Tongwei Mo

3.3k total citations · 2 hit papers
15 papers, 2.4k citations indexed

About

Tongwei Mo is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Immunology. According to data from OpenAlex, Tongwei Mo has authored 15 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 7 papers in Pathology and Forensic Medicine and 7 papers in Immunology. Recurrent topics in Tongwei Mo's work include Lymphoma Diagnosis and Treatment (7 papers), Chronic Lymphocytic Leukemia Research (6 papers) and Ubiquitin and proteasome pathways (3 papers). Tongwei Mo is often cited by papers focused on Lymphoma Diagnosis and Treatment (7 papers), Chronic Lymphocytic Leukemia Research (6 papers) and Ubiquitin and proteasome pathways (3 papers). Tongwei Mo collaborates with scholars based in United States and Italy. Tongwei Mo's co-authors include Riccardo Dalla‐Favera, Qiong Shen, Marie Lia, Ulf Klein, Giorgio Cattoretti, Laura Pasqualucci, Klaus Rajewsky, Govind Bhagat, Thomas Ludwig and Stefano Casola and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Medicine and Nature Communications.

In The Last Decade

Tongwei Mo

15 papers receiving 2.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The DLEU2/miR-15a/16-1 Cluster Controls B Cell Proliferation and Its Deletion Leads to Chronic Lymphocytic Leukemia

2010 600 citations Ulf Klein, Marie Lia et al. Cancer Cell profile →
Transcription factor IRF4 controls plasma cell differentiation and class-switch recombination

2006 570 citations Ulf Klein, Stefano Casola et al. Nature Immunology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Tongwei Mo United States	9	1.1k	1.0k	673	546	537	15	2.4k
Lakshmi Srinivasan United States	11	1.3k 1.2×	753 0.7×	380 0.6×	920 1.7×	439 0.8×	29	2.3k
Marie Lia United States	11	1.3k 1.1×	642 0.6×	508 0.8×	600 1.1×	413 0.8×	17	2.2k
Tony G. Willis United Kingdom	13	675 0.6×	513 0.5×	890 1.3×	404 0.7×	526 1.0×	17	1.8k
Anna Migliazza Italy	19	1.0k 0.9×	877 0.8×	1.2k 1.7×	720 1.3×	884 1.6×	28	2.7k
Katerina Hatzi United States	24	1.1k 1.0×	1.1k 1.0×	711 1.1×	165 0.3×	500 0.9×	46	2.5k
Sandrine Sander Germany	15	1.6k 1.4×	441 0.4×	368 0.5×	359 0.7×	343 0.6×	20	2.2k
Peter Ouillette United States	19	797 0.7×	434 0.4×	750 1.1×	250 0.5×	508 0.9×	29	1.8k
Sabine Quief France	19	944 0.8×	472 0.4×	520 0.8×	196 0.4×	401 0.7×	29	1.8k
Daniel J. Hodson United Kingdom	18	635 0.6×	352 0.3×	513 0.8×	323 0.6×	404 0.8×	45	1.4k
Rewas Fatah United Kingdom	9	388 0.3×	1.0k 1.0×	408 0.6×	161 0.3×	1.1k 2.1×	13	1.9k

Countries citing papers authored by Tongwei Mo

Since Specialization

Citations

This map shows the geographic impact of Tongwei Mo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tongwei Mo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tongwei Mo more than expected).

Fields of papers citing papers by Tongwei Mo

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tongwei Mo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tongwei Mo. The network helps show where Tongwei Mo may publish in the future.

