Salomé McAllen

956 total citations
17 papers, 665 citations indexed

About

Salomé McAllen is a scholar working on Molecular Biology, Physiology and Neurology. According to data from OpenAlex, Salomé McAllen has authored 17 papers receiving a total of 665 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 8 papers in Physiology and 6 papers in Neurology. Recurrent topics in Salomé McAllen's work include Alzheimer's disease research and treatments (8 papers), Neuroinflammation and Neurodegeneration Mechanisms (4 papers) and Prion Diseases and Protein Misfolding (3 papers). Salomé McAllen is often cited by papers focused on Alzheimer's disease research and treatments (8 papers), Neuroinflammation and Neurodegeneration Mechanisms (4 papers) and Prion Diseases and Protein Misfolding (3 papers). Salomé McAllen collaborates with scholars based in United States, Italy and Hungary. Salomé McAllen's co-authors include Rakez Kayed, Mauro Montalbano, Urmi Sengupta, Nemil Bhatt, Nicha Puangmalai, Stephanie García, Sagar Gaikwad, Anna Ellsworth, Alice Bittar and Michael G. Kharas and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and Blood.

In The Last Decade

Salomé McAllen

16 papers receiving 662 citations

Peers

Salomé McAllen

PHYSI

NEURO

CMN

NEURO

Robert M. Crescentini United States

Kazumi Motoki Japan

Sergey Larionov Germany

Mary Dabney Davis United States

VIRGINIA M.‐Y. LEE United States

Ekaterina V. Ilieva Spain

Esther Barth Germany

Benedikt Bader Germany

Yogita Dheer Australia

Julien Chapuis France

Robert M. Crescentini United States

Salomé McAllen

416 ×1.1

PHYSI

187 ×0.7

280 ×1.4

NEURO

110 ×0.9

CMN

62 ×0.5

NEURO

Citations per year, relative to Salomé McAllen Salomé McAllen (= 1×) peers Robert M. Crescentini

Countries citing papers authored by Salomé McAllen

Since Specialization

Citations

This map shows the geographic impact of Salomé McAllen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Salomé McAllen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Salomé McAllen more than expected).

Fields of papers citing papers by Salomé McAllen

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Salomé McAllen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Salomé McAllen. The network helps show where Salomé McAllen may publish in the future.

Co-authorship network of co-authors of Salomé McAllen

This figure shows the co-authorship network connecting the top 25 collaborators of Salomé McAllen. A scholar is included among the top collaborators of Salomé McAllen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Salomé McAllen. Salomé McAllen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Nair, Ranjit, Jingjing Liu, Chen Zhou, et al.. (2024). An Integrated Spatial, Codex, and Genomic Analysis Predicts Responsiveness and Survival in the Phase II Combination of Pembrolizumab and Romidepsin in PTCL. Blood. 144(Supplement 1). 455–455. 1 indexed citations

Sun, Baohua, et al.. (2023). Multiplexed Barcoding Image Analysis for Immunoprofiling and Spatial Mapping Characterization in the Single-Cell Analysis of Paraffin Tissue Samples. Journal of Visualized Experiments.

Hussong, Stacy A., Candice E. Van Skike, Matthew J. Hart, et al.. (2023). Soluble pathogenic tau enters brain vascular endothelial cells and drives cellular senescence and brain microvascular dysfunction in a mouse model of tauopathy. Nature Communications. 14(1). 2367–2367. 37 indexed citations

Batra, Harsh Vardhan, Renganayaki Pandurengan, Heladio P. Ibarguen, et al.. (2023). Abstract P2-21-07: Exploring spatial correlations in Breast invasive Lobular Carcinoma subtypes using a novel CAF multiplex immunofluorescence panel. Cancer Research. 83(5_Supplement). P2–21. 1 indexed citations

Puangmalai, Nicha, Urmi Sengupta, Nemil Bhatt, et al.. (2022). Lysine 63-linked ubiquitination of tau oligomers contributes to the pathogenesis of Alzheimer’s disease. Journal of Biological Chemistry. 298(4). 101766–101766. 45 indexed citations

Bittar, Alice, Nemil Bhatt, Mauro Montalbano, et al.. (2022). Passive Immunotherapy Targeting Tau Oligomeric Strains Reverses Tauopathy Phenotypes in Aged Human-Tau Mice in a Mouse Model-Specific Manner. Journal of Alzheimer s Disease. 90(3). 1103–1122. 15 indexed citations

Gaikwad, Sagar, Nicha Puangmalai, Alice Bittar, et al.. (2021). Tau oligomer induced HMGB1 release contributes to cellular senescence and neuropathology linked to Alzheimer’s disease and frontotemporal dementia. Cell Reports. 36(3). 109419–109419. 153 indexed citations

Montalbano, Mauro, Elizabeth Jaworski, Stephanie García, et al.. (2021). Tau Modulates mRNA Transcription, Alternative Polyadenylation Profiles of hnRNPs, Chromatin Remodeling and Spliceosome Complexes. Frontiers in Molecular Neuroscience. 14. 742790–742790. 22 indexed citations

Cascio, Filippa Lo, Stephanie García, Mauro Montalbano, et al.. (2020). Modulating disease-relevant tau oligomeric strains by small molecules. Journal of Biological Chemistry. 295(44). 14807–14825. 40 indexed citations

10.

Puangmalai, Nicha, Nemil Bhatt, Mauro Montalbano, et al.. (2020). Internalization mechanisms of brain-derived tau oligomers from patients with Alzheimer’s disease, progressive supranuclear palsy and dementia with Lewy bodies. Cell Death and Disease. 11(5). 314–314. 75 indexed citations

11.

Montalbano, Mauro, Salomé McAllen, Nicha Puangmalai, et al.. (2020). RNA-binding proteins Musashi and tau soluble aggregates initiate nuclear dysfunction. Nature Communications. 11(1). 4305–4305. 67 indexed citations

12.

Montalbano, Mauro, Salomé McAllen, Filippa Lo Cascio, et al.. (2020). TDP-43 and Tau Oligomers in Alzheimer's Disease, Amyotrophic Lateral Sclerosis, and Frontotemporal Dementia. Neurobiology of Disease. 146. 105130–105130. 77 indexed citations

13.

Gaikwad, Sagar, Nicha Puangmalai, Alice Bittar, et al.. (2020). Tau Oligomer Induced HMGB1 Release Contributes to Cellular Senescence and Neuropathology Linked to Alzheimer's Disease and Frontotemporal Dementia. SSRN Electronic Journal. 6 indexed citations

14.

McAllen, Salomé, Urmi Sengupta, Nicha Puangmalai, et al.. (2019). Tau oligomers mediate aggregation of RNA‐binding proteins Musashi1 and Musashi2 inducing Lamin alteration. Aging Cell. 18(6). e13035–e13035. 31 indexed citations

15.

Bittar, Alice, Nemil Bhatt, Tasneem F. Hasan, et al.. (2019). Neurotoxic tau oligomers after single versus repetitive mild traumatic brain injury. Brain Communications. 1(1). fcz004–fcz004. 39 indexed citations

16.

Sengupta, Urmi, Mauro Montalbano, Salomé McAllen, et al.. (2018). Formation of Toxic Oligomeric Assemblies of RNA-binding Protein: Musashi in Alzheimer’s disease. Acta Neuropathologica Communications. 6(1). 113–113. 32 indexed citations

17.

Rothrock, John F., et al.. (1989). Acute anticoagulation following cardioembolic stroke.. Stroke. 20(6). 730–734. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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