Tomas Luyten
- Molecular Biology top 10%
- Epidemiology top 10%
- Cell Biology top 5%
- Physiology top 2%
- Cellular and Molecular Neuroscience
- Co-authors
- Geert BultynckJan B. ParysLudwig MissiaenKirsten WelkenhuyzenHumbert De SmedtRaf PonsaertsJean-Paul DecuypereGiovanni Monaco
- Topics
- Calcium signaling and nucleotide metabolism (11 papers)Autophagy in Disease and Therapy (8 papers)Cell death mechanisms and regulation (8 papers)
- Journals
- Journal of Biological ChemistrySHILAP Revista de lepidopterologíaPLoS ONE
- Partner nations
- BelgiumUnited StatesUnited Kingdom
In The Last Decade
Tomas Luyten
39 papers receiving 1.1k citations
Peers
Comparison fields: 5 of 77
- Molecular Biology 693
- Epidemiology 290
- Cell Biology 282
- Physiology 185
- Cellular and Molecular Neuroscience 113
Countries citing papers authored by Tomas Luyten
This map shows the geographic impact of Tomas Luyten's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomas Luyten with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomas Luyten more than expected).
Fields of papers citing papers by Tomas Luyten
This network shows the impact of papers produced by Tomas Luyten. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomas Luyten. The network helps show where Tomas Luyten may publish in the future.
Co-authorship network of co-authors of Tomas Luyten
This figure shows the co-authorship network connecting the top 25 collaborators of Tomas Luyten. A scholar is included among the top collaborators of Tomas Luyten based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tomas Luyten. Tomas Luyten is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 5 | |
| 2 | 10 | |
| 3 | 2 | |
| 4 | 2 | |
| 5 | 15 | |
| 6 | 35 | |
| 7 | 45 | |
| 8 | 32 | |
| 9 | 10 | |
| 10 | 9 | |
| 11 | 37 | |
| 12 | 36 | |
| 13 | 12 | |
| 14 | Development of inositol-based antagonists for the D-myo-inositol 1,4,5-trisphosphate receptor | 1 |
| 15 | 72 | |
| 16 | 18 | |
| 17 | 53 | |
| 18 | 7 | |
| 19 | 83 | |
| 20 | 6 |
About Tomas Luyten
Tomas Luyten is a scholar working on Physiology, Sensory Systems and Toxicology, having authored 39 papers that have together received 1.1k indexed citations. Recurring topics across this work include Calcium signaling and nucleotide metabolism (11 papers), Autophagy in Disease and Therapy (8 papers) and Cell death mechanisms and regulation (8 papers). The work is most often cited by research in Physiology (185 citations), Sensory Systems (99 citations) and Cell Biology (282 citations). Tomas Luyten has collaborated with scholars based in Belgium, United States and United Kingdom. Frequent co-authors include Geert Bultynck, Jan B. Parys, Ludwig Missiaen, Kirsten Welkenhuyzen, Humbert De Smedt, Raf Ponsaerts, Jean-Paul Decuypere, Giovanni Monaco, Hristina Ivanova and Humbert De Smedt. Their work appears in journals such as Journal of Biological Chemistry, SHILAP Revista de lepidopterología and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.