Sarah L. Miles

1.1k total citations
27 papers, 723 citations indexed

About

Sarah L. Miles is a scholar working on Molecular Biology, Sensory Systems and Toxicology. According to data from OpenAlex, Sarah L. Miles has authored 27 papers receiving a total of 723 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 8 papers in Sensory Systems and 5 papers in Toxicology. Recurrent topics in Sarah L. Miles's work include Ion Channels and Receptors (8 papers), Bioactive Compounds and Antitumor Agents (5 papers) and Piperaceae Chemical and Biological Studies (5 papers). Sarah L. Miles is often cited by papers focused on Ion Channels and Receptors (8 papers), Bioactive Compounds and Antitumor Agents (5 papers) and Piperaceae Chemical and Biological Studies (5 papers). Sarah L. Miles collaborates with scholars based in United States, Netherlands and Canada. Sarah L. Miles's co-authors include Richard M. Niles, Adam Fischer, Piyali Dasgupta, Margaret McFarland, Kathleen C. Brown, Monica Valentovic, Nicholas Nolan, Austin T. Akers, Maria Tria Tirona and Jamie K. Lau and has published in prestigious journals such as The FASEB Journal, Biochemical and Biophysical Research Communications and International Journal of Molecular Sciences.

In The Last Decade

Sarah L. Miles

27 papers receiving 703 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Sarah L. Miles United States 14 308 127 113 80 74 27 723
Kevin Zhai Qatar 17 419 1.4× 36 0.3× 12 0.1× 40 0.5× 69 0.9× 26 911
Ke Qian China 14 252 0.8× 29 0.2× 11 0.1× 49 0.6× 51 0.7× 35 607
Yosuke Nakazawa Japan 16 345 1.1× 8 0.1× 118 1.0× 25 0.3× 100 1.4× 66 818
Aistė Jekabsone Lithuania 19 446 1.4× 58 0.5× 5 0.0× 19 0.2× 248 3.4× 32 1.1k
Hyonok Yoon South Korea 14 360 1.2× 37 0.3× 8 0.1× 8 0.1× 62 0.8× 28 889
Jun Deguchi Japan 20 281 0.9× 91 0.7× 11 0.1× 11 0.1× 12 0.2× 49 884
Nihat Dilsiz Türkiye 17 500 1.6× 19 0.1× 71 0.6× 3 0.0× 78 1.1× 36 860
Entaz Bahar South Korea 11 271 0.9× 22 0.2× 8 0.1× 9 0.1× 51 0.7× 18 687
María L. Rodríguez Spain 10 374 1.2× 10 0.1× 109 1.0× 6 0.1× 72 1.0× 12 837
M. Balasubramanyam United States 11 266 0.9× 5 0.0× 37 0.3× 43 0.5× 76 1.0× 16 541

Countries citing papers authored by Sarah L. Miles

Since Specialization
Citations

This map shows the geographic impact of Sarah L. Miles's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sarah L. Miles with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sarah L. Miles more than expected).

Fields of papers citing papers by Sarah L. Miles

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sarah L. Miles. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sarah L. Miles. The network helps show where Sarah L. Miles may publish in the future.

