Antonio Postigo

9.5k total citations · 4 hit papers
74 papers, 7.7k citations indexed

About

Antonio Postigo is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Antonio Postigo has authored 74 papers receiving a total of 7.7k indexed citations (citations by other indexed papers that have themselves been cited), including 46 papers in Molecular Biology, 22 papers in Oncology and 19 papers in Immunology. Recurrent topics in Antonio Postigo's work include Cell Adhesion Molecules Research (12 papers), Cancer-related gene regulation (10 papers) and Epigenetics and DNA Methylation (10 papers). Antonio Postigo is often cited by papers focused on Cell Adhesion Molecules Research (12 papers), Cancer-related gene regulation (10 papers) and Epigenetics and DNA Methylation (10 papers). Antonio Postigo collaborates with scholars based in Spain, United States and United Kingdom. Antonio Postigo's co-authors include Douglas C. Dean, Robin X Luo, J. William Harbour, Ester Sànchez‐Tilló, Míriam Cuatrecasas, Antoni Castells, Laura Siles, Oriol de Barrios, Francisco Sánchez‐Madrid and Douglas S. Darling and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Antonio Postigo

72 papers receiving 7.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Cdk Phosphorylation Triggers Sequential Intramolecular Interactions that Progressively Block Rb Functions as Cells Move through G1

1999 835 citations Robin X Luo, Antonio Postigo et al. Cell profile →
Rb Interacts with Histone Deacetylase to Repress Transcription

1998 819 citations Robin X Luo, Antonio Postigo et al. Cell profile →
Exit from G1 and S Phase of the Cell Cycle Is Regulated by Repressor Complexes Containing HDAC-Rb-hSWI/SNF and Rb-hSWI/SNF

2000 540 citations Antonio Postigo, Robin X Luo et al. Cell profile →
EMT-activating transcription factors in cancer: beyond EMT and tumor invasiveness

2012 426 citations Ester Sànchez‐Tilló, Yongqing Liu et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Antonio Postigo Spain	40	5.0k	2.8k	1.3k	1.2k	725	74	7.7k
Kentaro Semba Japan	42	4.6k 0.9×	3.0k 1.1×	1.3k 0.9×	1.0k 0.9×	723 1.0×	143	7.8k
Nora Heisterkamp United States	57	6.7k 1.3×	2.4k 0.9×	1.5k 1.1×	881 0.8×	1.6k 2.2×	220	15.2k
Masabumi Shibuya Japan	41	4.6k 0.9×	1.7k 0.6×	990 0.7×	1.2k 1.0×	455 0.6×	77	6.6k
Marikki Laiho Finland	42	6.8k 1.4×	2.8k 1.0×	496 0.4×	1.7k 1.4×	572 0.8×	116	9.2k
John Groffen United States	55	6.3k 1.3×	2.1k 0.8×	1.3k 1.0×	803 0.7×	1.6k 2.2×	195	13.0k
Bradley Spencer‐Dene United Kingdom	48	5.2k 1.1×	2.2k 0.8×	869 0.7×	1.1k 0.9×	1.0k 1.4×	83	8.0k
Wallace Y. Langdon Australia	52	5.9k 1.2×	2.4k 0.9×	2.7k 2.1×	718 0.6×	623 0.9×	122	9.2k
Katia Manova United States	47	7.4k 1.5×	1.9k 0.7×	1000 0.8×	1.6k 1.4×	1.7k 2.4×	79	10.7k
Brad St. Croix United States	32	4.1k 0.8×	1.8k 0.7×	761 0.6×	1.4k 1.2×	444 0.6×	58	6.0k
Douglas C. Dean United States	54	8.4k 1.7×	4.6k 1.7×	1.4k 1.0×	1.9k 1.6×	1.2k 1.6×	107	11.9k

Countries citing papers authored by Antonio Postigo

Since Specialization

Citations

This map shows the geographic impact of Antonio Postigo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Antonio Postigo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Antonio Postigo more than expected).

Fields of papers citing papers by Antonio Postigo

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Antonio Postigo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Antonio Postigo. The network helps show where Antonio Postigo may publish in the future.

Co-authorship network of co-authors of Antonio Postigo

This figure shows the co-authorship network connecting the top 25 collaborators of Antonio Postigo. A scholar is included among the top collaborators of Antonio Postigo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Antonio Postigo. Antonio Postigo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Chamorro-Jorganes, Aránzazu, Juan García Valero, Diana Reyes-Garau, et al.. (2025). YPEL2 regulates the efficacy of BRD4-EZH2 dual targeting in EZH2Y641mut germinal center-derived lymphoma. Neoplasia. 61. 101131–101131. 2 indexed citations

Sànchez‐Tilló, Ester, Leire Pedrosa, Balázs Győrffy, et al.. (2023). The EMT factor ZEB1 paradoxically inhibits EMT in BRAF-mutant carcinomas. JCI Insight. 8(20). 7 indexed citations

Rodríguez, A., Laia Fernández‐Barat, F. Espósito, et al.. (2022). Mutational Status of SMAD4 and FBXW7 Affects Clinical Outcome in TP53–Mutated Metastatic Colorectal Cancer. Cancers. 14(23). 5921–5921. 3 indexed citations

