J. Ross Chapman

8.0k total citations · 3 hit papers
25 papers, 4.9k citations indexed

About

J. Ross Chapman is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, J. Ross Chapman has authored 25 papers receiving a total of 4.9k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 12 papers in Oncology and 3 papers in Cancer Research. Recurrent topics in J. Ross Chapman's work include DNA Repair Mechanisms (24 papers), CRISPR and Genetic Engineering (12 papers) and PARP inhibition in cancer therapy (10 papers). J. Ross Chapman is often cited by papers focused on DNA Repair Mechanisms (24 papers), CRISPR and Genetic Engineering (12 papers) and PARP inhibition in cancer therapy (10 papers). J. Ross Chapman collaborates with scholars based in United Kingdom, Switzerland and United States. J. Ross Chapman's co-authors include Simon J. Boulton, Martin R.G. Taylor, Stephen P. Jackson, Serge Gravel, Christine Magill, Stephanie Panier, Laurence Pelletier, Richard Chahwan, Daniel Durocher and Timothy M. Thomson and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

J. Ross Chapman

24 papers receiving 4.8k citations

Hit Papers

3 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Playing the End Game: DNA Double-Strand Break Repair Pathway Choice

2012 1.2k citations J. Ross Chapman, Martin R.G. Taylor et al. Molecular Cell profile →
Orchestration of the DNA-Damage Response by the RNF8 Ubiquitin Ligase

2007 719 citations Nadine K. Kolas, J. Ross Chapman et al. Science profile →
RIF1 Is Essential for 53BP1-Dependent Nonhomologous End Joining and Suppression of DNA Double-Strand Break Resection

2013 479 citations J. Ross Chapman, Patricia Barral et al. Molecular Cell profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
J. Ross Chapman United Kingdom	21	4.6k	1.7k	608	532	470	25	4.9k
Julia Coates United Kingdom	19	4.3k 0.9×	1.6k 0.9×	712 1.2×	424 0.8×	538 1.1×	22	4.5k
Didier Trouche France	36	5.1k 1.1×	1.8k 1.1×	584 1.0×	613 1.2×	359 0.8×	75	5.8k
Zuzana Hořejšı́ United Kingdom	16	3.3k 0.7×	1.6k 0.9×	674 1.1×	339 0.6×	489 1.0×	19	3.8k
Sophie E. Polo France	26	4.4k 1.0×	1.3k 0.7×	413 0.7×	366 0.7×	284 0.6×	44	4.8k
Anja Groth Denmark	36	5.4k 1.2×	854 0.5×	364 0.6×	490 0.9×	496 1.1×	66	6.0k
Stephanie Panier Canada	20	4.1k 0.9×	1.6k 0.9×	424 0.7×	393 0.7×	406 0.9×	21	4.5k
Andrew J. Pierce United States	29	5.1k 1.1×	1.8k 1.0×	1.1k 1.8×	972 1.8×	388 0.8×	76	5.8k
Phillip B. Carpenter United States	23	3.6k 0.8×	1.0k 0.6×	610 1.0×	398 0.7×	442 0.9×	31	4.0k
Stefan Gaubatz Germany	28	2.7k 0.6×	1.4k 0.8×	305 0.5×	343 0.6×	462 1.0×	45	3.4k
Dana Branzei Italy	38	5.0k 1.1×	986 0.6×	965 1.6×	659 1.2×	1.0k 2.2×	84	5.3k

Countries citing papers authored by J. Ross Chapman

Since Specialization

Citations

This map shows the geographic impact of J. Ross Chapman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. Ross Chapman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. Ross Chapman more than expected).

Fields of papers citing papers by J. Ross Chapman

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J. Ross Chapman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. Ross Chapman. The network helps show where J. Ross Chapman may publish in the future.

Co-authorship network of co-authors of J. Ross Chapman

This figure shows the co-authorship network connecting the top 25 collaborators of J. Ross Chapman. A scholar is included among the top collaborators of J. Ross Chapman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J. Ross Chapman. J. Ross Chapman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Ruan, Dike, Marieke Everts, J. Ross Chapman, et al.. (2025). CIP2A mediates mitotic recruitment of SLX4/MUS81/XPF to resolve replication stress-induced DNA lesions. Nature Communications. 17(1). 13–13.

