Tomas Luyten

1.5k citations

39 papers · 1.1k indexed · h-index 17

Physiology top 2%
- Calcium signaling and nucleotide metabolism 11
Sensory Systems top 5%
- Ion Channels and Receptors 5
Cell Biology top 5%
- Endoplasmic Reticulum Stress and Disease 7
- Cellular transport and secretion 7
Molecular Biology top 10%
- Cell death mechanisms and regulation 8
- Ion channel regulation and function 7
Epidemiology top 10%
- Autophagy in Disease and Therapy 8
Toxicology
- Bioactive Compounds and Antitumor Agents 3

Co-authors: Geert Bultynck Jan B. Parys Ludwig Missiaen Kirsten Welkenhuyzen Humbert De Smedt Raf Ponsaerts Jean-Paul Decuypere Giovanni Monaco
Cited by: Physiology Sensory Systems Cell Biology
Partner nations: Belgium United States United Kingdom

In The Last Decade

Tomas Luyten

39 papers receiving 1.1k citations

Peers

Countries citing papers authored by Tomas Luyten

Since Specialization

Citations

This map shows the geographic impact of Tomas Luyten's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomas Luyten with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomas Luyten more than expected).

Fields of papers citing papers by Tomas Luyten

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tomas Luyten. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomas Luyten. The network helps show where Tomas Luyten may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Tomas Luyten, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Tomas Luyten Line = papers co-authored together Tomas Luyten links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

