Ryan M. Cinalli
- Cancer Research top 0.5%
- Cancer, Hypoxia, and Metabolism 2
- Aging top 5%
- Molecular Biology top 2%
- Epigenetics and DNA Methylation 2
- PI3K/AKT/mTOR signaling in cancer 2
- Protein Kinase Regulation and GTPase Signaling 2
- Immunology top 5%
- Immune Cell Function and Interaction 2
- Oncology top 5%
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- Cancer Mechanisms and Therapy 2
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- Cellular transport and secretion 2
- Microtubule and mitosis dynamics 2
- Co-authors
- Craig B. ThompsonDavid R. PlasJeffrey C. RathmellCasey FoxRebecca ElstromPeter S. HammermanDavid P. BartelMargaret E. Glasner
- Partner nations
- United StatesUnited KingdomSlovakia
In The Last Decade
Ryan M. Cinalli
15 papers receiving 4.0k citations
Hit Papers
Peers
Comparison fields: 5 of 122
- Cancer Research 1.8k
- Aging 89
- Molecular Biology 3.0k
- Immunology 578
- Oncology 557
Countries citing papers authored by Ryan M. Cinalli
This map shows the geographic impact of Ryan M. Cinalli's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ryan M. Cinalli with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ryan M. Cinalli more than expected).
Fields of papers citing papers by Ryan M. Cinalli
This network shows the impact of papers produced by Ryan M. Cinalli. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ryan M. Cinalli. The network helps show where Ryan M. Cinalli may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Ryan M. Cinalli, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2017 | 131 | |
| 2 | 2017 | 29 | |
| 3 | 2013 | 48 | |
| 4 | 2008 | 88 | |
| 5 | 2008 | 100 | |
| 6 | MicroRNAs Regulate Brain Morphogenesis in Zebrafishbreakdown → | 2005 | 1030 |
| 7 | 2005 | 15 | |
| 8 | 2004 | 125 | |
| 9 | Akt Stimulates Aerobic Glycolysis in Cancer Cellsbreakdown → | 2004 | 1171 |
| 10 | 2004 | 78 | |
| 11 | 2003 | 271 | |
| 12 | 2003 | 478 | |
| 13 | 2003 | 158 | |
| 14 | 2003 | 107 | |
| 15 | 2002 | 242 |
About Ryan M. Cinalli
Ryan M. Cinalli is a scholar working on Aging, Cell Biology and Developmental Neuroscience, having authored 15 papers that have together received 4.1k indexed citations. Recurring topics across this work include Cancer Mechanisms and Therapy (2 papers), Cancer, Hypoxia, and Metabolism (2 papers), Epigenetics and DNA Methylation (2 papers), Cellular transport and secretion (2 papers), Microtubule and mitosis dynamics (2 papers), Immune Cell Function and Interaction (2 papers), PI3K/AKT/mTOR signaling in cancer (2 papers) and Protein Kinase Regulation and GTPase Signaling (2 papers). The work is most often cited by research in Cancer Research (1.8k citations), Aging (89 citations) and Molecular Biology (3.0k citations). Ryan M. Cinalli has collaborated with scholars based in United States, United Kingdom and Slovakia. Frequent co-authors include Craig B. Thompson, David R. Plas, Jeffrey C. Rathmell, Casey Fox, Rebecca Elstrom, Peter S. Hammerman, David P. Bartel, Margaret E. Glasner, Antonio J. Giráldez and Scott Baskerville. Their work appears in journals such as Science, Developmental Cell, European Journal of Immunology, Cancer Research and Genes & Development.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.