Metson, Jean, Robert Parker, Catarina Oliveira, et al.. (2024). Shieldin and CST co-orchestrate DNA polymerase-dependent tailed-end joining reactions independently of 53BP1-governed repair pathway choice. Nature Structural & Molecular Biology. 32(1). 86–97. 6 indexed citations

Jones, Samuel E., J.V. Bannister, Iolanda Vendrell, et al.. (2024). BLM and BRCA1-BARD1 coordinate complementary mechanisms of joint DNA molecule resolution. Molecular Cell. 84(4). 640–658.e10. 8 indexed citations

Becker, Jordan R., et al.. (2021). BARD1 reads H2A lysine 15 ubiquitination to direct homologous recombination. Nature. 596(7872). 433–437. 120 indexed citations

Lambrus, Bramwell G., Rebecca Marlow, Phillip M. Scott, et al.. (2020). Targeting TRIM37-driven centrosome dysfunction in 17q23-amplified breast cancer. Nature. 585(7825). 447–452. 77 indexed citations

Nakamura, Kyosuke, Giulia Saredi, Jordan R. Becker, et al.. (2019). H4K20me0 recognition by BRCA1–BARD1 directs homologous recombination to sister chromatids. Nature Cell Biology. 21(3). 311–318. 150 indexed citations

Barazas, Marco, Stefano Annunziato, Stephen J. Pettitt, et al.. (2018). The CST Complex Mediates End Protection at Double-Strand Breaks and Promotes PARP Inhibitor Sensitivity in BRCA1-Deficient Cells. Cell Reports. 23(7). 2107–2118. 102 indexed citations

Botuyan, Maria Victoria, Gaofeng Cui, Pascal Drané, et al.. (2018). Mechanism of 53BP1 activity regulation by RNA-binding TIRR and a designer protein. Nature Structural & Molecular Biology. 25(7). 591–600. 35 indexed citations

Oliveira, Catarina, Jordan R. Becker, Daniela Moralli, et al.. (2018). 53BP1 cooperation with the REV7–shieldin complex underpins DNA structure-specific NHEJ. Nature. 560(7716). 122–127. 215 indexed citations

10.

Becker, Jordan R., Raquel Cuella-Martin, Marco Barazas, et al.. (2018). The ASCIZ-DYNLL1 axis promotes 53BP1-dependent non-homologous end joining and PARP inhibitor sensitivity. Nature Communications. 9(1). 5406–5406. 83 indexed citations

11.

Cuella-Martin, Raquel, et al.. (2016). 53BP1 Integrates DNA Repair and p53-Dependent Cell Fate Decisions via Distinct Mechanisms. Molecular Cell. 64(1). 51–64. 127 indexed citations

12.

Chapman, J. Ross, Patricia Barral, Jean‐Baptiste Vannier, et al.. (2013). RIF1 Is Essential for 53BP1-Dependent Nonhomologous End Joining and Suppression of DNA Double-Strand Break Resection. Molecular Cell. 49(5). 858–871. 479 indexed citations breakdown →

13.

Polo, Sophie E., Andrew N. Blackford, J. Ross Chapman, et al.. (2012). Regulation of DNA-End Resection by hnRNPU-like Proteins Promotes DNA Double-Strand Break Signaling and Repair. Molecular Cell. 45(4). 505–516. 143 indexed citations

14.

Chapman, J. Ross, Martin R.G. Taylor, & Simon J. Boulton. (2012). Playing the End Game: DNA Double-Strand Break Repair Pathway Choice. Molecular Cell. 47(4). 497–510. 1246 indexed citations breakdown →

15.

Townsend, Kelly, Helen Mason, Andrew N. Blackford, et al.. (2009). Mediator of DNA Damage Checkpoint 1 (MDC1) Regulates Mitotic Progression. Journal of Biological Chemistry. 284(49). 33939–33948. 39 indexed citations

16.

Taylor, Elaine M., et al.. (2009). The Mre11/Rad50/Nbs1 complex functions in resection-based DNA end joining in Xenopus laevis. Nucleic Acids Research. 38(2). 441–454. 41 indexed citations

17.

Chapman, J. Ross, Julie A. Clapperton, L.F. Haire, et al.. (2009). A Supramodular FHA/BRCT-Repeat Architecture Mediates Nbs1 Adaptor Function in Response to DNA Damage. Cell. 139(1). 100–111. 133 indexed citations

18.

Gravel, Serge, J. Ross Chapman, Christine Magill, & Stephen P. Jackson. (2008). DNA helicases Sgs1 and BLM promote DNA double-strand break resection. Genes & Development. 22(20). 2767–2772. 472 indexed citations

19.

Kolas, Nadine K., J. Ross Chapman, Shinichiro Nakada, et al.. (2007). Orchestration of the DNA-Damage Response by the RNF8 Ubiquitin Ligase. Science. 318(5856). 1637–1640. 719 indexed citations breakdown →

20.

Jones, Rhiannon E., J. Ross Chapman, Chandrakala Puligilla, et al.. (2003). XRad17 Is Required for the Activation of XChk1 But Not XCds1 during Checkpoint Signaling inXenopus. Molecular Biology of the Cell. 14(9). 3898–3910. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact