Tomás L. Falzone

3.4k total citations · 1 hit paper
37 papers, 2.7k citations indexed

About

Tomás L. Falzone is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, Tomás L. Falzone has authored 37 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 14 papers in Cellular and Molecular Neuroscience and 13 papers in Physiology. Recurrent topics in Tomás L. Falzone's work include Alzheimer's disease research and treatments (12 papers), Neuroscience and Neuropharmacology Research (7 papers) and Prion Diseases and Protein Misfolding (6 papers). Tomás L. Falzone is often cited by papers focused on Alzheimer's disease research and treatments (12 papers), Neuroscience and Neuropharmacology Research (7 papers) and Prion Diseases and Protein Misfolding (6 papers). Tomás L. Falzone collaborates with scholars based in Argentina, United States and Czechia. Tomás L. Falzone's co-authors include Gorazd B. Stokin, Lawrence S.B. Goldstein, Concepción Lillo, David S. Williams, Marcelo Rubinstein, Victorio M. Pozo Devoto, Eliezer Masliah, Peter Davies, Edward Rockenstein and Richard G. Brusch and has published in prestigious journals such as Science, Cell and Journal of Biological Chemistry.

In The Last Decade

Tomás L. Falzone

37 papers receiving 2.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Axonopathy and Transport Deficits Early in the Pathogenesis of Alzheimer's Disease

2005 960 citations Gorazd B. Stokin, Concepción Lillo et al. profile →

Peers

Tomás L. Falzone

CMN

PHYSI

NEURO

Stephen R. Salton United States

Guomei Tang United States

Donna L. McPhie United States

Esther Asan Germany

Dane M. Chetkovich United States

Rudolf Kraftsik Switzerland

Michael E. Cahill United States

Hélène Marie France

Grace E. Stutzmann United States

Stephen R. Salton United States

Tomás L. Falzone

1.5k ×1.3

1.6k ×1.4

CMN

910 ×1.0

PHYSI

552 ×1.2

211 ×0.6

NEURO

Citations per year, relative to Tomás L. Falzone Tomás L. Falzone (= 1×) peers Stephen R. Salton

Countries citing papers authored by Tomás L. Falzone

Since Specialization

Citations

This map shows the geographic impact of Tomás L. Falzone's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomás L. Falzone with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomás L. Falzone more than expected).

Fields of papers citing papers by Tomás L. Falzone

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tomás L. Falzone. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomás L. Falzone. The network helps show where Tomás L. Falzone may publish in the future.

Co-authorship network of co-authors of Tomás L. Falzone

This figure shows the co-authorship network connecting the top 25 collaborators of Tomás L. Falzone. A scholar is included among the top collaborators of Tomás L. Falzone based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tomás L. Falzone. Tomás L. Falzone is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Otero, María Gabriela, et al.. (2025). Hyperglycemia-induced mitochondrial abnormalities in autonomic neurons via the RAGE axis. Scientific Reports. 15(1). 25231–25231. 2 indexed citations

Devoto, Victorio M. Pozo, et al.. (2024). Swedish Alzheimer’s disease variant perturbs activity of retrograde molecular motors and causes widespread derangement of axonal transport pathways. Journal of Biological Chemistry. 300(4). 107137–107137. 1 indexed citations

Navarro, Arcadi, et al.. (2024). Early Unguided Human Brain Organoid Neurovascular Niche Modeling into the Permissive Chick Embryo Chorioallantoic Membrane. Journal of Visualized Experiments. 2 indexed citations

Holubiec, Mariana, et al.. (2023). Mitochondrial vulnerability to oxidation in human brain organoids modelling Alzheimer's disease. Free Radical Biology and Medicine. 208. 394–401. 14 indexed citations

Karmirian, Karina, Mariana Holubiec, Livia Goto‐Silva, et al.. (2022). Modeling Alzheimer’s Disease Using Human Brain Organoids. Methods in molecular biology. 2561. 135–158. 15 indexed citations

