Toshio Murase

6.0k total citations
163 papers, 4.2k citations indexed

About

Toshio Murase is a scholar working on Endocrinology, Diabetes and Metabolism, Surgery and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Toshio Murase has authored 163 papers receiving a total of 4.2k indexed citations (citations by other indexed papers that have themselves been cited), including 73 papers in Endocrinology, Diabetes and Metabolism, 66 papers in Surgery and 49 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Toshio Murase's work include Diabetes, Cardiovascular Risks, and Lipoproteins (47 papers), Lipid metabolism and disorders (42 papers) and Lipoproteins and Cardiovascular Health (33 papers). Toshio Murase is often cited by papers focused on Diabetes, Cardiovascular Risks, and Lipoproteins (47 papers), Lipid metabolism and disorders (42 papers) and Lipoproteins and Cardiovascular Health (33 papers). Toshio Murase collaborates with scholars based in Japan, United States and Argentina. Toshio Murase's co-authors include Minoru Okubo, Tetsuro Kobayashi, Kōji Nakanishi, Fumimaro Takaku, Shun Ishibashi, Hiroshige Itakura, Kinori Kosáka, Nobuhiro Yamada, Hitoshi Shimano and Masanobu Kawakami and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and The Lancet.

In The Last Decade

Toshio Murase

161 papers receiving 4.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Toshio Murase Japan 36 1.8k 1.7k 1.1k 914 815 163 4.2k
Marta Letizia Hribal Italy 45 2.0k 1.1× 1.3k 0.8× 682 0.6× 2.3k 2.5× 807 1.0× 124 5.7k
Hideichi Makino Japan 38 1.5k 0.8× 1.5k 0.9× 1.3k 1.2× 1.2k 1.4× 345 0.4× 156 4.2k
Kimberly D. Kluckman United States 15 493 0.3× 1.3k 0.8× 771 0.7× 1.7k 1.9× 391 0.5× 17 4.5k
Yiu-Fai Chen United States 40 917 0.5× 568 0.3× 759 0.7× 1.2k 1.4× 1.1k 1.4× 90 4.2k
Yukio Tanizawa Japan 41 1.9k 1.1× 2.5k 1.5× 1.2k 1.1× 2.6k 2.8× 347 0.4× 152 5.9k
Maria‐Christina Zennaro France 41 3.3k 1.8× 1.7k 1.0× 599 0.5× 1.8k 2.0× 537 0.7× 130 4.9k
Murielle M. Véniant United States 44 1.5k 0.8× 1.4k 0.8× 423 0.4× 3.7k 4.0× 1.0k 1.3× 95 6.5k
Marianne Böni‐Schnetzler Switzerland 38 1.4k 0.8× 1.5k 0.9× 1.1k 1.0× 1.6k 1.7× 263 0.3× 66 4.3k
Stanley C. Rall United States 18 914 0.5× 1.1k 0.6× 275 0.3× 994 1.1× 399 0.5× 21 2.9k
Hiroto Furuta Japan 32 1.0k 0.6× 1.8k 1.0× 1.2k 1.1× 1.5k 1.6× 269 0.3× 98 3.7k

Countries citing papers authored by Toshio Murase

Since Specialization
Citations

This map shows the geographic impact of Toshio Murase's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Toshio Murase with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Toshio Murase more than expected).

Fields of papers citing papers by Toshio Murase

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Toshio Murase. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Toshio Murase. The network helps show where Toshio Murase may publish in the future.

