Govind Bhagat
- Molecular Biology top 0.5%
- Epidemiology top 0.2%
- Immunology top 0.2%
- Oncology top 0.5%
- Pathology and Forensic Medicine top 0.1%
- Co-authors
- Riccardo Dalla‐FaveraBeth LevineLaura PasqualucciPeter H.R. GreenSuzanne K. LewisHanina HibshooshPeter H. GreenGiorgio Cattoretti
- Topics
- Lymphoma Diagnosis and Treatment (97 papers)Celiac Disease Research and Management (70 papers)Microscopic Colitis (46 papers)
- Partner nations
- United StatesItalySwitzerland
In The Last Decade
Govind Bhagat
280 papers receiving 18.8k citations
Hit Papers
Peers
Comparison fields: 5 of 158
- Molecular Biology 6.5k
- Epidemiology 5.2k
- Immunology 5.1k
- Oncology 4.1k
- Pathology and Forensic Medicine 3.8k
Countries citing papers authored by Govind Bhagat
This map shows the geographic impact of Govind Bhagat's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Govind Bhagat with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Govind Bhagat more than expected).
Fields of papers citing papers by Govind Bhagat
This network shows the impact of papers produced by Govind Bhagat. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Govind Bhagat. The network helps show where Govind Bhagat may publish in the future.
Co-authorship network of co-authors of Govind Bhagat
This figure shows the co-authorship network connecting the top 25 collaborators of Govind Bhagat. A scholar is included among the top collaborators of Govind Bhagat based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Govind Bhagat. Govind Bhagat is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 7 | |
| 3 | 1 | |
| 4 | 53 | |
| 5 | 21 | |
| 6 | 7 | |
| 7 | 1 | |
| 8 | 192 | |
| 9 | 39 | |
| 10 | 49 | |
| 11 | 71 | |
| 12 | 16 | |
| 13 | 19 | |
| 14 | Akt-Mediated Regulation of Autophagy and Tumorigenesis Through Beclin 1 Phosphorylationbreakdown → | 601 |
| 15 | 100 | |
| 16 | 184 | |
| 17 | 99 | |
| 18 | 8 | |
| 19 | 219 | |
| 20 | Promotion of tumorigenesis by heterozygous disruption of the beclin 1 autophagy genebreakdown → | 1844 |
About Govind Bhagat
Govind Bhagat is a scholar working on Gastroenterology, Pathology and Forensic Medicine and Genetics, having authored 286 papers that have together received 19.1k indexed citations. Recurring topics across this work include Lymphoma Diagnosis and Treatment (97 papers), Celiac Disease Research and Management (70 papers) and Microscopic Colitis (46 papers). The work is most often cited by research in Gastroenterology (3.0k citations), Immunology (5.1k citations) and Pathology and Forensic Medicine (3.8k citations). Govind Bhagat has collaborated with scholars based in United States, Italy and Switzerland. Frequent co-authors include Riccardo Dalla‐Favera, Beth Levine, Laura Pasqualucci, Peter H.R. Green, Suzanne K. Lewis, Hanina Hibshoosh, Peter H. Green, Giorgio Cattoretti, Jie Yu and Xueping Qu. Their work appears in journals such as Nature, Science and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.