Hidesato Ogawa

3.5k total citations · 2 hit papers
38 papers, 2.7k citations indexed

About

Hidesato Ogawa is a scholar working on Molecular Biology, Genetics and Epidemiology. According to data from OpenAlex, Hidesato Ogawa has authored 38 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Molecular Biology, 21 papers in Genetics and 7 papers in Epidemiology. Recurrent topics in Hidesato Ogawa's work include Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (8 papers), Estrogen and related hormone effects (8 papers) and Autophagy in Disease and Therapy (6 papers). Hidesato Ogawa is often cited by papers focused on Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (8 papers), Estrogen and related hormone effects (8 papers) and Autophagy in Disease and Therapy (6 papers). Hidesato Ogawa collaborates with scholars based in Japan, United States and Poland. Hidesato Ogawa's co-authors include Yoshihiro Nakatani, Kei‐ichiro Ishiguro, David M. Livingston, Stefan Gaubatz, Kazuhiko Umesono, Fei Lan, Margaret Po-shan Luke, Yujiang Geno Shi, Yang Shi and Guangchao Sui and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Hidesato Ogawa

38 papers receiving 2.6k citations

Hit Papers

Coordinated histone modifications mediated by a CtBP co-r... 2002 2026 2010 2018 2003 2002 200 400 600

Peers

Hidesato Ogawa
Comparison fields: 5 of 111
  • Molecular Biology 2.1k
  • Genetics 765
  • Oncology 289
  • Cancer Research 195
  • Epidemiology 162
Ramiro Ramírez‐Solis United Kingdom
Marek Bartkuhn Germany
Masafumi Muratani Japan
Michèle Sawadogo United States
Jan Parker‐Thornburg United States
Dario De Cesare Italy
David J. Picketts Canada
Frédérique Gay United States
Chih‐Lin Hsieh United States
Stefan G. E. Roberts United Kingdom
Ramiro Ramírez‐Solis United Kingdom View profile →
Citations per field, relative to Hidesato Ogawa
Hidesato Ogawa · 1×
Citations per year, relative to Hidesato Ogawa
Hidesato Ogawa · 1×

Countries citing papers authored by Hidesato Ogawa

Since Specialization
Citations

This map shows the geographic impact of Hidesato Ogawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hidesato Ogawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hidesato Ogawa more than expected).

Fields of papers citing papers by Hidesato Ogawa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hidesato Ogawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hidesato Ogawa. The network helps show where Hidesato Ogawa may publish in the future.

Co-authorship network of co-authors of Hidesato Ogawa

This figure shows the co-authorship network connecting the top 25 collaborators of Hidesato Ogawa. A scholar is included among the top collaborators of Hidesato Ogawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hidesato Ogawa. Hidesato Ogawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 10
2 28
3 17
4 12
5 27
6 1
7
Depletion of autophagy receptor p62/SQSTM1 enhances the efficiency of gene delivery in mammalian cells
1
8 7
9 57
10 54
11 13
12 65
13 21
14 18
15
Coordinated histone modifications mediated by a CtBP co-repressor complex breakdown →
647
16
A Complex with Chromatin Modifiers That Occupies E2F- and Myc-Responsive Genes in G 0 Cells breakdown →
610
17 258
18 152
19 36
20 10

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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2026