Serena Altilia
- Co-authors
- Claudio FranceschiIgor B. RoninsonJiaxin LiangEugenia V. BroudeMengqian ChenStefano SalvioliFederica SeviniDominique Lison
- Topics
- Cancer-related Molecular Pathways (9 papers)Mitochondrial Function and Pathology (4 papers)Advanced Breast Cancer Therapies (4 papers)
- Cited by
- AgingOncologyNephrology
- Journals
- Proceedings of the National Academy of SciencesSHILAP Revista de lepidopterologíaCancer Research
- Partner nations
- ItalyUnited StatesGreece
In The Last Decade
Serena Altilia
20 papers receiving 582 citations
Peers
Comparison fields: 5 of 117
- Molecular Biology 268
- Oncology 162
- Pulmonary and Respiratory Medicine 110
- Genetics 68
- Immunology 67
Countries citing papers authored by Serena Altilia
This map shows the geographic impact of Serena Altilia's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Serena Altilia with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Serena Altilia more than expected).
Fields of papers citing papers by Serena Altilia
This network shows the impact of papers produced by Serena Altilia. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Serena Altilia. The network helps show where Serena Altilia may publish in the future.
Co-authorship network of co-authors of Serena Altilia
This figure shows the co-authorship network connecting the top 25 collaborators of Serena Altilia. A scholar is included among the top collaborators of Serena Altilia based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Serena Altilia. Serena Altilia is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 68 | |
| 2 | 1 | |
| 3 | 94 | |
| 4 | 89 | |
| 5 | 1 | |
| 6 | 8 | |
| 7 | 1 | |
| 8 | 1 | |
| 9 | 23 | |
| 10 | 6 | |
| 11 | 1 | |
| 12 | 58 | |
| 13 | 7 | |
| 14 | Longevity genes across species: conservation versus evolvability | 1 |
| 15 | 49 | |
| 16 | 12 | |
| 17 | 56 | |
| 18 | 28 | |
| 19 | 85 | |
| 20 | Role of crystalline structure and state of the surface on the in vitro cytotoxicity and trasforming potency of silica dusts | 1 |
About Serena Altilia
Serena Altilia is a scholar working on Aging, Oncology and Virology, having authored 20 papers that have together received 590 indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (9 papers), Mitochondrial Function and Pathology (4 papers) and Advanced Breast Cancer Therapies (4 papers). The work is most often cited by research in Aging (28 citations), Oncology (162 citations) and Nephrology (39 citations). Serena Altilia has collaborated with scholars based in Italy, United States and Greece. Frequent co-authors include Claudio Franceschi, Igor B. Roninson, Jiaxin Liang, Eugenia V. Broude, Mengqian Chen, Stefano Salvioli, Federica Sevini, Dominique Lison, Umberto Saffiotti and Bice Fubini. Their work appears in journals such as Proceedings of the National Academy of Sciences, SHILAP Revista de lepidopterología and Cancer Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.