Tomohiro Ide

6.5k total citations · 1 hit paper
29 papers, 2.6k citations indexed

About

Tomohiro Ide is a scholar working on Molecular Biology, Surgery and Physiology. According to data from OpenAlex, Tomohiro Ide has authored 29 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Molecular Biology, 11 papers in Surgery and 9 papers in Physiology. Recurrent topics in Tomohiro Ide's work include Peroxisome Proliferator-Activated Receptors (16 papers), Metabolism, Diabetes, and Cancer (9 papers) and Adipose Tissue and Metabolism (9 papers). Tomohiro Ide is often cited by papers focused on Peroxisome Proliferator-Activated Receptors (16 papers), Metabolism, Diabetes, and Cancer (9 papers) and Adipose Tissue and Metabolism (9 papers). Tomohiro Ide collaborates with scholars based in Japan, United Kingdom and United States. Tomohiro Ide's co-authors include Koji Murakami, Hitoshi Shimano, Naoya Yahagi, Nobuhiro Yamada, Takashi Matsuzaka, Hirohito Sone, Akimitsu Takahashi, Ryozo Nagai, Masanori Nakakuki and Shun Ishibashi and has published in prestigious journals such as Journal of Biological Chemistry, Nature Medicine and Nature Cell Biology.

In The Last Decade

Tomohiro Ide

29 papers receiving 2.5k citations

Hit Papers

The Mechanisms by Which B... 2001 2026 2009 2017 2001 100 200 300 400 500

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author Last Decade Papers Cites
Tomohiro Ide 1.5k 813 752 641 402 29 2.6k
Edwards A. Park 1.8k 1.2× 987 1.2× 587 0.8× 639 1.0× 397 1.0× 74 3.2k
Motohiro Sekiya 1.2k 0.8× 532 0.7× 746 1.0× 615 1.0× 391 1.0× 58 2.5k
Isabelle Hainault 1.4k 1.0× 821 1.0× 1.2k 1.5× 967 1.5× 420 1.0× 45 3.1k
Kiyoto Motojima 2.0k 1.4× 1.0k 1.3× 654 0.9× 334 0.5× 513 1.3× 81 3.0k
X. Charlie Dong 1.6k 1.1× 621 0.8× 1.3k 1.7× 647 1.0× 228 0.6× 59 3.3k
Daniel Lindén 1.0k 0.7× 653 0.8× 919 1.2× 477 0.7× 394 1.0× 53 2.5k
Yong‐Ho Ahn 1.8k 1.2× 506 0.6× 398 0.5× 824 1.3× 243 0.6× 88 3.0k
Kimihiko Matsusue 2.3k 1.5× 1.3k 1.6× 1.5k 2.0× 614 1.0× 635 1.6× 51 3.8k
Masanori Nakakuki 848 0.6× 391 0.5× 530 0.7× 701 1.1× 357 0.9× 30 1.9k
Katsumi Iizuka 1.3k 0.9× 585 0.7× 736 1.0× 1.2k 1.8× 356 0.9× 86 2.8k

Countries citing papers authored by Tomohiro Ide

Since Specialization
Citations

This map shows the geographic impact of Tomohiro Ide's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomohiro Ide with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomohiro Ide more than expected).

Fields of papers citing papers by Tomohiro Ide

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tomohiro Ide. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomohiro Ide. The network helps show where Tomohiro Ide may publish in the future.

