Thomas MacVicar

2.2k total citations · 2 hit papers
24 papers, 1.6k citations indexed

About

Thomas MacVicar is a scholar working on Molecular Biology, Clinical Biochemistry and Epidemiology. According to data from OpenAlex, Thomas MacVicar has authored 24 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 8 papers in Clinical Biochemistry and 5 papers in Epidemiology. Recurrent topics in Thomas MacVicar's work include Mitochondrial Function and Pathology (17 papers), Metabolism and Genetic Disorders (8 papers) and ATP Synthase and ATPases Research (7 papers). Thomas MacVicar is often cited by papers focused on Mitochondrial Function and Pathology (17 papers), Metabolism and Genetic Disorders (8 papers) and ATP Synthase and ATPases Research (7 papers). Thomas MacVicar collaborates with scholars based in Germany, United Kingdom and China. Thomas MacVicar's co-authors include Thomas Langer, Jon D. Lane, Hendrik Nolte, Marcus Krüger, Frederik Tellkamp, Amir Bahat, Christiane J. Bruns, Yohsuke Ohba, Hans‐Georg Sprenger and Jens C. Brüning and has published in prestigious journals such as Nature, The Journal of Cell Biology and The EMBO Journal.

In The Last Decade

Thomas MacVicar

22 papers receiving 1.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

OPA1 processing in cell death and disease – the long and short of it

2016 337 citations Thomas MacVicar, Thomas Langer Journal of Cell Science profile →
Mitochondrial metabolism coordinates stage-specific repair processes in macrophages during wound healing

2021 183 citations Sebastian Willenborg, David E. Sanin et al. Cell Metabolism profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Thomas MacVicar Germany	18	1.2k	272	222	211	209	24	1.6k
Ula V. Jurkunas United States	38	919 0.8×	222 0.8×	234 1.1×	112 0.5×	259 1.2×	114	3.9k
Ivan Nemazanyy France	26	1.2k 1.0×	253 0.9×	116 0.5×	189 0.9×	129 0.6×	81	1.7k
Valeria Manganelli Italy	27	1.3k 1.1×	362 1.3×	105 0.5×	113 0.5×	239 1.1×	77	2.1k
Jianfei Chen China	19	869 0.7×	101 0.4×	107 0.5×	186 0.9×	160 0.8×	74	1.5k
Susan Chung United States	16	1.4k 1.2×	143 0.5×	166 0.7×	213 1.0×	227 1.1×	21	2.0k
Marilyn Chwa United States	28	906 0.7×	97 0.4×	181 0.8×	244 1.2×	166 0.8×	81	2.4k
Liying Liu China	21	482 0.4×	268 1.0×	56 0.3×	195 0.9×	225 1.1×	57	1.1k
Saška Ivanova Russia	18	893 0.7×	390 1.4×	65 0.3×	167 0.8×	127 0.6×	43	1.4k
Joel S. Riley United Kingdom	14	1.5k 1.3×	461 1.7×	71 0.3×	221 1.0×	696 3.3×	21	2.1k
Bernadette Zwaans United States	17	1.2k 1.0×	474 1.7×	92 0.4×	302 1.4×	137 0.7×	44	2.2k

Countries citing papers authored by Thomas MacVicar

Since Specialization

Citations

This map shows the geographic impact of Thomas MacVicar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas MacVicar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas MacVicar more than expected).

Fields of papers citing papers by Thomas MacVicar

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas MacVicar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas MacVicar. The network helps show where Thomas MacVicar may publish in the future.

Co-authorship network of co-authors of Thomas MacVicar

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas MacVicar. A scholar is included among the top collaborators of Thomas MacVicar based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas MacVicar. Thomas MacVicar is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Dias, Marília M., Martin King, Engy Shokry, et al.. (2025). SLC25A45 is required for mitochondrial uptake of methylated amino acids and de novo carnitine biosynthesis. Molecular Cell. 85(21). 4093–4104.e8. 1 indexed citations

Martinelli, Silvia, Ian Powley, Leah Officer-Jones, et al.. (2024). Mitochondrial double-stranded RNA homeostasis depends on cell-cycle progression. Life Science Alliance. 7(11). e202402764–e202402764.