Martinez, Emanuel Ricardo Monteiro, Karina Karmirian, Mariana Holubiec, et al.. (2022). DYRK1A Regulates the Bidirectional Axonal Transport of APP in Human-Derived Neurons. Journal of Neuroscience. 42(33). 6344–6358. 13 indexed citations

Falzone, Tomás L., et al.. (2021). Differential mitochondrial roles for α-synuclein in DRP1-dependent fission and PINK1/Parkin-mediated oxidation. Cell Death and Disease. 12(9). 796–796. 43 indexed citations

Fontanet, Paula, et al.. (2019). Tetraspanin1 promotes NGF signaling by controlling TrkA receptor proteostasis. Cellular and Molecular Life Sciences. 77(11). 2217–2233. 2 indexed citations

Devoto, Victorio M. Pozo, M. Belén Pardi, Trinidad Saez, et al.. (2017). αSynuclein control of mitochondrial homeostasis in human-derived neurons is disrupted by mutations associated with Parkinson’s disease. Scientific Reports. 7(1). 5042–5042. 83 indexed citations

10.

Bruno, Luciana, et al.. (2017). Methods for Quantitative Analysis of Axonal Cargo Transport. Methods in molecular biology. 1727. 217–226. 3 indexed citations

11.

Molina, Lis C. Puga, Ana Romarowski, Alejandra M. Vitale, et al.. (2016). Mouse sperm begin to undergo acrosomal exocytosis in the upper isthmus of the oviduct. Developmental Biology. 411(2). 172–182. 98 indexed citations

12.

Devoto, Victorio M. Pozo, Mária Čarná, Giancarlo Forte, et al.. (2016). Tau Isoforms Imbalance Impairs the Axonal Transport of the Amyloid Precursor Protein in Human Neurons. Journal of Neuroscience. 37(1). 58–69. 71 indexed citations

13.

Falzone, Tomás L., et al.. (2016). Hippocampal to basal forebrain transport of Mn 2+ is impaired by deletion of KLC1, a subunit of the conventional kinesin microtubule-based motor. NeuroImage. 145(Pt A). 44–57. 12 indexed citations

14.

Almenar‐Queralt, Angels, Tomás L. Falzone, Zhouxin Shen, et al.. (2014). UV Irradiation Accelerates Amyloid Precursor Protein (APP) Processing and Disrupts APP Axonal Transport. Journal of Neuroscience. 34(9). 3320–3339. 23 indexed citations

15.

Otero, María Gabriela, Angels Almenar‐Queralt, Sandra E. Encalada, et al.. (2014). Fast axonal transport of the proteasome complex depends on membrane interaction and molecular motor function. Journal of Cell Science. 127(Pt 7). 1537–49. 45 indexed citations

16.

Falzone, Tomás L., et al.. (2009). Axonal Stress Kinase Activation and Tau Misbehavior Induced by Kinesin-1 Transport Defects. Journal of Neuroscience. 29(18). 5758–5767. 78 indexed citations

17.

Stokin, Gorazd B., Angels Almenar‐Queralt, Shermali Gunawardena, et al.. (2008). Amyloid precursor protein-induced axonopathies are independent of amyloid-β peptides. Human Molecular Genetics. 17(22). 3474–3486. 62 indexed citations

18.

Gan, Lu, Tomás L. Falzone, Kehong Zhang, et al.. (2004). Enhanced Expression of Dopamine D<SUB>1</SUB> and Glutamate NMDA Receptors in Dopamine D<SUB>4</SUB> Receptor Knockout Mice. Journal of Molecular Neuroscience. 22(3). 167–178. 46 indexed citations

19.

Avale, María Elena, Tomás L. Falzone, Diego M. Gelman, et al.. (2003). The dopamine D4 receptor is essential for hyperactivity and impaired behavioral inhibition in a mouse model of attention deficit/hyperactivity disorder. Molecular Psychiatry. 9(7). 718–726. 120 indexed citations

20.

Falzone, Tomás L., et al.. (2000). Quantitative analysis of the dopamine D4 receptor in the mouse brain. Journal of Neuroscience Research. 59(2). 202–202. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact