Gustavo Paratcha

4.0k total citations
55 papers, 3.2k citations indexed

About

Gustavo Paratcha is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Developmental Neuroscience. According to data from OpenAlex, Gustavo Paratcha has authored 55 papers receiving a total of 3.2k indexed citations (citations by other indexed papers that have themselves been cited), including 45 papers in Cellular and Molecular Neuroscience, 26 papers in Molecular Biology and 23 papers in Developmental Neuroscience. Recurrent topics in Gustavo Paratcha's work include Neurogenesis and neuroplasticity mechanisms (23 papers), Neuroscience and Neuropharmacology Research (20 papers) and Nerve injury and regeneration (20 papers). Gustavo Paratcha is often cited by papers focused on Neurogenesis and neuroplasticity mechanisms (23 papers), Neuroscience and Neuropharmacology Research (20 papers) and Nerve injury and regeneration (20 papers). Gustavo Paratcha collaborates with scholars based in Argentina, Sweden and Brazil. Gustavo Paratcha's co-authors include Fernanda Ledda, Carlos F. Ibáñez, Jorge H. Medina, Miguelina Levi de Stein, Martı́n Cammarota, Iván Izquierdo, Lia R. Bevilaqua, Patrícia Grolli Ardenghi, Paula Fontanet and Antonella S. Ríos and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Gustavo Paratcha

54 papers receiving 3.1k citations

Peers

Gustavo Paratcha
Chia-Jen Siao United States
Henry Teng United States
Michael Dragunow New Zealand
Patricia E. Phelps United States
Gang‐Yi Wu United States
Daniel G. Herrera United States
Uta B. Schambra United States
W. Ernest Lyons United States
Newton H. Woo United States
Chia-Jen Siao United States
Gustavo Paratcha
Citations per year, relative to Gustavo Paratcha Gustavo Paratcha (= 1×) peers Chia-Jen Siao

Countries citing papers authored by Gustavo Paratcha

Since Specialization
Citations

This map shows the geographic impact of Gustavo Paratcha's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gustavo Paratcha with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gustavo Paratcha more than expected).

Fields of papers citing papers by Gustavo Paratcha

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gustavo Paratcha. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gustavo Paratcha. The network helps show where Gustavo Paratcha may publish in the future.

Co-authorship network of co-authors of Gustavo Paratcha

This figure shows the co-authorship network connecting the top 25 collaborators of Gustavo Paratcha. A scholar is included among the top collaborators of Gustavo Paratcha based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gustavo Paratcha. Gustavo Paratcha is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Ledda, Fernanda, et al.. (2023). Sprouty4 at the crossroads of Trk neurotrophin receptor signaling suppression by glucocorticoids. Frontiers in Molecular Neuroscience. 16. 1090824–1090824. 3 indexed citations
3.
Fontanet, Paula, et al.. (2019). Tetraspanin1 promotes NGF signaling by controlling TrkA receptor proteostasis. Cellular and Molecular Life Sciences. 77(11). 2217–2233. 2 indexed citations
4.
Ríos, Antonella S., et al.. (2019). Mechanisms That Modulate and Diversify BDNF Functions: Implications for Hippocampal Synaptic Plasticity. Frontiers in Cellular Neuroscience. 13. 135–135. 117 indexed citations
5.
Ledda, Fernanda & Gustavo Paratcha. (2017). Mechanisms regulating dendritic arbor patterning. Cellular and Molecular Life Sciences. 74(24). 4511–4537. 33 indexed citations
6.
Trinchero, Mariela F., et al.. (2017). High Plasticity of New Granule Cells in the Aging Hippocampus. Cell Reports. 21(5). 1129–1139. 69 indexed citations
7.
Fontanet, Paula, et al.. (2013). Pea3 Transcription Factor Family Members Etv4 and Etv5 Mediate Retrograde Signaling and Axonal Growth of DRG Sensory Neurons in Response to NGF. Journal of Neuroscience. 33(40). 15940–15951. 46 indexed citations
8.
Ledda, Fernanda, Gustavo Paratcha, Tatiana Sandoval‐Guzmán, & Carlos F. Ibáñez. (2007). GDNF and GFRα1 promote formation of neuronal synapses by ligand-induced cell adhesion. Nature Neuroscience. 10(3). 293–300. 138 indexed citations
9.
Sieber, Beth‐Anne, Alexander Kuzmin, Josep M. Canals, et al.. (2004). Disruption of EphA/ephrin-A signaling in the nigrostriatal system reduces dopaminergic innervation and dissociates behavioral responses to amphetamine and cocaine. Molecular and Cellular Neuroscience. 26(3). 418–428. 45 indexed citations
10.
Lucas, Guilherme, Panu Hendolin, Tibor Harkany, et al.. (2003). Neurotrophin-4 mediated TrkB activation reinforces morphine-induced analgesia. Nature Neuroscience. 6(3). 221–222. 20 indexed citations
11.
Paratcha, Gustavo, Fernanda Ledda, & Carlos F. Ibáñez. (2003). The Neural Cell Adhesion Molecule NCAM Is an Alternative Signaling Receptor for GDNF Family Ligands. Cell. 113(7). 867–879. 491 indexed citations
12.
Paratcha, Gustavo & Carlos F. Ibáñez. (2002). Lipid rafts and the control of neurotrophic factor signaling in the nervous system: variations on a theme. Current Opinion in Neurobiology. 12(5). 542–549. 103 indexed citations
13.
Cammarota, Martı́n, Miguelina Levi de Stein, Gustavo Paratcha, et al.. (2000). Rapid and Transient Learning-Associated Increase in NMDA NR1 Subunit in the Rat Hippocampus. Neurochemical Research. 25(5). 567–572. 48 indexed citations
14.
Paratcha, Gustavo, Melina Furman, Lia R. Bevilaqua, et al.. (2000). Involvement of hippocampal PKCβI isoform in the early phase of memory formation of an inhibitory avoidance learning. Brain Research. 855(2). 199–205. 45 indexed citations
15.
Wolansky, Marcelo J., et al.. (1999). Nerve growth factor preserves a critical motor period in rat striatum. Journal of Neurobiology. 38(1). 129–136. 5 indexed citations
16.
Bevilaqua, Lia R., Martı́n Cammarota, Gustavo Paratcha, et al.. (1999). Experience‐dependent increase in cAMP‐responsive element binding protein in synaptic and nonsynaptic mitochondria of the rat hippocampus. European Journal of Neuroscience. 11(10). 3753–3756. 29 indexed citations
17.
Paratcha, Gustavo, et al.. (1999). Decreased GAP-43/B-50 phosphorylation in striatal synaptic plasma membranes after circling motor behavior during development. Molecular Brain Research. 65(1). 34–43. 2 indexed citations
18.
Paratcha, Gustavo, et al.. (1998). Decreased Phosphorylation of GAP-43/B-50 in Striatal Synaptic Plasma Membranes after Circling Motor Activity. Neurochemical Research. 23(10). 1241–1249. 4 indexed citations
19.
Paratcha, Gustavo, et al.. (1997). Cold stress related alteration of RNA biosynthesis in brain cortex of mother-deprived newborn rats. Life Sciences. 60(24). 2165–2171. 5 indexed citations
20.
Paratcha, Gustavo, et al.. (1995). Permanent alteration of muscarinic acetylcholine receptor binding in rat striatum after circling training during development. Brain Research. 705(1-2). 39–44. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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