Esther Barth

1.3k total citations
17 papers, 985 citations indexed

About

Esther Barth is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Esther Barth has authored 17 papers receiving a total of 985 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 6 papers in Physiology and 5 papers in Cellular and Molecular Neuroscience. Recurrent topics in Esther Barth's work include Mitochondrial Function and Pathology (7 papers), Alzheimer's disease research and treatments (6 papers) and Endoplasmic Reticulum Stress and Disease (3 papers). Esther Barth is often cited by papers focused on Mitochondrial Function and Pathology (7 papers), Alzheimer's disease research and treatments (6 papers) and Endoplasmic Reticulum Stress and Disease (3 papers). Esther Barth collaborates with scholars based in Germany, Italy and United Kingdom. Esther Barth's co-authors include Elena I. Rugarli, Jochen Walter, Irfan Y. Tamboli, Michael T. Heneka, Dieter Lütjohann, Sigrun I. Korsching, H. Thoenen, Kai Prager, Thomas Langer and Paola Martinelli and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Nature Communications.

In The Last Decade

Esther Barth

17 papers receiving 968 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Esther Barth Germany 13 694 276 157 148 119 17 985
Catherine Theisler United States 9 542 0.8× 238 0.9× 236 1.5× 199 1.3× 121 1.0× 9 1.1k
Heike Naumann Germany 15 800 1.2× 135 0.5× 177 1.1× 164 1.1× 145 1.2× 18 1.3k
Diego Grassi United States 10 470 0.7× 466 1.7× 179 1.1× 42 0.3× 120 1.0× 17 1.1k
Masayuki Tobo Japan 20 940 1.4× 226 0.8× 90 0.6× 115 0.8× 132 1.1× 27 1.2k
Shunzhong Bao United States 19 598 0.9× 183 0.7× 52 0.3× 313 2.1× 248 2.1× 28 1.0k
Sonia S. Jung United States 12 364 0.5× 627 2.3× 159 1.0× 60 0.4× 72 0.6× 21 1.1k
Koei Shinzawa Japan 14 360 0.5× 105 0.4× 113 0.7× 74 0.5× 114 1.0× 20 755
Mu‐En Lin United States 7 1.1k 1.5× 186 0.7× 130 0.8× 68 0.5× 302 2.5× 7 1.3k
Anke Penno Switzerland 16 876 1.3× 353 1.3× 193 1.2× 58 0.4× 381 3.2× 19 1.2k
Simona Perga Italy 19 502 0.7× 322 1.2× 259 1.6× 197 1.3× 92 0.8× 28 992

Countries citing papers authored by Esther Barth

Since Specialization
Citations

This map shows the geographic impact of Esther Barth's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Esther Barth with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Esther Barth more than expected).

Fields of papers citing papers by Esther Barth

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Esther Barth. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Esther Barth. The network helps show where Esther Barth may publish in the future.

