Syed Haq

2.3k total citations
12 papers, 1.9k citations indexed

About

Syed Haq is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Surgery. According to data from OpenAlex, Syed Haq has authored 12 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 5 papers in Cardiology and Cardiovascular Medicine and 2 papers in Surgery. Recurrent topics in Syed Haq's work include Signaling Pathways in Disease (7 papers), Wnt/β-catenin signaling in development and cancer (3 papers) and Cardiac Fibrosis and Remodeling (2 papers). Syed Haq is often cited by papers focused on Signaling Pathways in Disease (7 papers), Wnt/β-catenin signaling in development and cancer (3 papers) and Cardiac Fibrosis and Remodeling (2 papers). Syed Haq collaborates with scholars based in United States, Canada and Italy. Syed Haq's co-authors include Thomas Force, Ashour Michael, Jeffery D. Molkentin, Gabriel Choukroun, Roger J. Hajjar, James R. Woodgett, Anthony Rosenzweig, Federica del Monte, Heiko Kilter and Richard D. Patten and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Circulation.

In The Last Decade

Syed Haq

12 papers receiving 1.9k citations

Peers

Syed Haq
Comparison fields: 5 of 87
  • Molecular Biology 1.3k
  • Cardiology and Cardiovascular Medicine 894
  • Surgery 225
  • Pathology and Forensic Medicine 179
  • Genetics 175
Heiko Kilter Germany
Tong Tang United States
Elie R. Chemaly United States
Christophe Montessuit Switzerland
Takehiko Mizuno Japan
Detlef Wencker United States
John W. Adams United States
Nathalie Mougenot France
Joerg Heineke Germany
Larissa Lipskaia France
Heiko Kilter Germany View profile →
Citations per field, relative to Syed Haq
Syed Haq · 1×
Citations per year, relative to Syed Haq
Syed Haq · 1×

Countries citing papers authored by Syed Haq

Since Specialization
Citations

This map shows the geographic impact of Syed Haq's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Syed Haq with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Syed Haq more than expected).

Fields of papers citing papers by Syed Haq

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Syed Haq. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Syed Haq. The network helps show where Syed Haq may publish in the future.

Co-authorship network of co-authors of Syed Haq

This figure shows the co-authorship network connecting the top 25 collaborators of Syed Haq. A scholar is included among the top collaborators of Syed Haq based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Syed Haq. Syed Haq is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
# Title Journal Authors Indexed citations
1 Activation of β-catenin Signaling Pathways by Classical G-Protein-Coupled Receptors: Mechanisms and Consequences in Cycling and Non-cycling Cells Cell Cycle Sergey P. Shevtsov, Syed Haq et al. 37
2 The β-Catenin/T-Cell Factor/Lymphocyte Enhancer Factor Signaling Pathway Is Required for Normal and Stress-Induced Cardiac Hypertrophy Molecular and Cellular Biology Xin Chen, Sergei Shevtsov et al. 111
3 Glycogen Synthase Kinase-3β Regulates Growth, Calcium Homeostasis, and Diastolic Function in the Heart Journal of Biological Chemistry Ashour Michael, Syed Haq et al. 110
4 17β-Estradiol Reduces Cardiomyocyte Apoptosis In Vivo and In Vitro via Activation of Phospho-Inositide-3 Kinase/Akt Signaling Circulation Research Richard D. Patten, Mark Aronovitz et al. 274
5 Renal ischemia/reperfusion and ATP depletion/repletion in LLC-PK1 cells result in phosphorylation of FKHR and FKHRL1 Kidney International Michele Andreucci, Ashour Michael et al. 73
6 Deletion of cytosolic phospholipase A2 promotes striated muscle growth Nature Medicine Syed Haq, Heiko Kilter et al. 74
7 Stabilization of β-catenin by a Wnt-independent mechanism regulates cardiomyocyte growth Proceedings of the National Academy of Sciences Syed Haq, Ashour Michael et al. 198
8 Stretch-activated pathways and left ventricular remodeling Journal of Cardiac Failure Thomas Force, Ashour Michael et al. 36
9 Differential Activation of Signal Transduction Pathways in Human Hearts With Hypertrophy Versus Advanced Heart Failure Circulation Syed Haq, Gabriel Choukroun et al. 355
10 Calcineurin Promotes Protein Kinase C and c-Jun NH2-terminal Kinase Activation in the Heart Journal of Biological Chemistry León J. De Windt, Hae W. Lim et al. 191
11 Glycogen Synthase Kinase-3β Is a Negative Regulator of Cardiomyocyte Hypertrophy The Journal of Cell Biology Syed Haq, Gabriel Choukroun et al. 337
12 Regulation of cardiac hypertrophy in vivo by the stress-activated protein kinases/c-Jun NH2-terminal kinases Journal of Clinical Investigation Gabriel Choukroun, Roger J. Hajjar et al. 152

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026