Stefan Gaubatz

4.2k total citations · 1 hit paper
45 papers, 3.4k citations indexed

About

Stefan Gaubatz is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Stefan Gaubatz has authored 45 papers receiving a total of 3.4k indexed citations (citations by other indexed papers that have themselves been cited), including 36 papers in Molecular Biology, 27 papers in Oncology and 18 papers in Cell Biology. Recurrent topics in Stefan Gaubatz's work include Cancer-related Molecular Pathways (25 papers), Genomics and Chromatin Dynamics (15 papers) and Microtubule and mitosis dynamics (13 papers). Stefan Gaubatz is often cited by papers focused on Cancer-related Molecular Pathways (25 papers), Genomics and Chromatin Dynamics (15 papers) and Microtubule and mitosis dynamics (13 papers). Stefan Gaubatz collaborates with scholars based in Germany, United States and United Kingdom. Stefan Gaubatz's co-authors include David M. Livingston, Yoshihiro Nakatani, Kei‐ichiro Ishiguro, Hidesato Ogawa, Martin Eilers, Geoffrey J. Lindeman, Fabienne Schmit, Jason G. Wood, Stefanie Hauser and Albrecht Meichle and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Stefan Gaubatz

44 papers receiving 3.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

A Complex with Chromatin Modifiers That Occupies E2F- and Myc-Responsive Genes in G ₀ Cells

2002 610 citations Hidesato Ogawa, Kei‐ichiro Ishiguro et al. Science profile →

Peers

Stefan Gaubatz

ONCOL

GENET

Sophie E. Polo France

Frederick A. Dick Canada

J. Ross Chapman United Kingdom

Monique Verhaegen United States

Tatiana García‐Muse Spain

Juan Zalvide Spain

Margarida Ruas United Kingdom

Shun‐ichiro Iemura Japan

Rob M.F. Wolthuis Netherlands

Chul Geun Kim South Korea

Sophie E. Polo France

Stefan Gaubatz

4.4k ×1.6

1.3k ×0.9

ONCOL

284 ×0.6

366 ×1.1

GENET

413 ×1.4

Citations per year, relative to Stefan Gaubatz Stefan Gaubatz (= 1×) peers Sophie E. Polo

Countries citing papers authored by Stefan Gaubatz

Since Specialization

Citations

This map shows the geographic impact of Stefan Gaubatz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stefan Gaubatz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stefan Gaubatz more than expected).

Fields of papers citing papers by Stefan Gaubatz

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stefan Gaubatz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stefan Gaubatz. The network helps show where Stefan Gaubatz may publish in the future.

Co-authorship network of co-authors of Stefan Gaubatz

This figure shows the co-authorship network connecting the top 25 collaborators of Stefan Gaubatz. A scholar is included among the top collaborators of Stefan Gaubatz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stefan Gaubatz. Stefan Gaubatz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Schulte, Clemens, et al.. (2024). Inhibition of the YAP-MMB interaction and targeting NEK2 as potential therapeutic strategies for YAP-driven cancers. Oncogene. 43(8). 578–593. 5 indexed citations

Leone, Marina, Fulvia Ferrazzi, Janica L. Wiederstein, et al.. (2021). IQGAP3, a YAP Target, Is Required for Proper Cell-Cycle Progression and Genome Stability. Molecular Cancer Research. 19(10). 1712–1726. 14 indexed citations

Walz, Susanne, et al.. (2020). Interaction of YAP with the Myb-MuvB (MMB) complex defines a transcriptional program to promote the proliferation of cardiomyocytes. PLoS Genetics. 16(5). e1008818–e1008818. 24 indexed citations

Francke, Stephan, Pascal Wolter, Stefanie Hauser, et al.. (2016). An important role for Myb-MuvB and its target gene KIF23 in a mouse model of lung adenocarcinoma. Oncogene. 36(1). 110–121. 35 indexed citations

Kumari, Geeta, et al.. (2014). Induction of p21CIP1 Protein and Cell Cycle Arrest after Inhibition of Aurora B Kinase Is Attributed to Aneuploidy and Reactive Oxygen Species. Journal of Biological Chemistry. 289(23). 16072–16084. 29 indexed citations