Lu, Xiaoqin, Yao Chen, Beatriz E. Rendon, et al.. (2021). Zeb1 induces immune checkpoints to form an immunosuppressive envelope around invading cancer cells. Science Advances. 7(21). 69 indexed citations

Barrios, Oriol de, Lidia Sánchez-Moral, Marlies Cortés, et al.. (2019). ZEB1 promotes inflammation and progression towards inflammation-driven carcinoma through repression of the DNA repair glycosylase MPG in epithelial cells. Gut. 68(12). 2129–2141. 42 indexed citations

Liu, Yongqing, Laura Siles, Antonio Postigo, & Douglas C. Dean. (2018). Epigenetically distinct sister chromatids and asymmetric generation of tumor initiating cells. Cell Cycle. 17(18). 2221–2229. 2 indexed citations

Cortés, Marlies, Lidia Sánchez-Moral, Oriol de Barrios, et al.. (2017). Tumor‐associated macrophages (TAMs) depend on ZEB1 for their cancer‐promoting roles. The EMBO Journal. 36(22). 3336–3355. 114 indexed citations

Barrios, Oriol de, Balázs Győrffy, María Jesús Fernández‐Aceñero, et al.. (2016). ZEB1-induced tumourigenesis requires senescence inhibition via activation of DKK1/mutant p53/Mdm2/CtBP and repression of macroH2A1. Gut. 66(4). 666–682. 32 indexed citations

Sànchez‐Tilló, Ester, Oriol de Barrios, Laura Siles, et al.. (2013). ZEB1 Promotes Invasiveness of Colorectal Carcinoma Cells through the Opposing Regulation of uPA and PAI-1. Clinical Cancer Research. 19(5). 1071–1082. 55 indexed citations

10.

Liu, Yongqing, Ester Sànchez‐Tilló, Xiaoqin Lu, et al.. (2013). Sequential Inductions of the ZEB1 Transcription Factor Caused by Mutation of Rb and Then Ras Proteins Are Required for Tumor Initiation and Progression. Journal of Biological Chemistry. 288(16). 11572–11580. 28 indexed citations

11.

Liu, Yongqing, Ester Sànchez‐Tilló, Xiaoqin Lu, et al.. (2013). The ZEB1 Transcription Factor Acts in a Negative Feedback Loop with miR200 Downstream of Ras and Rb1 to Regulate Bmi1 Expression. Journal of Biological Chemistry. 289(7). 4116–4125. 35 indexed citations

12.

Sànchez‐Tilló, Ester, Ariadna Lázaro, Míriam Cuatrecasas, et al.. (2010). ZEB1 represses E-cadherin and induces an EMT by recruiting the SWI/SNF chromatin-remodeling protein BRG1. Oncogene. 29(24). 3490–3500. 369 indexed citations

13.

Mattia, Fabrizio De, Caroline Gubser Keller, Henk-Jan Visch, et al.. (2009). Human Golgi Antiapoptotic Protein Modulates Intracellular Calcium Fluxes. Molecular Biology of the Cell. 20(16). 3638–3645. 53 indexed citations

14.

Postigo, Antonio, Justin R. Cross, Julian Downward, & Michael Way. (2006). Interaction of F1L with the BH3 domain of Bak is responsible for inhibiting vaccinia-induced apoptosis. Cell Death and Differentiation. 13(10). 1651–1662. 75 indexed citations

15.

Postigo, Antonio & Douglas C. Dean. (1999). Independent Repressor Domains in ZEB Regulate Muscle and T-Cell Differentiation. Molecular and Cellular Biology. 19(12). 7961–7971. 84 indexed citations

16.

Luo, Robin X, Antonio Postigo, & Douglas C. Dean. (1998). Rb Interacts with Histone Deacetylase to Repress Transcription. Cell. 92(4). 463–473. 819 indexed citations breakdown →

17.

Marazuela, Mónica, Antonio Postigo, Agustín Acevedo, et al.. (1994). Adhesion molecules from the LFA‐1/ICAM‐1, 3 and VLA‐4/VCAM‐1 pathways on T lymphocytes and vascular endothelium in Graves' and Hashimoto's thyroid glands. European Journal of Immunology. 24(10). 2483–2490. 56 indexed citations

18.

Postigo, Antonio, Joaquı́n Teixidó, & Francisco Sánchez‐Madrid. (1993). The α4β1/VCAM-1 adhesion pathway in physiology and disease. Research in Immunology. 144(9). 723–735. 73 indexed citations

19.

Postigo, Antonio, Paloma Sánchez‐Mateos, A I Lazarovits, Francisco Sánchez‐Madrid, & Manuel O. Landázuri. (1993). Alpha 4 beta 7 integrin mediates B cell binding to fibronectin and vascular cell adhesion molecule-1. Expression and function of alpha 4 integrins on human B lymphocytes.. The Journal of Immunology. 151(5). 2471–2483. 118 indexed citations

20.

Postigo, Antonio, Rosario García‐Vicuña, Federico Díaz‐González, et al.. (1992). Increased binding of synovial T lymphocytes from rheumatoid arthritis to endothelial-leukocyte adhesion molecule-1 (ELAM-1) and vascular cell adhesion molecule-1 (VCAM-1).. Journal of Clinical Investigation. 89(5). 1445–1452. 75 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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