Shintaro Sato
- Immunology top 0.01%
- Molecular Biology top 0.5%
- Epidemiology top 0.2%
- Cancer Research top 0.1%
- Oncology top 0.5%
- Co-authors
- Shizuo AkiraOsamu TakeuchiKiyoshi TakedaTaro KawaiKatsuaki HoshinoMasahiro YamamotoHiroaki HemmiHideki Sanjo
- Topics
- Immune Response and Inflammation (49 papers)NF-κB Signaling Pathways (33 papers)Immune Cell Function and Interaction (19 papers)
- Partner nations
- JapanUnited StatesGermany
In The Last Decade
Shintaro Sato
104 papers receiving 33.8k citations
Hit Papers
Peers
Comparison fields: 5 of 155
- Immunology 26.0k
- Molecular Biology 9.4k
- Epidemiology 5.9k
- Cancer Research 5.2k
- Oncology 3.4k
Countries citing papers authored by Shintaro Sato
This map shows the geographic impact of Shintaro Sato's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shintaro Sato with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shintaro Sato more than expected).
Fields of papers citing papers by Shintaro Sato
This network shows the impact of papers produced by Shintaro Sato. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shintaro Sato. The network helps show where Shintaro Sato may publish in the future.
Co-authorship network of co-authors of Shintaro Sato
This figure shows the co-authorship network connecting the top 25 collaborators of Shintaro Sato. A scholar is included among the top collaborators of Shintaro Sato based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shintaro Sato. Shintaro Sato is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 2 | |
| 3 | 0 | |
| 4 | 0 | |
| 5 | 6 | |
| 6 | 11 | |
| 7 | 13 | |
| 8 | 10 | |
| 9 | 5 | |
| 10 | 7 | |
| 11 | LGP2 is a positive regulator of RIG-I– and MDA5-mediated antiviral responsesbreakdown → | 519 |
| 12 | 1 | |
| 13 | 32 | |
| 14 | 152 | |
| 15 | 71 | |
| 16 | Role of Adaptor TRIF in the MyD88-Independent Toll-Like Receptor Signaling Pathwaybreakdown → | 2604 |
| 17 | Toll/IL-1 Receptor Domain-Containing Adaptor Inducing IFN-β (TRIF) Associates with TNF Receptor-Associated Factor 6 and TANK-Binding Kinase 1, and Activates Two Distinct Transcription Factors, NF-κB and IFN-Regulatory Factor-3, in the Toll-Like Receptor Signalingbreakdown → | 568 |
| 18 | Cutting Edge: Role of Toll-Like Receptor 1 in Mediating Immune Response to Microbial Lipoproteinsbreakdown → | 1054 |
| 19 | Lipopolysaccharide Stimulates the MyD88-Independent Pathway and Results in Activation of IFN-Regulatory Factor 3 and the Expression of a Subset of Lipopolysaccharide-Inducible Genesbreakdown → | 892 |
| 20 | 337 |
About Shintaro Sato
Shintaro Sato is a scholar working on Immunology, Cancer Research and Endocrinology, having authored 111 papers that have together received 34.4k indexed citations. Recurring topics across this work include Immune Response and Inflammation (49 papers), NF-κB Signaling Pathways (33 papers) and Immune Cell Function and Interaction (19 papers). The work is most often cited by research in Immunology (26.0k citations), Cancer Research (5.2k citations) and Microbiology (1.9k citations). Shintaro Sato has collaborated with scholars based in Japan, United States and Germany. Frequent co-authors include Shizuo Akira, Osamu Takeuchi, Kiyoshi Takeda, Taro Kawai, Katsuaki Hoshino, Masahiro Yamamoto, Hiroaki Hemmi, Hideki Sanjo, Tsuneyasu Kaisho and Satoshi Uematsu. Their work appears in journals such as Nature, Science and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.