Stephen Lynn

1.2k citations
24 papers · 692 indexed · h-index 17

Impact in

Papers in

Stephen Lynn

23 papers receiving 675 citations

Peers

Stephen Lynn
Comparison fields: 5 of 80
  • Clinical Biochemistry 132
  • Sensory Systems 44
  • Physiology 32
  • Molecular Biology 462
  • Cell Biology 93
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Moeenaldeen AlSayed Saudi Arabia
Morad Khayat Israel
Marcello Niceta Italy
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Juliana Pérez‐Miguelsanz Spain
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Elizabeth Spellacy United Kingdom
Junko Kanno Japan
Shu-Chuan Chiang Taiwan
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Citations per field
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Citations per year

Countries citing papers authored by Stephen Lynn

Since Specialization
Citations

This map shows the geographic impact of Stephen Lynn's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stephen Lynn with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stephen Lynn more than expected).

Fields of papers citing papers by Stephen Lynn

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stephen Lynn. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stephen Lynn. The network helps show where Stephen Lynn may publish in the future.

Co-authors

The 25 scholars most cited alongside Stephen Lynn, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Stephen Lynn Line = papers co-authored together Stephen Lynn links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 24 papers — load more, or switch the sort, to bring in the rest.

#Work
1 200871
2 200267
3 200058
4 200551
5 200451
6 199942
7 200339
8 199836
9 199333
10 201431
11 200929
12 199528
13 200228
14 200526
15 201625
16
Collaborating to bring new therapies to the patient--the TREAT-NMD model.
200923
17 199518
18
Human cartilage aggrecan CS1 region contains cryptic T-cell recognition sites.
199314
19 19938
20 20177

About Stephen Lynn

Stephen Lynn is a scholar working on Molecular Biology, Clinical Biochemistry, Surgery, Cardiology and Cardiovascular Medicine and Physiology, having authored 24 papers that have together received 692 indexed citations. Recurring topics across this work include Metabolism and Genetic Disorders (5 papers), Ion channel regulation and function (5 papers), Mitochondrial Function and Pathology (5 papers), Adipose Tissue and Metabolism (3 papers), Ion Channels and Receptors (3 papers), Muscle Physiology and Disorders (3 papers), Pancreatic function and diabetes (2 papers) and Cell Adhesion Molecules Research (2 papers). The work is most often cited by research in Clinical Biochemistry (132 citations), Sensory Systems (44 citations), Physiology (32 citations), Molecular Biology (462 citations) and Cell Biology (93 citations). Stephen Lynn has collaborated with scholars based in United Kingdom, United States and France. Frequent co-authors include Douglass M. Turnbull, Mark Walker, James Gillespie, Patrick F. Chinnery, Volker Straub, Kate Bushby, Lynsey Cree, Stephen J. Yeaman, Timothy M. Frayling and Sandra L. Jackson. Their work appears in journals such as Diabetes, FEBS Letters, Diabetologia, Neuromuscular Disorders and Orphanet Journal of Rare Diseases.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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