Subhadra V. Nandula

3.7k total citations · 1 hit paper
30 papers, 2.7k citations indexed

About

Subhadra V. Nandula is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Cancer Research. According to data from OpenAlex, Subhadra V. Nandula has authored 30 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 12 papers in Pathology and Forensic Medicine and 10 papers in Cancer Research. Recurrent topics in Subhadra V. Nandula's work include Lymphoma Diagnosis and Treatment (11 papers), Chronic Lymphocytic Leukemia Research (6 papers) and Genomic variations and chromosomal abnormalities (5 papers). Subhadra V. Nandula is often cited by papers focused on Lymphoma Diagnosis and Treatment (11 papers), Chronic Lymphocytic Leukemia Research (6 papers) and Genomic variations and chromosomal abnormalities (5 papers). Subhadra V. Nandula collaborates with scholars based in United States, Colombia and Switzerland. Subhadra V. Nandula's co-authors include Vundavalli V. Murty, Riccardo Dalla‐Favera, Laura Pasqualucci, Mara Compagno, Govind Bhagat, Qiong Shen, Adina Grunn, Francesco Bertoni, Mahesh Mansukhani and Gopeshwar Narayan and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Subhadra V. Nandula

30 papers receiving 2.6k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Mutations of multiple genes cause deregulation of NF-κB in diffuse large B-cell lymphoma

2009 795 citations Mara Compagno, Wei Keat Lim et al. Nature profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Subhadra V. Nandula United States	16	1.4k	894	885	782	690	30	2.7k
Claudia Döring Germany	28	1.1k 0.8×	712 0.8×	849 1.0×	686 0.9×	461 0.7×	82	2.3k
Natasha G. Deane United States	15	1.3k 1.0×	918 1.0×	528 0.6×	461 0.6×	450 0.7×	22	2.3k
Kolja Eppert Canada	16	1.8k 1.3×	841 0.9×	322 0.4×	491 0.6×	605 0.9×	27	3.1k
Jianguo Tao United States	25	1.8k 1.3×	642 0.7×	448 0.5×	680 0.9×	617 0.9×	76	2.8k
Luca Agnelli Italy	35	2.0k 1.4×	749 0.8×	550 0.6×	1.1k 1.4×	324 0.5×	92	2.9k
Elizabeth H. Stover United States	16	1.2k 0.9×	1.2k 1.4×	350 0.4×	414 0.5×	508 0.7×	46	2.7k
Ian J. Majewski Australia	24	1.5k 1.1×	744 0.8×	557 0.6×	486 0.6×	289 0.4×	46	2.7k
Marek Mráz Czechia	24	1.2k 0.9×	430 0.5×	551 0.6×	917 1.2×	650 0.9×	48	2.4k
Mark A. Pershouse United States	15	2.4k 1.7×	615 0.7×	433 0.5×	487 0.6×	289 0.4×	33	3.1k
Mamta Gupta United States	26	1.2k 0.9×	754 0.8×	824 0.9×	380 0.5×	524 0.8×	66	2.3k

Countries citing papers authored by Subhadra V. Nandula

Since Specialization

Citations

This map shows the geographic impact of Subhadra V. Nandula's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Subhadra V. Nandula with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Subhadra V. Nandula more than expected).

Fields of papers citing papers by Subhadra V. Nandula

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Subhadra V. Nandula. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Subhadra V. Nandula. The network helps show where Subhadra V. Nandula may publish in the future.

Co-authorship network of co-authors of Subhadra V. Nandula

This figure shows the co-authorship network connecting the top 25 collaborators of Subhadra V. Nandula. A scholar is included among the top collaborators of Subhadra V. Nandula based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Subhadra V. Nandula. Subhadra V. Nandula is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Zhang, Liqun, et al.. (2016). Chronic Lymphocytic Leukemia Transdifferentiates into Neuroendocrine Carcinoma: A Case Report of a New Phenomenon. North American Journal of Medicine and Science. 9(3). 1 indexed citations

Narayan, Gopeshwar, Dongxu Xie, Allen J. Freddy, et al.. (2013). PCDH10 promoter hypermethylation is frequent in most histologic subtypes of mature lymphoid malignancies and occurs early in lymphomagenesis. Genes Chromosomes and Cancer. 52(11). 1030–1041. 15 indexed citations

Narayan, Gopeshwar, Allen J. Freddy, Dongxu Xie, et al.. (2011). Promoter methylation‐mediated inactivation of PCDH10 in acute lymphoblastic leukemia contributes to chemotherapy resistance. Genes Chromosomes and Cancer. 50(12). 1043–1053. 27 indexed citations

Gonda, Tamas A., Michael Glick, Amrita Sethi, et al.. (2011). Polysomy and p16 deletion by fluorescence in situ hybridization in the diagnosis of indeterminate biliary strictures. Gastrointestinal Endoscopy. 75(1). 74–79. 79 indexed citations