#	Work
1	The endoplasmic reticulum pool of Bcl-xL prevents cell death through IP3R-dependent calcium release Rudy Gadet,Lea Jabbour,Trang Thi Minh Nguyen,Olivier Lohez,Ivan Mikaélian,Philippe Gonzalo,Tomas Luyten,Mounira Chalabi‐Dchar,Anne Wierinckx,Olivier Marcillat,Geert Bultynck,Ruth Rimokh,Nikolay Popgeorgiev,Germain Gillet	2024	5
2	LRRK2 phosphorylation status and kinase activity regulate (macro)autophagy in a Rab8a/Rab10-dependent manner Elżbieta Kania,Jaclyn S. Long,David G. McEwan,Kirsten Welkenhuyzen,Rita La Rovere,Tomas Luyten,J. C. Halpin,Evy Lobbestael,Veerle Baekelandt,Geert Bultynck,Kevin M. Ryan,Jan B. Parys	2023	10
3	Loss of INPP5K attenuates IP3-induced Ca2+ responses in the glioblastoma cell line U-251 MG cells Jens Loncke,Tomas Luyten,Ana Raquel Ramos,Christophé Erneux,Geert Bultynck	2023	2
4	Synthesis and Characterization of Store-Operated Calcium Entry Inhibitors Active in the Submicromolar Range Camille Le Guilcher,Tomas Luyten,Jan B. Parys,Mathieu Pucheault,Olivier Dellis	2020	2
5	Ion and pH Sensitivity of a TMBIM Ca2+ Channel Gongrui Guo,Ming Xu,Yanqi Chang,Tomas Luyten,Bruno Seitaj,Wu Liu,Ping Zhu,Geert Bultynck,Lei Shi,Matthias Quick,Qun Liu	2019	15
6	Bcl-2 and IP3 compete for the ligand-binding domain of IP3Rs modulating Ca2+ signaling output Hristina Ivanova,Larry E. Wagner,Akihiko Tanimura,Elien Vandermarliere,Tomas Luyten,Kirsten Welkenhuyzen,Kamil J. Alzayady,Liwei Wang,Kozo Hamada,Katsuhiko Mikoshiba,Humbert De Smedt,Lennart Martens,David I. Yule,Jan B. Parys,Geert Bultynck	2019	35
7	Resveratrol-induced autophagy is dependent on IP3Rs and on cytosolic Ca2+ Tomas Luyten,Kirsten Welkenhuyzen,Gemma Roest,Elżbieta Kania,Liwei Wang,Mart Bittremieux,David I. Yule,Jan B. Parys,Geert Bultynck	2017	45
8	The selective Bcl-2 inhibitor venetoclax, a BH3 mimetic, does not dysregulate intracellular Ca 2+ signaling Tamara Vervloessem,Hristina Ivanova,Tomas Luyten,Jan B. Parys,Geert Bultynck	2016	32
9	Measurement of Intracellular Ca²⁺ Release in Permeabilized Cells Using ⁴⁵Ca²⁺ Tomas Luyten,Geert Bultynck,Jan B. Parys,Humbert De Smedt,Ludwig Missiaen	2014	10
10	Curcumin affects proprotein convertase activity: Elucidation of the molecular and subcellular mechanism Jingjing Zhu,Geert Bultynck,Tomas Luyten,Jan B. Parys,John W.M. Creemers,Wim J.M. Van de Ven,A.J.M. Vermorken	2013	9
11	Alzheimer's disease-associated peptide Aβ42 mobilizes ER Ca2+ via InsP3R-dependent and -independent mechanisms Laura E. Jensen,Geert Bultynck,Tomas Luyten,Hozeefa Amijee,Martin D. Bootman,H. Llewelyn Roderick	2013	37
12	Polycystin-1 and polycystin-2 are both required to amplify inositol-trisphosphate-induced Ca2+ release Djalila Mekahli,Eva Sammels,Tomas Luyten,Kirsten Welkenhuyzen,Lambertus P. van den Heuvel,Elena Levtchenko,Rik Gijsbers,Geert Bultynck,Jan B. Parys,Humbert De Smedt,Ludwig Missiaen	2012	36
13	Neuronal overexpression of IP3 receptor 2 is detrimental in mutant SOD1 mice Kim A. Staats,Elke Bogaert,Nicole Hersmus,Tom Jaspers,Tomas Luyten,Geert Bultynck,Jan B. Parys,Chihiro Hisatsune,Katsuhiko Mikoshiba,Philip Van Damme,Wim Robberecht,Ludo Van Den Bosch	2012	12
14	Development of inositol-based antagonists for the D-myo-inositol 1,4,5-trisphosphate receptor Y Ye,Neil S. Keddie,Tashfeen Aslam,Tomas Luyten,Davide Bello,Clive Garnham,Geert Bultynck,(unknown),Stuart J. Conway	2011	1
15	The C Terminus of Bax Inhibitor-1 Forms a Ca2+-permeable Channel Pore Geert Bultynck,Santeri Kiviluoto,Nadine Henke,Hristina Ivanova,Lars Schneider,Volodymyr Rybalchenko,Tomas Luyten,Koen Nuyts,Wim M. De Borggraeve,Ilya Bezprozvanny,Jan B. Parys,Humbert De Smedt,Ludwig Missiaen,Axel Methner	2011	72
16	Development of inositol-based antagonists for thed-myo-inositol 1,4,5-trisphosphate receptor Neil S. Keddie,Yulin Ye,Tariq Aslam,Tomas Luyten,Davide Bello,Clive Garnham,Geert Bultynck,(unknown),Stuart J. Conway	2010	18
17	Human Golgi Antiapoptotic Protein Modulates Intracellular Calcium Fluxes Fabrizio De Mattia,Caroline Gubser Keller,Michiel M.T. van Dommelen,Henk-Jan Visch,Felix Distelmaier,Antonio Postigo,Tomas Luyten,Jan B. Parys,Humbert De Smedt,Geoffey L. Smith,Peter H.G.M. Willems,Frank J. M. van Kuppeveld	2009	53
18	A type 2 Ferrier rearrangement-based synthesis of d-myo-inositol 1,4,5-trisphosphate Neil S. Keddie,Geert Bultynck,Tomas Luyten,Alexandra M. Z. Slawin,Stuart J. Conway	2009	7
19	The Coxsackievirus 2B Protein Increases Efflux of Ions from the Endoplasmic Reticulum and Golgi, thereby Inhibiting Protein Trafficking through the Golgi Arjan S. de Jong,Henk-Jan Visch,Fabrizio De Mattia,Michiel M. van Dommelen,Herman G.P. Swarts,Tomas Luyten,Geert Callewaert,Willem J. G. Melchers,Peter H.G.M. Willems,Frank J. M. van Kuppeveld	2006	83
20	Hyperosmotic stress stimulates inositol 1,4,5-trisphosphate and inositol 1,3,4,5-tetrakisphosphate formation independently of bis-diphosphoinositol tetrakisphosphate modulation Xavier Pesesse,Alexandre Leyman,Tomas Luyten,Ludwig Missiaen,Christophé Erneux	2005	6

About Tomas Luyten

Tomas Luyten is a scholar working on Physiology, Sensory Systems and Toxicology, having authored 39 papers that have together received 1.1k indexed citations. Recurring topics across this work include Calcium signaling and nucleotide metabolism (11 papers), Autophagy in Disease and Therapy (8 papers), Cell death mechanisms and regulation (8 papers), Endoplasmic Reticulum Stress and Disease (7 papers), Cellular transport and secretion (7 papers), Ion channel regulation and function (7 papers), Ion Channels and Receptors (5 papers) and Bioactive Compounds and Antitumor Agents (3 papers). The work is most often cited by research in Physiology (185 citations), Sensory Systems (99 citations) and Cell Biology (282 citations). Tomas Luyten has collaborated with scholars based in Belgium, United States and United Kingdom. Frequent co-authors include Geert Bultynck, Jan B. Parys, Ludwig Missiaen, Kirsten Welkenhuyzen, Humbert De Smedt, Raf Ponsaerts, Jean-Paul Decuypere, Giovanni Monaco, Hristina Ivanova and Humbert De Smedt.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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