Co-authorship network of co-authors of Tomohiro Ide

This figure shows the co-authorship network connecting the top 25 collaborators of Tomohiro Ide. A scholar is included among the top collaborators of Tomohiro Ide based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tomohiro Ide. Tomohiro Ide is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Tsutsui, Yusuke, Naoki Kobayashi, Takashi Komine, et al.. (2023). Activation of AMP-activated protein kinase (AMPK) through inhibiting interaction with prohibitins. iScience. 26(4). 106293–106293. 6 indexed citations
2.
Ito, Minoru, Michiaki Nagasawa, Naoki Omae, et al.. (2013). A novel JNK2/SREBP-1c pathway involved in insulin-induced fatty acid synthesis in human adipocytes. Journal of Lipid Research. 54(6). 1531–1540. 35 indexed citations
3.
Ito, Minoru, et al.. (2011). Differential regulation of CIDEA and CIDEC expression by insulin via Akt1/2- and JNK2-dependent pathways in human adipocytes. Journal of Lipid Research. 52(8). 1450–1460. 25 indexed citations
4.
Omae, Naoki, Minoru Ito, Michiaki Nagasawa, et al.. (2011). Suppression of FoxO1/cell death-inducing DNA fragmentation factor α-like effector A (Cidea) axis protects mouse β-cells against palmitic acid-induced apoptosis. Molecular and Cellular Endocrinology. 348(1). 297–304. 21 indexed citations
5.
Ito, Minoru, et al.. (2010). Differential roles of CIDEA and CIDEC in insulin-induced anti-apoptosis and lipid droplet formation in human adipocytes. Journal of Lipid Research. 51(7). 1676–1684. 32 indexed citations
6.
Tsunoda, Masaki, et al.. (2010). Direct evidence for leptin-induced lipid oxidation independent of long-form leptin receptor. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. 1801(10). 1115–1122. 29 indexed citations
7.
Tsunoda, Masaki, et al.. (2008). A novel PPARα agonist ameliorates insulin resistance in dogs fed a high-fat diet. American Journal of Physiology-Endocrinology and Metabolism. 294(5). E833–E840. 28 indexed citations
8.
Kurabe, Nobuya, Satoko Arai, Naoto Kubota, et al.. (2008). The Death Effector Domain-containing DEDD Supports S6K1 Activity via Preventing Cdk1-dependent Inhibitory Phosphorylation. Journal of Biological Chemistry. 284(8). 5050–5055. 9 indexed citations
9.
Nakagawa, Yoshimi, Hitoshi Shimano, Tomohiro Yoshikawa, et al.. (2005). TFE3 transcriptionally activates hepatic IRS-2, participates in insulin signaling and ameliorates diabetes. Nature Medicine. 12(1). 107–113. 127 indexed citations
10.
Yamamoto, Takashi, Hitoshi Shimano, Yoshimi Nakagawa, et al.. (2004). SREBP-1 Interacts with Hepatocyte Nuclear Factor-4α and Interferes with PGC-1 Recruitment to Suppress Hepatic Gluconeogenic Genes. Journal of Biological Chemistry. 279(13). 12027–12035. 121 indexed citations
11.
Nagasawa, Michiaki, Tomohiro Ide, Masahiro Suzuki, et al.. (2004). Pharmacological characterization of a human-specific peroxisome proliferater-activated receptor α (PPARα) agonist in dogs. Biochemical Pharmacology. 67(11). 2057–2069. 24 indexed citations
13.
Yahagi, Naoya, Hitoshi Shimano, Takashi Matsuzaka, et al.. (2003). p53 Activation in Adipocytes of Obese Mice. Journal of Biological Chemistry. 278(28). 25395–25400. 178 indexed citations
14.
Yahagi, Naoya, Hitoshi Shimano, Alyssa H. Hasty, et al.. (2002). Absence of Sterol Regulatory Element-binding Protein-1 (SREBP-1) Ameliorates Fatty Livers but Not Obesity or Insulin Resistance in Lep/Lep Mice. Journal of Biological Chemistry. 277(22). 19353–19357. 301 indexed citations
15.
Yoshikawa, Tomohiro, Hitoshi Shimano, Naoya Yahagi, et al.. (2002). Polyunsaturated Fatty Acids Suppress Sterol Regulatory Element-binding Protein 1c Promoter Activity by Inhibition of Liver X Receptor (LXR) Binding to LXR Response Elements. Journal of Biological Chemistry. 277(3). 1705–1711. 330 indexed citations
16.
Miyachi, Hiroyuki, Masahiro Nomura, Tomohiro Ide, et al.. (2002). Design, synthesis and evaluation of substituted phenylpropanoic acid derivatives as peroxisome proliferator-activated receptor (PPAR) activators: novel human PPARα-selective activators. Bioorganic & Medicinal Chemistry Letters. 12(1). 77–80. 32 indexed citations
19.
Murakami, Koji, et al.. (1999). Evidence for Direct Binding of Fatty Acids and Eicosanoids to Human Peroxisome Proliferators-Activated Receptor α. Biochemical and Biophysical Research Communications. 260(3). 609–613. 62 indexed citations
20.
Murakami, Koji, et al.. (1999). Amelioration by KRP-297, a new thiazolidinedione, of impaired glucose uptake in skeletal muscle from obese insulin-resistant animals. Metabolism. 48(11). 1450–1454. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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