Cai Bowen

1.1k total citations
19 papers, 882 citations indexed

About

Cai Bowen is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Cai Bowen has authored 19 papers receiving a total of 882 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 8 papers in Oncology and 5 papers in Immunology. Recurrent topics in Cai Bowen's work include Cell death mechanisms and regulation (4 papers), Ubiquitin and proteasome pathways (4 papers) and Cancer-related Molecular Pathways (4 papers). Cai Bowen is often cited by papers focused on Cell death mechanisms and regulation (4 papers), Ubiquitin and proteasome pathways (4 papers) and Cancer-related Molecular Pathways (4 papers). Cai Bowen collaborates with scholars based in United States, China and Switzerland. Cai Bowen's co-authors include Edward P. Gelmann, Kotohiko Kimura, Sarah Spiegel, Lukas Bubendorf, Mark C. Markowski, Heinz Voeller, Niels Willi, Olli Kallioniemi, Thomas C. Gasser and G Sauter and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Blood.

In The Last Decade

Cai Bowen

16 papers receiving 869 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Cai Bowen United States	12	573	360	247	198	77	19	882
Diego Iglesias‐Gato Denmark	18	509 0.9×	353 1.0×	209 0.8×	228 1.2×	72 0.9×	24	889
Sarab Lizard‐Nacol France	18	627 1.1×	261 0.7×	393 1.6×	331 1.7×	105 1.4×	33	1.1k
JI Epstein United States	4	334 0.6×	200 0.6×	202 0.8×	120 0.6×	46 0.6×	6	598
Shiva S. Forootan United Kingdom	15	621 1.1×	180 0.5×	161 0.7×	394 2.0×	44 0.6×	22	838
Natasja F. Dits Netherlands	16	575 1.0×	539 1.5×	158 0.6×	342 1.7×	143 1.9×	21	1.0k
Yuanjie Niu China	20	703 1.2×	582 1.6×	279 1.1×	413 2.1×	114 1.5×	49	1.2k
Holger H.H. Erb Germany	17	626 1.1×	453 1.3×	366 1.5×	393 2.0×	82 1.1×	58	1.2k
Jeanie Wu Singapore	13	552 1.0×	149 0.4×	218 0.9×	258 1.3×	81 1.1×	14	904
Simeng Wen China	17	559 1.0×	310 0.9×	224 0.9×	407 2.1×	59 0.8×	30	933
Emily K. Kleczko United States	14	435 0.8×	249 0.7×	249 1.0×	143 0.7×	74 1.0×	22	746

Countries citing papers authored by Cai Bowen

Since Specialization

Citations

This map shows the geographic impact of Cai Bowen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Cai Bowen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Cai Bowen more than expected).

Fields of papers citing papers by Cai Bowen

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Cai Bowen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Cai Bowen. The network helps show where Cai Bowen may publish in the future.

Co-authorship network of co-authors of Cai Bowen

This figure shows the co-authorship network connecting the top 25 collaborators of Cai Bowen. A scholar is included among the top collaborators of Cai Bowen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Cai Bowen. Cai Bowen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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19 of 19 papers shown

Bowen, Cai, et al.. (2025). FKBP5 Induces Senescence in BMSCs and Inhibits Osteogenic Differentiation Through the Canonical WNT/β‐Catenin Signalling Pathway in Senile Osteoporosis. Journal of Cellular and Molecular Medicine. 29(8). e70552–e70552.

Scuoppo, Claudio, Cai Bowen, Kenneth Ofori, et al.. (2024). Repurposing NAMPT Inhibitors for Germinal Center B Cell–Like Diffuse Large B-Cell Lymphoma. Blood Cancer Discovery. 5(6). 417–427.

Kim, Andrew, Yunchao Chang, Antony B. Holmes, et al.. (2024). Differential Role of Crebbp Missense and Truncating Mutations in the Malignant Transformation of Germinal Center B Cells. Blood. 144(Supplement 1). 47–47.

