 0.9× 6.3k 0.9× 1.5k 0.7× 1.4k 0.9× 634 0.7× 127 11.4k
Ute M. Moll United States 64 10.6k 1.2× 7.9k 1.1× 3.4k 1.6× 1.9k 1.2× 694 0.7× 157 15.7k
Gerard P. Zambetti United States 58 10.6k 1.2× 8.0k 1.1× 3.2k 1.5× 1.7k 1.1× 758 0.8× 133 15.7k
Donna L. George United States 39 8.4k 0.9× 7.0k 1.0× 2.1k 1.0× 1.6k 1.0× 1.1k 1.2× 80 11.7k
Todd Waldman United States 39 8.0k 0.9× 5.7k 0.8× 2.0k 0.9× 946 0.6× 997 1.0× 66 10.9k
Klas G. Wiman Sweden 67 9.3k 1.0× 7.0k 1.0× 2.3k 1.1× 1.6k 1.0× 961 1.0× 179 14.1k
Xinbin Chen United States 56 7.5k 0.8× 4.4k 0.6× 1.9k 0.9× 977 0.6× 679 0.7× 176 9.8k
Maureen E. Murphy United States 54 8.4k 0.9× 5.4k 0.8× 2.4k 1.1× 868 0.6× 1.0k 1.1× 143 12.8k
Giovanni Blandino Italy 61 9.2k 1.0× 5.2k 0.7× 4.0k 1.9× 1.2k 0.8× 863 0.9× 266 12.8k
Gabriela Dontu United States 29 8.1k 0.9× 10.8k 1.5× 4.4k 2.1× 796 0.5× 1.2k 1.3× 42 14.7k

Countries citing papers authored by Ygal Haupt

Since Specialization
Citations

This map shows the geographic impact of Ygal Haupt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ygal Haupt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ygal Haupt more than expected).

Fields of papers citing papers by Ygal Haupt

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ygal Haupt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ygal Haupt. The network helps show where Ygal Haupt may publish in the future.

Co-authorship network of co-authors of Ygal Haupt

This figure shows the co-authorship network connecting the top 25 collaborators of Ygal Haupt. A scholar is included among the top collaborators of Ygal Haupt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ygal Haupt. Ygal Haupt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Caramia, Franco, Terence P. Speed, Hui Shen, Ygal Haupt, & Sue Haupt. (2023). Establishing the Link between X-Chromosome Aberrations and TP53 Status, with Breast Cancer Patient Outcomes. Cells. 12(18). 2245–2245. 5 indexed citations
2.
Fujihara, Kenji M., Carlos S. Cabalag, Hyun Soo Ko, et al.. (2021). SLC7A11 Is a Superior Determinant of APR-246 (Eprenetapopt) Response than TP53 Mutation Status. Molecular Cancer Therapeutics. 20(10). 1858–1867. 28 indexed citations
3.
Keam, Simon P., Heloise M. Halse, Thu Quynh Nguyen, et al.. (2020). High dose-rate brachytherapy of localized prostate cancer converts tumors from cold to hot. Journal for ImmunoTherapy of Cancer. 8(1). e000792–e000792. 51 indexed citations
4.
Williams, Scott, Han Xian Aw Yeang, Catherine Mitchell, et al.. (2020). Immune molecular profiling of a multiresistant primary prostate cancer with a neuroendocrine-like phenotype: a case report. BMC Urology. 20(1). 171–171. 9 indexed citations
5.
Haupt, Sue, Franco Caramia, Thierry Soussi, et al.. (2019). Identification of cancer sex-disparity in the functional integrity of p53 and its X chromosome network. Nature Communications. 10(1). 5385–5385. 64 indexed citations
6.
Haupt, Sue, et al.. (2017). The role of MDM2 and MDM4 in breast cancer development and prevention. Journal of Molecular Cell Biology. 9(1). 53–61. 63 indexed citations
7.
Liu, David S., Cuong Duong, Sue Haupt, et al.. (2017). Inhibiting the system xC−/glutathione axis selectively targets cancers with mutant-p53 accumulation. Nature Communications. 8(1). 14844–14844. 260 indexed citations
8.
Shapiro, Beny, et al.. (2014). HPV16 E6 and E6AP differentially cooperate to stimulate or augment Wnt signaling. Virology. 468-470. 510–523. 31 indexed citations
9.
Sionov, Ronit Vogt, et al.. (2013). The Regulation of p53 Growth Suppression. 8 indexed citations
11.
Haupt, Sue, Silvia Di Agostino, Osnat Alsheich-Bartok, et al.. (2009). Promyelocytic Leukemia Protein is Required for Gain of Function by Mutant p53. Cancer Research. 69(11). 4818–4826. 52 indexed citations
12.
Lichtig, Hava, et al.. (2009). HPV16 E6 augments Wnt signaling in an E6AP-dependent manner. Virology. 396(1). 47–58. 55 indexed citations
13.
Shaked, Helena, et al.. (2008). Chromatin Immunoprecipitation–on-Chip Reveals Stress-Dependent p53 Occupancy in Primary Normal Cells but Not in Established Cell Lines. Cancer Research. 68(23). 9671–9677. 49 indexed citations
14.
Haupt, Sue, et al.. (2006). Celecoxib can induce cell death independently of cyclooxygenase-2, p53, Mdm2, c-Abl and reactive oxygen species. Anti-Cancer Drugs. 17(6). 609–619. 5 indexed citations
15.
Shimada, Hideaki, Takenori Ochiai, Takanori Shimizu, et al.. (2002). Facilitation of adenoviral wild-type p53-induced apoptotic cell death by overexpression of p33ING1 in T.Tn human esophageal carcinoma cells. Oncogene. 21(8). 1208–1216. 37 indexed citations
16.
Haupt, Susan, Igal Louria‐Hayon, & Ygal Haupt. (2002). P53 licensed to kill? Operating the assassin. Journal of Cellular Biochemistry. 88(1). 76–82. 29 indexed citations
17.
Haupt, Ygal, et al.. (2000). p53 and thymic ‘death by neglect’: thymic epithelial cell-induced apoptosis of CD4+8+ thymocytes is p53-independent. Cell Death and Differentiation. 7(3). 241–249. 6 indexed citations
18.
Haupt, Ygal, Sheldon Rowan, & Moshe Oren. (1995). p53-mediated apoptosis in HeLa cells can be overcome by excess pRB.. PubMed. 10(8). 1563–71. 118 indexed citations
19.
Haupt, Ygal, Alan W. Harris, & Jerry M. Adams. (1993). Moloney virus induction of T‐cell lymphomas in a plasmacytomagenic strain of Eμ‐V‐ABL transgenic mice. International Journal of Cancer. 55(4). 623–629. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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