Thomas Langer
- Molecular Biology top 1%
- Materials Chemistry top 5%
- Organic Chemistry top 2%
- Cell Biology top 2%
- Immunology top 5%
- Co-authors
- F. Ulrich HartlJohn M. FlanaganBernd BukauHartwig SchröderJörg MartinHarrison EcholsManajit Hayer‐HartlChi Zen Lu
- Topics
- Heat shock proteins research (15 papers)Enzyme Structure and Function (11 papers)Protein Structure and Dynamics (10 papers)
- Cited by
- Molecular BiologyCell BiologyAging
- Partner nations
- GermanyUnited KingdomFrance
In The Last Decade
Thomas Langer
91 papers receiving 6.5k citations
Hit Papers
Peers
Comparison fields: 5 of 161
- Molecular Biology 4.9k
- Materials Chemistry 1.3k
- Organic Chemistry 863
- Cell Biology 736
- Immunology 623
Countries citing papers authored by Thomas Langer
This map shows the geographic impact of Thomas Langer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Langer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Langer more than expected).
Fields of papers citing papers by Thomas Langer
This network shows the impact of papers produced by Thomas Langer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Langer. The network helps show where Thomas Langer may publish in the future.
Co-authorship network of co-authors of Thomas Langer
This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Langer. A scholar is included among the top collaborators of Thomas Langer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Langer. Thomas Langer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 1 | |
| 3 | 3 | |
| 4 | 2 | |
| 5 | 8 | |
| 6 | 92 | |
| 7 | 96 | |
| 8 | 16 | |
| 9 | 58 | |
| 10 | 25 | |
| 11 | 5 | |
| 12 | 35 | |
| 13 | 54 | |
| 14 | 131 | |
| 15 | 44 | |
| 16 | 0 | |
| 17 | 3 | |
| 18 | Successive action of DnaK, DnaJ and GroEL along the pathway of chaperone-mediated protein foldingbreakdown → | 812 |
| 19 | Chaperonin-mediated protein folding at the surface of groEL through a 'molten globule'-like intermediatebreakdown → | 748 |
| 20 | 65 |
About Thomas Langer
Thomas Langer is a scholar working on Organic Chemistry, Molecular Biology and Automotive Engineering, having authored 94 papers that have together received 6.7k indexed citations. Recurring topics across this work include Heat shock proteins research (15 papers), Enzyme Structure and Function (11 papers) and Protein Structure and Dynamics (10 papers). The work is most often cited by research in Molecular Biology (4.9k citations), Cell Biology (736 citations) and Aging (70 citations). Thomas Langer has collaborated with scholars based in Germany, United Kingdom and France. Frequent co-authors include F. Ulrich Hartl, John M. Flanagan, Bernd Bukau, Hartwig Schröder, Jörg Martin, Harrison Echols, Manajit Hayer‐Hartl, Chi Zen Lu, Raina Boteva and Arthur L. Horwich. Their work appears in journals such as Nature, Science and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.