McGarry, Lynn, Hendrik Nolte, Soni Deshwal, et al.. (2023). Ribonucleotide synthesis by NME6 fuels mitochondrial gene expression. The EMBO Journal. 42(18). e113256–e113256. 14 indexed citations

Sprenger, Hans‐Georg, Thomas MacVicar, Amir Bahat, et al.. (2021). Cellular pyrimidine imbalance triggers mitochondrial DNA–dependent innate immunity. Nature Metabolism. 3(5). 636–650. 97 indexed citations

Willenborg, Sebastian, David E. Sanin, Alexander Jaïs, et al.. (2021). Mitochondrial metabolism coordinates stage-specific repair processes in macrophages during wound healing. Cell Metabolism. 33(12). 2398–2414.e9. 183 indexed citations breakdown →

Bahat, Amir, Thomas MacVicar, & Thomas Langer. (2021). Metabolism and Innate Immunity Meet at the Mitochondria. Frontiers in Cell and Developmental Biology. 9. 720490–720490. 61 indexed citations

MacVicar, Thomas, et al.. (2021). Protein Import Assay into Mitochondria Isolated from Human Cells. BIO-PROTOCOL. 11(12). e4057–e4057. 14 indexed citations

Ohba, Yohsuke, Thomas MacVicar, & Thomas Langer. (2020). Regulation of mitochondrial plasticity by the i-AAA protease YME1L. Biological Chemistry. 401(6-7). 877–890. 41 indexed citations

MacVicar, Thomas, et al.. (2020). The C-terminal region of the oxidoreductase MIA40 stabilizes its cytosolic precursor during mitochondrial import. BMC Biology. 18(1). 96–96. 18 indexed citations

10.

Wang, Zhefang, Fanyu Liu, Ningbo Fan, et al.. (2020). Targeting Glutaminolysis: New Perspectives to Understand Cancer Development and Novel Strategies for Potential Target Therapies. Frontiers in Oncology. 10. 589508–589508. 136 indexed citations

11.

Ahola, Sofia, Thomas Langer, & Thomas MacVicar. (2019). Mitochondrial Proteolysis and Metabolic Control. Cold Spring Harbor Perspectives in Biology. 11(7). a033936–a033936. 26 indexed citations

12.

MacVicar, Thomas, Yohsuke Ohba, Hendrik Nolte, et al.. (2019). Lipid signalling drives proteolytic rewiring of mitochondria by YME1L. Nature. 575(7782). 361–365. 125 indexed citations

13.

Nolte, Hendrik, Thomas MacVicar, Frederik Tellkamp, & Marcus Krüger. (2018). Instant Clue: A Software Suite for Interactive Data Visualization and Analysis. Scientific Reports. 8(1). 12648–12648. 159 indexed citations

14.

Sprenger, Hans‐Georg, Tim König, Maria Patrón, et al.. (2018). Loss of the mitochondrial i ‐ AAA protease YME 1L leads to ocular dysfunction and spinal axonopathy. EMBO Molecular Medicine. 11(1). 44 indexed citations

15.

Wai, Timothy, Hao Hu, Thomas MacVicar, et al.. (2016). Homozygous YME1L1 mutation causes mitochondriopathy with optic atrophy and mitochondrial network fragmentation. eLife. 5. 96 indexed citations

16.

MacVicar, Thomas & Thomas Langer. (2016). OPA1 processing in cell death and disease – the long and short of it. Journal of Cell Science. 129(12). 2297–2306. 337 indexed citations breakdown →

17.

MacVicar, Thomas, et al.. (2015). Targeted siRNA Screens Identify ER-to-Mitochondrial Calcium Exchange in Autophagy and Mitophagy Responses in RPE1 Cells. International Journal of Molecular Sciences. 16(6). 13356–13380. 48 indexed citations

18.

MacVicar, Thomas & Jon D. Lane. (2014). Impaired OMA1 dependent OPA1 cleavage and reduced DRP1 fission activity combine to prevent mitophagy in OXPHOS dependent cells. Journal of Cell Science. 127(Pt 10). 2313–25. 86 indexed citations

19.

Betin, Virginie M.S., Thomas MacVicar, Stephen F. Parsons, David J. Anstee, & Jon D. Lane. (2012). A cryptic mitochondrial targeting motif in Atg4D links caspase cleavage with mitochondrial import and oxidative stress. Autophagy. 8(4). 664–676. 43 indexed citations

20.

Humphrey, Suzanne, Thomas MacVicar, Andrew Stevenson, et al.. (2011). SulA-induced filamentation in Salmonella enterica serovar Typhimurium: effects on SPI-1 expression and epithelial infection. Journal of Applied Microbiology. 111(1). 185–196. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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