Eugenia V. Broude

5.4k total citations · 2 hit papers
56 papers, 4.1k citations indexed

About

Eugenia V. Broude is a scholar working on Oncology, Molecular Biology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Eugenia V. Broude has authored 56 papers receiving a total of 4.1k indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Oncology, 29 papers in Molecular Biology and 16 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Eugenia V. Broude's work include Cancer-related Molecular Pathways (23 papers), Advanced Breast Cancer Therapies (15 papers) and Ubiquitin and proteasome pathways (7 papers). Eugenia V. Broude is often cited by papers focused on Cancer-related Molecular Pathways (23 papers), Advanced Breast Cancer Therapies (15 papers) and Ubiquitin and proteasome pathways (7 papers). Eugenia V. Broude collaborates with scholars based in United States, Hungary and Russia. Eugenia V. Broude's co-authors include Igor B. Roninson, Bey-Dih Chang, Jing Fang, Yongzhi Xuan, Hongming Zhu, Amer Alam, Kaspar P. Locher, Julia Kowal, T. V. Kalinichenko and Jason C. Poole and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Cancer Research.

In The Last Decade

Eugenia V. Broude

52 papers receiving 4.0k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

If not apoptosis, then what? Treatment-induced senescence and mitotic catastrophe in tumor cells

2001 601 citations Igor B. Roninson, Eugenia V. Broude et al. profile →
Structural insight into substrate and inhibitor discrimination by human P-glycoprotein

2019 372 citations Amer Alam, Julia Kowal et al. Science profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Eugenia V. Broude United States	28	2.7k	1.8k	727	484	457	56	4.1k
W. Robert Bishop United States	35	4.4k 1.7×	1.9k 1.0×	398 0.5×	685 1.4×	527 1.2×	71	5.9k
Olivier Coqueret France	31	2.1k 0.8×	1.4k 0.8×	312 0.4×	332 0.7×	565 1.2×	69	3.4k
Annamaria Biroccio Italy	41	3.7k 1.4×	1.3k 0.7×	747 1.0×	191 0.4×	679 1.5×	117	5.1k
Ajay Rana United States	33	2.2k 0.8×	998 0.6×	342 0.5×	339 0.7×	568 1.2×	97	3.7k
Young‐Ah Suh South Korea	29	3.1k 1.2×	1.4k 0.8×	710 1.0×	276 0.6×	742 1.6×	58	4.7k
Carmen Blanco‐Aparicio Spain	31	2.6k 1.0×	936 0.5×	541 0.7×	197 0.4×	590 1.3×	79	3.9k
Hiroyuki Seimiya Japan	37	3.9k 1.5×	900 0.5×	1.0k 1.4×	298 0.6×	1.1k 2.3×	117	5.1k
Jordan S. Fridman United States	22	3.6k 1.4×	2.0k 1.1×	531 0.7×	222 0.5×	679 1.5×	41	6.0k
Donatella Del Bufalo Italy	42	3.0k 1.1×	1.4k 0.8×	300 0.4×	354 0.7×	1.0k 2.3×	133	4.7k
Tohru Kamata Japan	31	2.7k 1.0×	881 0.5×	656 0.9×	464 1.0×	495 1.1×	74	4.6k

Countries citing papers authored by Eugenia V. Broude

Since Specialization

Citations

This map shows the geographic impact of Eugenia V. Broude's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eugenia V. Broude with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eugenia V. Broude more than expected).

Fields of papers citing papers by Eugenia V. Broude

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eugenia V. Broude. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eugenia V. Broude. The network helps show where Eugenia V. Broude may publish in the future.

Co-authorship network of co-authors of Eugenia V. Broude

This figure shows the co-authorship network connecting the top 25 collaborators of Eugenia V. Broude. A scholar is included among the top collaborators of Eugenia V. Broude based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eugenia V. Broude. Eugenia V. Broude is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Kumari, Asha, et al.. (2025). Targeting Mediator Kinase Cyclin-Dependent Kinases 8/19 Potentiates Chemotherapeutic Responses, Reverses Tumor Growth, and Prolongs Survival from Ovarian Clear Cell Carcinoma. Cancers. 17(6). 941–941.

Vorobyeva, Nadezhda E., Alexander Tyakht, Jing Li, et al.. (2025). CDK8/19 inhibition attenuates G1 arrest induced by BCR-ABL antagonists and accelerates death of chronic myelogenous leukemia cells. Cell Death Discovery. 11(1). 62–62.

