Eva Sammels

554 total citations
10 papers, 442 citations indexed

About

Eva Sammels is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Genetics. According to data from OpenAlex, Eva Sammels has authored 10 papers receiving a total of 442 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Molecular Biology, 4 papers in Pathology and Forensic Medicine and 4 papers in Genetics. Recurrent topics in Eva Sammels's work include Genetic and Kidney Cyst Diseases (4 papers), Biomedical Research and Pathophysiology (4 papers) and Pancreatic function and diabetes (2 papers). Eva Sammels is often cited by papers focused on Genetic and Kidney Cyst Diseases (4 papers), Biomedical Research and Pathophysiology (4 papers) and Pancreatic function and diabetes (2 papers). Eva Sammels collaborates with scholars based in Belgium, United States and United Kingdom. Eva Sammels's co-authors include Jan B. Parys, Ludwig Missiaen, Geert Bultynck, Humbert De Smedt, Humbert De Smedt, Benoit Devogelaere, Djalila Mekahli, Yiqiang Cai, Stefan Somlo and Elena Levtchenko and has published in prestigious journals such as Journal of Biological Chemistry, Biochemical Journal and Endocrinology.

In The Last Decade

Eva Sammels

10 papers receiving 439 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Eva Sammels Belgium 8 273 127 111 85 49 10 442
Na Luo United States 12 284 1.0× 179 1.4× 143 1.3× 78 0.9× 20 0.4× 20 468
Zhejun Ji China 11 330 1.2× 29 0.2× 96 0.9× 27 0.3× 19 0.4× 14 592
Katsuki Kobayashi Japan 7 617 2.3× 70 0.6× 76 0.7× 14 0.2× 68 1.4× 8 737
Chris Partridge United Kingdom 6 311 1.1× 136 1.1× 53 0.5× 19 0.2× 100 2.0× 7 578
Patrick Lam Canada 15 308 1.1× 74 0.6× 254 2.3× 21 0.2× 62 1.3× 27 814
Hilde Verboomen Belgium 8 431 1.6× 45 0.4× 104 0.9× 19 0.2× 90 1.8× 8 531
Nita Sachan United States 7 552 2.0× 49 0.4× 27 0.2× 49 0.6× 51 1.0× 9 721
Daniel Helman United States 7 232 0.8× 62 0.5× 64 0.6× 8 0.1× 53 1.1× 8 505
Huanli Xie Canada 13 331 1.2× 45 0.4× 220 2.0× 53 0.6× 99 2.0× 26 559
Martin Krenn Austria 13 135 0.5× 60 0.5× 24 0.2× 14 0.2× 86 1.8× 36 394

Countries citing papers authored by Eva Sammels

Since Specialization
Citations

This map shows the geographic impact of Eva Sammels's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva Sammels with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva Sammels more than expected).

Fields of papers citing papers by Eva Sammels

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva Sammels. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva Sammels. The network helps show where Eva Sammels may publish in the future.

Co-authorship network of co-authors of Eva Sammels

This figure shows the co-authorship network connecting the top 25 collaborators of Eva Sammels. A scholar is included among the top collaborators of Eva Sammels based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eva Sammels. Eva Sammels is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Mekahli, Djalila, Jean-Paul Decuypere, Eva Sammels, et al.. (2013). Polycystin-1 but not polycystin-2 deficiency causes upregulation of the mTOR pathway and can be synergistically targeted with rapamycin and metformin. Pflügers Archiv - European Journal of Physiology. 466(8). 1591–604. 27 indexed citations
2.
Mekahli, Djalila, Eva Sammels, Tomas Luyten, et al.. (2012). Polycystin-1 and polycystin-2 are both required to amplify inositol-trisphosphate-induced Ca2+ release. Cell Calcium. 51(6). 452–458. 36 indexed citations
3.
Sammels, Eva, et al.. (2011). PROVIDING OPPORTUNITIES FOR PERSONAL AND CAREER DEVELOPMENT OF ENGINEERING STUDENTS. 25(7). 1858–1869. 1 indexed citations
4.
Martins, Joana Raquel, Patthara Kongsuphol, Eva Sammels, et al.. (2011). F508del-CFTR increases intracellular Ca2+ signaling that causes enhanced calcium-dependent Cl− conductance in cystic fibrosis. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1812(11). 1385–1392. 33 indexed citations
5.
Sammels, Eva, Jan B. Parys, Ludwig Missiaen, Humbert De Smedt, & Geert Bultynck. (2010). Intracellular Ca2+ storage in health and disease: A dynamic equilibrium. Cell Calcium. 47(4). 297–314. 156 indexed citations
6.
Sammels, Eva, Benoit Devogelaere, Djalila Mekahli, et al.. (2010). Polycystin-2 Activation by Inositol 1,4,5-Trisphosphate-induced Ca2+ Release Requires Its Direct Association with the Inositol 1,4,5-Trisphosphate Receptor in a Signaling Microdomain. Journal of Biological Chemistry. 285(24). 18794–18805. 101 indexed citations
7.
Sammels, Eva, Benoit Devogelaere, Djalila Mekahli, et al.. (2010). Unraveling the role of polycystin-2/inositol 1,4,5-trisphosphate receptor interaction in Ca2+ signaling. Communicative & Integrative Biology. 3(6). 530–532. 8 indexed citations
8.
Drigo, Rafael Arrojo e, Stijn L. J. Van Herck, Eva Sammels, et al.. (2009). Knockdown of the Type 3 Iodothyronine Deiodinase (D3) Interacting Protein Peroxiredoxin 3 Decreases D3-Mediated Deiodination in Intact Cells. Endocrinology. 150(11). 5171–5180. 2 indexed citations
9.
Devogelaere, Benoit, Eva Sammels, & Humbert De Smedt. (2008). The IRBIT domain adds new functions to the AHCY family. BioEssays. 30(7). 642–652. 36 indexed citations
10.
Devogelaere, Benoit, Monique Beullens, Eva Sammels, et al.. (2007). Protein phosphatase-1 is a novel regulator of the interaction between IRBIT and the inositol 1,4,5-trisphosphate receptor. Biochemical Journal. 407(2). 303–311. 42 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026