Esterlechner, Jasmina, et al.. (2013). LIN9, a Subunit of the DREAM Complex, Regulates Mitotic Gene Expression and Proliferation of Embryonic Stem Cells. PLoS ONE. 8(5). e62882–e62882. 17 indexed citations

Kumari, Geeta, et al.. (2013). A role for p38 in transcriptional elongation of p21^CIP1in response to Aurora B inhibition. Cell Cycle. 12(13). 2051–2060. 14 indexed citations

Houben, Roland, Christian Adam, Sonja Hesbacher, et al.. (2011). An intact retinoblastoma protein‐binding site in Merkel cell polyomavirus large T antigen is required for promoting growth of Merkel cell carcinoma cells. International Journal of Cancer. 130(4). 847–856. 163 indexed citations

Hauser, Stefanie, et al.. (2011). Loss of LIN9, a member of the DREAM complex, cooperates with SV40 large T antigen to induce genomic instability and anchorage-independent growth. Oncogene. 31(14). 1859–1868. 16 indexed citations

10.

Meierjohann, Svenja, Anita Hufnagel, Elisabeth Wende, et al.. (2010). MMP13 mediates cell cycle progression in melanocytes and melanoma cells: in vitro studies of migration and proliferation. Molecular Cancer. 9(1). 201–201. 50 indexed citations

11.

Gaubatz, Stefan, et al.. (2009). B-MYB Is Required for Recovery from the DNA Damage–Induced G2 Checkpoint in p53 Mutant Cells. Cancer Research. 69(9). 4073–4080. 73 indexed citations

12.

Schmit, Fabienne, et al.. (2009). LIN54 is an essential core subunit of the DREAM/LINC complex that binds to the cdc2 promoter in a sequence‐specific manner. FEBS Journal. 276(19). 5703–5716. 86 indexed citations

13.

Wagner, Toni U., et al.. (2009). lin9 Is Required for Mitosis and Cell Survival during Early Zebrafish Development. Journal of Biological Chemistry. 284(19). 13119–13127. 10 indexed citations

14.

Schmit, Fabienne, Michael Korenjak, Claudia Franke, et al.. (2007). LINC, a Human Complex That is Related to pRB-Containing Complexes in Invertebrates Regulates the Expression of G₂/M Genes. Cell Cycle. 6(15). 1903–1913. 157 indexed citations

15.

Schäfer, Andrea, Nina Reichert, José Luís Barbero, et al.. (2005). Silencing of the Meiotic Genes SMC1β and STAG3 in Somatic Cells by E2F6. Journal of Biological Chemistry. 280(50). 41380–41386. 27 indexed citations

16.

Gagrica, Sladjana, Stefanie Hauser, Ingrid Kolfschoten, et al.. (2004). Inhibition of oncogenic transformation by mammalian Lin‐9, a pRB‐associated protein. The EMBO Journal. 23(23). 4627–4638. 52 indexed citations

17.

Giangrande, Paloma H., Wencheng Zhu, Susanne Schlisio, et al.. (2004). A role for E2F6 in distinguishing G1/S- and G2/M-specific transcription. Genes & Development. 18(23). 2941–2951. 87 indexed citations

18.

Ogawa, Hidesato, Kei‐ichiro Ishiguro, Stefan Gaubatz, David M. Livingston, & Yoshihiro Nakatani. (2002). A Complex with Chromatin Modifiers That Occupies E2F- and Myc-Responsive Genes in G ₀ Cells. Science. 296(5570). 1132–1136. 610 indexed citations breakdown →

19.

Gaubatz, Stefan, Geoffrey J. Lindeman, Seiichi Ishida, et al.. (2000). E2F4 and E2F5 Play an Essential Role in Pocket Protein–Mediated G1 Control. Molecular Cell. 6(3). 729–735. 229 indexed citations

20.

Elliott, Katherine J., Daitoku Sakamuro, Wei Du, et al.. (1999). Bin1 functionally interacts with Myc and inhibits cell proliferation via multiple mechanisms. Oncogene. 18(24). 3564–3573. 98 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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