Tomás L. Falzone
- Molecular Biology top 10%
- Cellular and Molecular Neuroscience top 1%
- Physiology top 2%
- Cell Biology top 5%
- Neurology top 5%
- Co-authors
- Gorazd B. StokinLawrence S.B. GoldsteinConcepción LilloDavid S. WilliamsMarcelo RubinsteinVictorio M. Pozo DevotoEliezer MasliahPeter Davies
- Topics
- Alzheimer's disease research and treatments (12 papers)Neuroscience and Neuropharmacology Research (7 papers)Prion Diseases and Protein Misfolding (6 papers)
- Partner nations
- ArgentinaUnited StatesCzechia
In The Last Decade
Tomás L. Falzone
37 papers receiving 2.7k citations
Hit Papers
Peers
Comparison fields: 5 of 107
- Molecular Biology 1.2k
- Cellular and Molecular Neuroscience 1.1k
- Physiology 934
- Cell Biology 444
- Neurology 358
Countries citing papers authored by Tomás L. Falzone
This map shows the geographic impact of Tomás L. Falzone's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomás L. Falzone with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomás L. Falzone more than expected).
Fields of papers citing papers by Tomás L. Falzone
This network shows the impact of papers produced by Tomás L. Falzone. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomás L. Falzone. The network helps show where Tomás L. Falzone may publish in the future.
Co-authorship network of co-authors of Tomás L. Falzone
This figure shows the co-authorship network connecting the top 25 collaborators of Tomás L. Falzone. A scholar is included among the top collaborators of Tomás L. Falzone based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tomás L. Falzone. Tomás L. Falzone is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 1 | |
| 3 | 2 | |
| 4 | 14 | |
| 5 | 15 | |
| 6 | 13 | |
| 7 | 43 | |
| 8 | 2 | |
| 9 | 83 | |
| 10 | 3 | |
| 11 | 98 | |
| 12 | 71 | |
| 13 | 12 | |
| 14 | 23 | |
| 15 | 45 | |
| 16 | 78 | |
| 17 | 62 | |
| 18 | 46 | |
| 19 | 120 | |
| 20 | 3 |
About Tomás L. Falzone
Tomás L. Falzone is a scholar working on Developmental Neuroscience, Cellular and Molecular Neuroscience and Cell Biology, having authored 37 papers that have together received 2.7k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (12 papers), Neuroscience and Neuropharmacology Research (7 papers) and Prion Diseases and Protein Misfolding (6 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (1.1k citations), Physiology (934 citations) and Developmental Neuroscience (140 citations). Tomás L. Falzone has collaborated with scholars based in Argentina, United States and Czechia. Frequent co-authors include Gorazd B. Stokin, Lawrence S.B. Goldstein, Concepción Lillo, David S. Williams, Marcelo Rubinstein, Victorio M. Pozo Devoto, Eliezer Masliah, Peter Davies, Edward Rockenstein and Richard G. Brusch. Their work appears in journals such as Science, Cell and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.