Aksenova, Marina, Aliaksandra Sikirzhytskaya, Vitali Sikirzhytski, et al.. (2024). Suppression of HIV-TAT and cocaine-induced neurotoxicity and inflammation by cell penetrable itaconate esters. Journal of NeuroVirology. 30(4). 337–352. 1 indexed citations

Штро, Анна А., Victor V. Tatarskiy, Аlexander А. Shtil, et al.. (2023). Inhibition of Cyclin-Dependent Kinases 8/19 Restricts Bacterial and Virus-Induced Inflammatory Responses in Monocytes. Viruses. 15(6). 1292–1292. 1 indexed citations

Zhang, Li, Jing Li, Eugenia V. Broude, et al.. (2023). Abstract 5018: PROTAC degraders of CDK8/CDK19 Mediator kinases potently suppress multiple myeloma proliferation. Cancer Research. 83(7_Supplement). 5018–5018. 1 indexed citations

Aksenova, Marina, David J. Oliver, Elina Levina, et al.. (2022). Depletion of COPI in cancer cells: the role of reactive oxygen species in the induction of lipid accumulation, noncanonical lipophagy and apoptosis. Molecular Biology of the Cell. 33(14). ar135–ar135. 8 indexed citations

Sikirzhytski, Vitali, Marina Aksenova, Eugenia V. Broude, et al.. (2020). Pharmacological inhibition of DEAD-Box RNA Helicase 3 attenuates stress granule assembly. Biochemical Pharmacology. 182. 114280–114280. 27 indexed citations

Alam, Amer, Julia Kowal, Eugenia V. Broude, Igor B. Roninson, & Kaspar P. Locher. (2019). Structural insight into substrate and inhibitor discrimination by human P-glycoprotein. Science. 363(6428). 753–756. 372 indexed citations breakdown →

Singh, Priyanka, Prabhjot Kaur, Tapasya Srivastava, et al.. (2018). Inhibin Is a Novel Paracrine Factor for Tumor Angiogenesis and Metastasis. Cancer Research. 78(11). 2978–2989. 30 indexed citations

10.

Liang, Jiaxin, Mengqian Chen, Daniel Hughes, et al.. (2018). CDK8 Selectively Promotes the Growth of Colon Cancer Metastases in the Liver by Regulating Gene Expression of TIMP3 and Matrix Metalloproteinases. Cancer Research. 78(23). 6594–6606. 68 indexed citations

11.

Chumanevich, Alexander A., Jiaxin Liang, Michael L. Gatza, et al.. (2018). Mediator kinase CDK8/CDK19 drives YAP1-dependent BMP4-induced EMT in cancer. Oncogene. 37(35). 4792–4808. 48 indexed citations

12.

Broude, Eugenia V., Balázs Győrffy, Alexander A. Chumanevich, et al.. (2015). Expression of CDK8 and CDK8-interacting Genes as Potential Biomarkers in Breast Cancer. Current Cancer Drug Targets. 15(8). 739–749. 62 indexed citations

13.

Ossovskaya, Valeria, Lingyun Li, Eugenia V. Broude, et al.. (2009). Abstract #5552: BSI-201 enhances the activity of multiple classes of cytotoxic agents and irradiation in triple negative breast cancer. Cancer Research. 69. 5552–5552. 13 indexed citations

14.

Demidenko, Zoya N., et al.. (2008). Mechanism of G1-like arrest by low concentrations of paclitaxel: next cell cycle p53-dependent arrest with sub G1 DNA content mediated by prolonged mitosis. Oncogene. 27(32). 4402–4410. 89 indexed citations

15.

Broude, Eugenia V., Mari E. Swift, Claire Vivo, et al.. (2007). p21Waf1/Cip1/Sdi1 mediates retinoblastoma protein degradation. Oncogene. 26(48). 6954–6958. 67 indexed citations

16.

Chang, Bey-Dih, Eugenia V. Broude, Jing Fang, et al.. (2000). p21Waf1/Cip1/Sdi1-induced growth arrest is associated with depletion of mitosis-control proteins and leads to abnormal mitosis and endoreduplication in recovering cells. Oncogene. 19(17). 2165–2170. 141 indexed citations

17.

Chang, Bey-Dih, Yongzhi Xuan, Eugenia V. Broude, et al.. (1999). Role of p53 and p21waf1/cip1 in senescence-like terminal proliferation arrest induced in human tumor cells by chemotherapeutic drugs. Oncogene. 18(34). 4808–4818. 327 indexed citations

18.

Broude, Eugenia V., Marietta McAtee, Melinda Kelley, & B.S. Bregman. (1999). Fetal Spinal Cord Transplants and Exogenous Neurotrophic Support Enhance c-Jun Expression in Mature Axotomized Neurons after Spinal Cord Injury. Experimental Neurology. 155(1). 65–78. 35 indexed citations

19.

Bregman, B.S., Eugenia V. Broude, Marietta McAtee, & Melinda Kelley. (1998). Transplants and Neurotrophic Factors Prevent Atrophy of Mature CNS Neurons after Spinal Cord Injury. Experimental Neurology. 149(1). 13–27. 120 indexed citations

20.

Broude, Eugenia V., Marietta McAtee, Melinda Kelley, & B.S. Bregman. (1997). c-Jun Expression in Adult Rat Dorsal Root Ganglion Neurons: Differential Response after Central or Peripheral Axotomy. Experimental Neurology. 148(1). 367–377. 115 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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