Co-authorship network of co-authors of Toshio Murase

This figure shows the co-authorship network connecting the top 25 collaborators of Toshio Murase. A scholar is included among the top collaborators of Toshio Murase based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Toshio Murase. Toshio Murase is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Murase, Toshio, et al.. (2012). Cognitive decline in STZ-3V rats is largely due to dysfunctional insulin signalling through the dentate gyrus. Behavioural Brain Research. 229(2). 378–383. 43 indexed citations
2.
Kito, Shozo, et al.. (2012). Intracerebroventricular administration of an insulin analogue recovers STZ-induced cognitive decline in rats. Behavioural Brain Research. 241. 105–111. 58 indexed citations
3.
Amemiya-Kudo, Michiyo, Yoshinori Takeuchi, Hiroaki Okazaki, et al.. (2011). Suppression of the Pancreatic Duodenal Homeodomain Transcription Factor-1 (Pdx-1) Promoter by Sterol Regulatory Element-binding Protein-1c (SREBP-1c). Journal of Biological Chemistry. 286(32). 27902–27914. 12 indexed citations
4.
Aoyama, Yoshiko, Tetsu Ebara, Toshio Murase, et al.. (2010). Novel AGL mutation in a Turkish patient with glycogen storage disease type IIIa. Pediatrics International. 52(1). 145–147. 2 indexed citations
5.
Okubo, Minoru, Mieko Saito, Tetsu Ebara, et al.. (2007). A novel complex deletion–insertion mutation mediated by Alu repetitive elements leads to lipoprotein lipase deficiency. Molecular Genetics and Metabolism. 92(3). 229–233. 31 indexed citations
6.
Aoyama, Yoshiko, et al.. (2005). Molecular characterization of Egyptian patients with glycogen storage disease type IIIa. Journal of Human Genetics. 50(10). 538–542. 13 indexed citations
7.
Okubo, Minoru, Nelson L.S. Tang, Joannie Hui, et al.. (2002). Mutational and haplotype analysis of AGL in patients with glycogen storage disease type III. Journal of Human Genetics. 47(2). 55–59. 18 indexed citations
8.
10.
Okubo, Minoru, Fumio Kanda, Keiichi Takahashi, et al.. (1999). Glycogen storage disease type IIIa: first report of a causative missense mutation (G1448R) of the glycogen debranching enzyme gene found in a homozygous patient. Human Mutation. 14(6). 542–543. 13 indexed citations
12.
Inoue, Yasushi, Kōji Nakanishi, Minoru Okubo, et al.. (1997). Recovery of Pancreatic β-cell Function in Hemochromatosis: Combined Treatment With Recombinant Human Erythropoietin and Phlebotomy. The American Journal of the Medical Sciences. 314(6). 401–402. 7 indexed citations
14.
Kobayashi, Tetsuro, Y. Oka, Hideki Katagiri, et al.. (1996). Association between HLA and islet cell antibodies in diabetic patients with a mitochondrial DNA mutation at base pair 3243. Diabetologia. 39(10). 1196–1200. 17 indexed citations
15.
Ito, Hideki, Yutaka Harano, Masaaki Suzuki, et al.. (1996). Risk Factor Analyses for Macrovascular Complication in Nonobese NIDDM Patients: Multiclinical Study for Diabetic Macroangiopathy (MSDM). Diabetes. 45(Supplement_3). S19–S23. 33 indexed citations
16.
Oka, Y., Hideki Katagiri, Y Yazaki, Toshio Murase, & Tetsuro Kobayashi. (1993). Mitochondrial gene mutation in islet-cell-antibody-positive patients who were initially non-insulin-dependent diabetics. The Lancet. 342(8870). 527–528. 75 indexed citations
17.
Nakanishi, Koji, et al.. (1993). Relationships among residual β cells, exocrine pancreas, and islet cell antibodies in insulin-dependent diabetes mellitus. Metabolism. 42(2). 196–203. 36 indexed citations
18.
Kajio, Hiroshi, Tetsuro Kobayashi, Mitsuru Hara, et al.. (1992). Islet amyloid polypeptide (IAPP) gene analysis in a Japanese diabetic with marked islet amyloid deposition. Diabetes Research and Clinical Practice. 15(1). 45–48. 3 indexed citations
19.
Ohno, M., Shun Ishibashi, Kazuwa Nakao, et al.. (1989). A neonatal case of apolipoprotein C-II deficiency. European Journal of Pediatrics. 148(6). 550–552. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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