Co-authorship network of co-authors of Tongwei Mo

This figure shows the co-authorship network connecting the top 25 collaborators of Tongwei Mo. A scholar is included among the top collaborators of Tongwei Mo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tongwei Mo. Tongwei Mo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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15 of 15 papers shown

Shen, Qiong, Antony B. Holmes, Tongwei Mo, et al.. (2024). MEF2B C-terminal mutations enhance transcriptional activity and stability to drive B cell lymphomagenesis. Nature Communications. 15(1). 7195–7195. 1 indexed citations

Vlasevska, Sofija, Laura Garcia‐Ibanez, Romain Duval, et al.. (2023). KMT2D acetylation by CREBBP reveals a cooperative functional interaction at enhancers in normal and malignant germinal center B cells. Proceedings of the National Academy of Sciences. 120(11). e2218330120–e2218330120. 8 indexed citations

Zhang, Jiyuan, Sofija Vlasevska, Victoria A. Wells, et al.. (2017). The CREBBP Acetyltransferase Is a Haploinsufficient Tumor Suppressor in B-cell Lymphoma. Cancer Discovery. 7(3). 322–337. 151 indexed citations

Dominguez-Sola, David, Jennifer Kung, Antony B. Holmes, et al.. (2015). The FOXO1 Transcription Factor Instructs the Germinal Center Dark Zone Program. Immunity. 43(6). 1064–1074. 183 indexed citations

Zhang, Jiyuan, David Dominguez-Sola, Shafinaz Hussein, et al.. (2015). Disruption of KMT2D perturbs germinal center B cell development and promotes lymphomagenesis. Nature Medicine. 21(10). 1190–1198. 312 indexed citations

Lia, Marie, Amanda Carette, Hongyan Tang, et al.. (2011). Functional dissection of the chromosome 13q14 tumor-suppressor locus using transgenic mouse lines. Blood. 119(13). 2981–2990. 52 indexed citations

Lia, Marie, Amanda Carette, Hongyan Tang, et al.. (2011). In Vivo Functional Dissection of the Chromosome 13q14 Tumor Suppressor Locus Reveals That Extent of Deletions Impacts Disease Course and Phenotype. Blood. 118(21). 468–468. 1 indexed citations

Klein, Ulf, Marie Lia, Marta Crespo, et al.. (2010). The DLEU2/miR-15a/16-1 Cluster Controls B Cell Proliferation and Its Deletion Leads to Chronic Lymphocytic Leukemia. Cancer Cell. 17(1). 28–40. 600 indexed citations breakdown →

Mandelbaum, Jonathan, Govind Bhagat, Hongyan Tang, et al.. (2010). BLIMP1 Is a Tumor Suppressor Gene Frequently Disrupted in Activated B Cell-like Diffuse Large B Cell Lymphoma. Cancer Cell. 18(6). 568–579. 208 indexed citations

10.

Mandelbaum, Jonathan, Govind Bhagat, Tongwei Mo, et al.. (2009). Blimp1 Is a Tumor Suppressor Gene in Diffuse Large B Cell Lymphoma.. Blood. 114(22). 446–446. 1 indexed citations

11.

Klein, Ulf, Marie Lia, Qiong Shen, et al.. (2008). The DLEU2/Mir-15a/Mir-16-1 Locus, Commonly Deleted in B-Cell Chronic Lymphocytic Leukemia (CLL), Controls B-Cell Compartment Expansion and Its Deletion Leads to CLL in Mice. Blood. 112(11). 25–25. 2 indexed citations

12.

Klein, Ulf, Stefano Casola, Giorgio Cattoretti, et al.. (2006). Transcription factor IRF4 controls plasma cell differentiation and class-switch recombination. Nature Immunology. 7(7). 773–782. 570 indexed citations breakdown →

13.

Pasqualucci, Laura, Mara Compagno, Tongwei Mo, et al.. (2006). Activation Induced Cytidine Deaminase (AID) Is Required for Germinal-Center Derived Lymphomagenesis.. Blood. 108(11). 223–223. 1 indexed citations

14.

Cattoretti, Giorgio, Laura Pasqualucci, Gianna Ballon, et al.. (2005). Deregulated BCL6 expression recapitulates the pathogenesis of human diffuse large B cell lymphomas in mice. Cancer Cell. 7(5). 445–455. 271 indexed citations

15.

Klein, Ulf, Stefano Casola, Giorgio Cattoretti, et al.. (2005). IRF-4/MUM-1 Expression Is a Critical Switch in the Generation of Plasma Cells Versus Memory B-Cells.. Blood. 106(11). 337–337. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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