Tamás Csikós
- Molecular Biology
- Cardiology and Cardiovascular Medicine
- Endocrinology, Diabetes and Metabolism
- Immunology
- Cellular and Molecular Neuroscience
- Co-authors
- Thomas UngerPeter DémantAnita KlousTom van WezelClaudia RuivenkampStefan GallinatČestmı́r VlčekVáclav Pačes
- Topics
- Receptor Mechanisms and Signaling (4 papers)Hormonal Regulation and Hypertension (4 papers)Renin-Angiotensin System Studies (3 papers)
- Cited by
- Behavioral NeuroscienceEndocrine and Autonomic SystemsEndocrinology, Diabetes and Metabolism
- Journals
- Nature GeneticsBloodOncogene
- Partner nations
- NetherlandsUnited StatesGermany
In The Last Decade
Tamás Csikós
17 papers receiving 468 citations
Peers
Comparison fields: 5 of 77
- Molecular Biology 288
- Cardiology and Cardiovascular Medicine 102
- Endocrinology, Diabetes and Metabolism 86
- Immunology 70
- Cellular and Molecular Neuroscience 64
Countries citing papers authored by Tamás Csikós
This map shows the geographic impact of Tamás Csikós's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tamás Csikós with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tamás Csikós more than expected).
Fields of papers citing papers by Tamás Csikós
This network shows the impact of papers produced by Tamás Csikós. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tamás Csikós. The network helps show where Tamás Csikós may publish in the future.
Co-authorship network of co-authors of Tamás Csikós
This figure shows the co-authorship network connecting the top 25 collaborators of Tamás Csikós. A scholar is included among the top collaborators of Tamás Csikós based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tamás Csikós. Tamás Csikós is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 8 | |
| 3 | 5 | |
| 4 | 14 | |
| 5 | 3 | |
| 6 | 18 | |
| 7 | 188 | |
| 8 | 31 | |
| 9 | 24 | |
| 10 | 23 | |
| 11 | 16 | |
| 12 | 7 | |
| 13 | 42 | |
| 14 | 16 | |
| 15 | 28 | |
| 16 | 9 | |
| 17 | 22 | |
| 18 | 19 |
About Tamás Csikós
Tamás Csikós is a scholar working on Behavioral Neuroscience, Endocrinology, Diabetes and Metabolism and Endocrine and Autonomic Systems, having authored 18 papers that have together received 473 indexed citations. Recurring topics across this work include Receptor Mechanisms and Signaling (4 papers), Hormonal Regulation and Hypertension (4 papers) and Renin-Angiotensin System Studies (3 papers). The work is most often cited by research in Behavioral Neuroscience (27 citations), Endocrine and Autonomic Systems (38 citations) and Endocrinology, Diabetes and Metabolism (86 citations). Tamás Csikós has collaborated with scholars based in Netherlands, United States and Germany. Frequent co-authors include Thomas Unger, Peter Démant, Anita Klous, Tom van Wezel, Claudia Ruivenkamp, Stefan Gallinat, Čestmı́r Vlček, Václav Pačes, Peter C. Groot and Wolter J. Mooi. Their work appears in journals such as Nature Genetics, Blood and Oncogene.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.