Murty, Vundavalli V., et al.. (2010). Cytogenetic abnormalities in reactive lymphoid hyperplasia: byproducts of the germinal centre reaction or indicators of lymphoma?. Hematological Oncology. 29(2). 81–90. 8 indexed citations

Novak, Urban, Andrea Rinaldi, Ivo Kwee, et al.. (2009). The NF-κB negative regulator TNFAIP3 (A20) is inactivated by somatic mutations and genomic deletions in marginal zone lymphomas. Blood. 113(20). 4918–4921. 184 indexed citations

Compagno, Mara, Wei Keat Lim, Adina Grunn, et al.. (2009). Mutations of multiple genes cause deregulation of NF-κB in diffuse large B-cell lymphoma. Nature. 459(7247). 717–721. 795 indexed citations breakdown →

Pasqualucci, Laura, Mara Compagno, Wei Keat Lim, et al.. (2008). Mutations in Multiple Genes Cause Deregulation of the NFkB Pathway in Diffuse Large B-Cell Lymphoma. Blood. 112(11). 801–801. 4 indexed citations

Nandula, Subhadra V., et al.. (2008). Diffuse large B-cell lymphoma with TEL/ETV6 translocation. Human Pathology. 40(4). 588–593. 4 indexed citations

10.

Scotto, Luigi, Gopeshwar Narayan, Subhadra V. Nandula, et al.. (2008). Identification of copy number gain and overexpressed genes on chromosome arm 20q by an integrative genomic approach in cervical cancer: Potential role in progression. Genes Chromosomes and Cancer. 47(9). 755–765. 270 indexed citations

11.

Shakya, Reena, Matthias Szabolcs, Katia Basso, et al.. (2008). The basal-like mammary carcinomas induced by Brca1 or Bard1 inactivation implicate the BRCA1/BARD1 heterodimer in tumor suppression. Proceedings of the National Academy of Sciences. 105(19). 7040–7045. 104 indexed citations

12.

Scotto, Luigi, Gopeshwar Narayan, Subhadra V. Nandula, et al.. (2008). Integrative genomics analysis of chromosome 5p gain in cervical cancer reveals target over-expressed genes, including Drosha. Molecular Cancer. 7(1). 58–58. 103 indexed citations

13.

Nandula, Subhadra V., Shuang Wang, Maria Jasin, et al.. (2007). Structural Requirements for the BARD1 Tumor Suppressor in Chromosomal Stability and Homology-directed DNA Repair. Journal of Biological Chemistry. 282(47). 34325–34333. 43 indexed citations

14.

Narayan, Gopeshwar, Seeta R. Chaganti, Hugo Arias‐Pulido, et al.. (2007). Gene dosage alterations revealed by cDNA microarray analysis in cervical cancer: Identification of candidate amplified and overexpressed genes. Genes Chromosomes and Cancer. 46(4). 373–384. 167 indexed citations

15.

Pasqualucci, Laura, Mara Compagno, Jane Houldsworth, et al.. (2006). Inactivation of the PRDM1/BLIMP1 gene in diffuse large B cell lymphoma. The Journal of Experimental Medicine. 203(2). 311–317. 263 indexed citations

16.

Nandula, Subhadra V., et al.. (2005). CongenitalMLL-positive B-cell acute lymphoblastic leukemia (B-ALL) switched lineage at relapse to acute myelocytic leukemia (AML) with persistent t(4;11) and t(1;6) translocations and JH gene rearrangement. Leukemia & lymphoma. 46(8). 1223–1227. 25 indexed citations

17.

Cattoretti, Giorgio, Laura Pasqualucci, Gianna Ballon, et al.. (2005). Deregulated BCL6 expression recapitulates the pathogenesis of human diffuse large B cell lymphomas in mice. Cancer Cell. 7(5). 445–455. 271 indexed citations

18.

Jakob, John, Satoru Nagase, Adi Gazdar, et al.. (2005). Two somatic biallelic lesions within and near SMAD4 in a human breast cancer cell line. Genes Chromosomes and Cancer. 42(4). 372–383. 9 indexed citations

19.

Nandula, Subhadra V., Alain Borczuk, & Vundavalli V. Murty. (2004). Unbalanced t(2;19) and t(2;16) in a neurofibroma. Cancer Genetics and Cytogenetics. 152(2). 169–171. 3 indexed citations

20.

Osunkwo, Ifeyinwa, Subhadra V. Nandula, Vundavalli V. Murty, et al.. (2004). Mature B-cell acute lymphoblastic leukemia with t(9;11) translocation: a distinct subset of B-cell acute lymphoblastic leukemia. Modern Pathology. 17(7). 832–839. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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