Zhu, Bin, et al.. (2024). Evaluating the outcomes of three dimensional printing-assisted osteotomy on treating varus knee deformity from old tibial plateau fractures. International Orthopaedics. 49(2). 429–435. 1 indexed citations

Vlasevska, Sofija, Laura Garcia‐Ibanez, Romain Duval, et al.. (2023). KMT2D acetylation by CREBBP reveals a cooperative functional interaction at enhancers in normal and malignant germinal center B cells. Proceedings of the National Academy of Sciences. 120(11). e2218330120–e2218330120. 8 indexed citations

Bowen, Cai, Maho Shibata, Hailan Zhang, et al.. (2020). CRISPR/Cas9-Mediated Point Mutation in Nkx3.1 Prolongs Protein Half-Life and Reverses Effects Nkx3.1 Allelic Loss. Cancer Research. 80(21). 4805–4814. 2 indexed citations

Bowen, Cai, Michael C. Ostrowski, Gustavo Leone, & Edward P. Gelmann. (2019). Loss of PTEN Accelerates NKX3.1 Degradation to Promote Prostate Cancer Progression. Cancer Research. 79(16). 4124–4134. 22 indexed citations

Bowen, Cai, et al.. (2013). Functional Activation of ATM by the Prostate Cancer Suppressor NKX3.1. Cell Reports. 4(3). 516–529. 35 indexed citations

Bowen, Cai & Edward P. Gelmann. (2010). NKX3.1 Activates Cellular Response to DNA Damage. Cancer Research. 70(8). 3089–3097. 53 indexed citations

10.

Markowski, Mark C., Cai Bowen, & Edward P. Gelmann. (2008). Inflammatory Cytokines Induce Phosphorylation and Ubiquitination of Prostate Suppressor Protein NKX3.1. Cancer Research. 68(17). 6896–6901. 49 indexed citations

11.

Bowen, Cai. (2005). ESTABLISHMENT AND EVALUATION OF ACUTE CLOSED BRAIN INJURY MODEL IN RATS ACCORDING TO FEENEY'S METHOD. 1 indexed citations

12.

Gelmann, Edward P., Cai Bowen, & Lukas Bubendorf. (2003). Expression of NKX3.1 in normal and malignant tissues. The Prostate. 55(2). 111–117. 66 indexed citations

13.

Bowen, Cai, Michael J. Birrer, & Edward P. Gelmann. (2002). Retinoblastoma Protein-mediated Apoptosis After γ-Irradiation. Journal of Biological Chemistry. 277(47). 44969–44979. 38 indexed citations

14.

Kimura, Kotohiko, Mark C. Markowski, Cai Bowen, & Edward P. Gelmann. (2001). Androgen blocks apoptosis of hormone-dependent prostate cancer cells.. PubMed. 61(14). 5611–8. 88 indexed citations

15.

Bowen, Cai, Lukas Bubendorf, Heinz Voeller, et al.. (2000). Loss of NKX3.1 expression in human prostate cancers correlates with tumor progression.. PubMed. 60(21). 6111–5. 281 indexed citations

16.

Bowen, Cai, Heinz Voeller, Kristine Kikly, & Edward P. Gelmann. (1999). Synthesis of procaspases-3 and -7 during apoptosis in prostate cancer cells. Cell Death and Differentiation. 6(5). 394–401. 36 indexed citations

17.

Mack, Philip C., David R. Gandara, Cai Bowen, et al.. (1999). RB status as a determinant of response to UCN-01 in non-small cell lung carcinoma.. PubMed. 5(9). 2596–604. 40 indexed citations

18.

Kimura, Kotohiko, Cai Bowen, Sarah Spiegel, & Edward P. Gelmann. (1999). Tumor necrosis factor-alpha sensitizes prostate cancer cells to gamma-irradiation-induced apoptosis.. PubMed. 59(7). 1606–14. 95 indexed citations

19.

Bowen, Cai, Sarah Spiegel, & Edward P. Gelmann. (1998). Radiation-induced apoptosis mediated by retinoblastoma protein.. PubMed. 58(15). 3275–81. 67 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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