Co-authorship network of co-authors of Sarah L. Miles

This figure shows the co-authorship network connecting the top 25 collaborators of Sarah L. Miles. A scholar is included among the top collaborators of Sarah L. Miles based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sarah L. Miles. Sarah L. Miles is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Brown, Kathleen C., et al.. (2024). An Experimental Protocol for the Boyden Chamber Invasion Assay With Absorbance Readout. BIO-PROTOCOL. 14(1350). e5040–e5040. 1 indexed citations
2.
Brown, Kathleen C., Ashley Cox, Sarah L. Miles, et al.. (2024). Anti-cancer activity of capsaicin and its analogs in gynecological cancers. Advances in cancer research. 164. 241–281. 2 indexed citations
3.
Pulido, José S., Patricia Chévez‐Barrios, Bin S. Teh, et al.. (2023). MULTIPLYING BROWN SPOTS. Retinal Cases & Brief Reports. 18(5). 642–646. 1 indexed citations
4.
Cox, Ashley, Kathleen C. Brown, Sarah L. Miles, et al.. (2022). Anti-cancer activity of sustained release capsaicin formulations. Pharmacology & Therapeutics. 238. 108177–108177. 59 indexed citations
5.
Gao, Ying, Sarah L. Miles, Piyali Dasgupta, et al.. (2021). Trichodermin Induces G0/G1 Cell Cycle Arrest by Inhibiting c-Myc in Ovarian Cancer Cells and Tumor Xenograft-Bearing Mice. International Journal of Molecular Sciences. 22(9). 5022–5022. 13 indexed citations
6.
Brown, Kathleen C., Krista L. Denning, Maria Tria Tirona, et al.. (2019). Capsaicinoids: Multiple effects on angiogenesis, invasion and metastasis in human cancers. Biomedicine & Pharmacotherapy. 118. 109317–109317. 46 indexed citations
7.
Perry, Haley E., Kathleen C. Brown, Austin T. Akers, et al.. (2019). Capsaicinoids enhance chemosensitivity to chemotherapeutic drugs. Advances in cancer research. 144. 263–298. 13 indexed citations
8.
Brown, Kathleen C., Nicholas Nolan, Austin T. Akers, et al.. (2018). Acetylcholine signaling system in progression of lung cancers. Pharmacology & Therapeutics. 194. 222–254. 85 indexed citations
9.
Akers, Austin T., Nicholas Nolan, Kathleen C. Brown, et al.. (2018). Anti‐cancer activity of non‐pungent capsaicin analogs: A Structure‐Activity Study. The FASEB Journal. 32(S1). 1 indexed citations
10.
Fischer, Adam & Sarah L. Miles. (2017). Silencing HIF-1α induces TET2 expression and augments ascorbic acid induced 5-hydroxymethylation of DNA in human metastatic melanoma cells. Biochemical and Biophysical Research Communications. 490(2). 176–181. 29 indexed citations
11.
Nolan, Nicholas, Kathleen C. Brown, Sarah L. Miles, et al.. (2017). Anticancer Activity of Natural and Synthetic Capsaicin Analogs. Journal of Pharmacology and Experimental Therapeutics. 364(3). 462–473. 60 indexed citations
13.
Miles, Sarah L., et al.. (2015). Ascorbic acid and ascorbate-2-phosphate decrease HIF activity and malignant properties of human melanoma cells. BMC Cancer. 15(1). 867–867. 39 indexed citations
14.
Calster, Joachim Van, José S. Pulido, Sarah L. Miles, et al.. (2015). Early diagnosis and successful treatment of paraneoplastic melanocytic proliferation. British Journal of Ophthalmology. 99(7). 943–948. 36 indexed citations
15.
Miles, Sarah L., Margaret McFarland, & Richard M. Niles. (2014). Molecular and physiological actions of quercetin: need for clinical trials to assess its benefits in human disease. Nutrition Reviews. 72(11). 720–734. 86 indexed citations
16.
Pulido, José S., Thomas J. Flotte, Harish Raja, et al.. (2013). Dermal and conjunctival melanocytic proliferations in diffuse uveal melanocytic proliferation. Eye. 27(9). 1058–1062. 21 indexed citations
17.
Bailey, Rachel M., Jason P. Covy, Heather L. Melrose, et al.. (2013). LRRK2 phosphorylates novel tau epitopes and promotes tauopathy. Acta Neuropathologica. 126(6). 809–827. 80 indexed citations
18.
Miles, Sarah L., Richard M. Niles, Sean J. Pittock, et al.. (2012). A FACTOR FOUND IN THE IGG FRACTION OF SERUM OF PATIENTS WITH PARANEOPLASTIC BILATERAL DIFFUSE UVEAL MELANOCYTIC PROLIFERATION CAUSES PROLIFERATION OF CULTURED HUMAN MELANOCYTES. Retina. 32(9). 1959–1966. 62 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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