Risto Kerkelä
- Cardiology and Cardiovascular Medicine top 0.5%
- Molecular Biology top 5%
- Oncology top 2%
- Pulmonary and Respiratory Medicine top 5%
- Hematology top 2%
- Co-authors
- Thomas ForceMing Hui ChenHeikki RuskoahoKathleen C. WoulfeJohanna MaggaJean‐Bernard DurandJohanna UlvilaSampsa Pikkarainen
- Topics
- Cardiac Fibrosis and Remodeling (30 papers)Signaling Pathways in Disease (18 papers)Cardiomyopathy and Myosin Studies (12 papers)
- Partner nations
- FinlandUnited StatesHungary
In The Last Decade
Risto Kerkelä
84 papers receiving 4.5k citations
Hit Papers
Peers
Comparison fields: 5 of 124
- Cardiology and Cardiovascular Medicine 2.2k
- Molecular Biology 2.0k
- Oncology 1.1k
- Pulmonary and Respiratory Medicine 621
- Hematology 569
Countries citing papers authored by Risto Kerkelä
This map shows the geographic impact of Risto Kerkelä's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Risto Kerkelä with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Risto Kerkelä more than expected).
Fields of papers citing papers by Risto Kerkelä
This network shows the impact of papers produced by Risto Kerkelä. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Risto Kerkelä. The network helps show where Risto Kerkelä may publish in the future.
Co-authorship network of co-authors of Risto Kerkelä
This figure shows the co-authorship network connecting the top 25 collaborators of Risto Kerkelä. A scholar is included among the top collaborators of Risto Kerkelä based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Risto Kerkelä. Risto Kerkelä is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 2 | |
| 3 | 1 | |
| 4 | 2 | |
| 5 | 4 | |
| 6 | miR-1468-3p Promotes Aging-Related Cardiac Fibrosis | 1 |
| 7 | 16 | |
| 8 | 25 | |
| 9 | 93 | |
| 10 | 7 | |
| 11 | 12 | |
| 12 | 9 | |
| 13 | 14 | |
| 14 | 5 | |
| 15 | 194 | |
| 16 | 40 | |
| 17 | 84 | |
| 18 | Cardiotoxicity of the cancer therapeutic agent imatinib mesylatebreakdown → | 858 |
| 19 | 19 | |
| 20 | 70 |
About Risto Kerkelä
Risto Kerkelä is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Cancer Research, having authored 86 papers that have together received 4.6k indexed citations. Recurring topics across this work include Cardiac Fibrosis and Remodeling (30 papers), Signaling Pathways in Disease (18 papers) and Cardiomyopathy and Myosin Studies (12 papers). The work is most often cited by research in Cardiology and Cardiovascular Medicine (2.2k citations), Hematology (569 citations) and Oncology (1.1k citations). Risto Kerkelä has collaborated with scholars based in Finland, United States and Hungary. Frequent co-authors include Thomas Force, Ming Hui Chen, Heikki Ruskoaho, Kathleen C. Woulfe, Johanna Magga, Jean‐Bernard Durand, Johanna Ulvila, Sampsa Pikkarainen, Sergei Shevtsov and Olli Vuolteenaho. Their work appears in journals such as Proceedings of the National Academy of Sciences, The Lancet and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.