Co-authorship network of co-authors of Esther Barth

This figure shows the co-authorship network connecting the top 25 collaborators of Esther Barth. A scholar is included among the top collaborators of Esther Barth based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Esther Barth. Esther Barth is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Zaninello, Marta, Hendrik Nolte, Esther Barth, et al.. (2024). CLUH maintains functional mitochondria and translation in motoneuronal axons and prevents peripheral neuropathy. Science Advances. 10(22). eadn2050–eadn2050. 5 indexed citations
2.
Iavarone, Francescopaolo, Marta Zaninello, Esther Barth, et al.. (2024). Fam134c and Fam134b shape axonal endoplasmic reticulum architecture in vivo. EMBO Reports. 25(8). 3651–3677. 6 indexed citations
3.
Nolte, Hendrik, et al.. (2023). SARM1 deletion delays cerebellar but not spinal cord degeneration in an enhanced mouse model of SPG7 deficiency. Brain. 146(10). 4117–4131. 6 indexed citations
4.
Schatton, Désirée, Giada Di Pietro, Karolina Szczepanowska, et al.. (2022). CLUH controls astrin-1 expression to couple mitochondrial metabolism to cell cycle progression. eLife. 11. 12 indexed citations
5.
Nemazanyy, Ivan, M Girard, Esther Barth, et al.. (2019). The class 3 PI3K coordinates autophagy and mitochondrial lipid catabolism by controlling nuclear receptor PPARα. Nature Communications. 10(1). 1566–1566. 82 indexed citations
6.
Heß, Simon, Esther Barth, Désirée Schatton, et al.. (2019). Astrocyte‐specific deletion of the mitochondrial m‐AAA protease reveals glial contribution to neurodegeneration. Glia. 67(8). 1526–1541. 35 indexed citations
7.
Sprenger, Hans‐Georg, Tim König, Maria Patrón, et al.. (2018). Loss of the mitochondrial i ‐ AAA protease YME 1L leads to ocular dysfunction and spinal axonopathy. EMBO Molecular Medicine. 11(1). 44 indexed citations
8.
Schatton, Désirée, David Pla‐Martín, Arnaud Mourier, et al.. (2017). CLUH regulates mitochondrial metabolism by controlling translation and decay of target mRNAs. The Journal of Cell Biology. 216(3). 675–693. 68 indexed citations
9.
Wang, Shuaiyu, Julie Jacquemyn, Paola Martinelli, et al.. (2016). The Mitochondrial m-AAA Protease Prevents Demyelination and Hair Greying. PLoS Genetics. 12(12). e1006463–e1006463. 23 indexed citations
10.
Gao, Jie, Désirée Schatton, Paola Martinelli, et al.. (2014). CLUH regulates mitochondrial biogenesis by binding mRNAs of nuclear-encoded mitochondrial proteins. The Journal of Cell Biology. 207(2). 213–223. 98 indexed citations
11.
Richter, Ricarda, Lars Paeger, Paola Martinelli, et al.. (2012). AFG3L2 supports mitochondrial protein synthesis and Purkinje cell survival. Journal of Clinical Investigation. 122(11). 4048–4058. 84 indexed citations
12.
Mancuso, Giuseppe, Esther Barth, Pietro Crivello, & Elena I. Rugarli. (2012). Alternative Splicing of Spg7, a Gene Involved in Hereditary Spastic Paraplegia, Encodes a Variant of Paraplegin Targeted to the Endoplasmic Reticulum. PLoS ONE. 7(5). e36337–e36337. 7 indexed citations
13.
Tamboli, Irfan Y., Esther Barth, Martin Siepmann, et al.. (2010). Statins Promote the Degradation of Extracellular Amyloid β-Peptide by Microglia via Stimulation of Exosome-associated Insulin-degrading Enzyme (IDE) Secretion. Journal of Biological Chemistry. 285(48). 37405–37414. 183 indexed citations
14.
Prager, Kai, Lihua Wang‐Eckhardt, Regina Fluhrer, et al.. (2007). A Structural Switch of Presenilin 1 by Glycogen Synthase Kinase 3β-mediated Phosphorylation Regulates the Interaction with β-Catenin and Its Nuclear Signaling. Journal of Biological Chemistry. 282(19). 14083–14093. 23 indexed citations
15.
Tamboli, Irfan Y., Kai Prager, Esther Barth, et al.. (2005). Inhibition of Glycosphingolipid Biosynthesis Reduces Secretion of the β-Amyloid Precursor Protein and Amyloid β-Peptide*[boxs]. Journal of Biological Chemistry. 280(30). 28110–28117. 114 indexed citations
16.
Lütjohann, Dieter, Esther Barth, Dorothée Abramowski, et al.. (2002). Profile of cholesterol-related sterols in aged amyloid precursor protein transgenic mouse brain. Journal of Lipid Research. 43(7). 1078–1085. 127 indexed citations
17.
Barth, Esther, Sigrun I. Korsching, & H. Thoenen. (1984). Regulation of nerve growth factor synthesis and release in organ cultures of rat iris.. The Journal of Cell Biology. 99(3). 839–843. 68 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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